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A Chimeric Toxin Vaccine Protects against Primary and Recurrent Clostridium difficile Infection

Infection and Immunity, 2012-08, Vol.80 (8), p.2678-2688

2012

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Identification of toxemia in patients with Clostridium difficile infection

PloS one, 2015-04, Vol.10 (4), p.e0124235-e0124235

2015

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A segment of 97 amino acids within the translocation domain of Clostridium difficile toxin B is essential for toxicity

PloS one, 2013-03, Vol.8 (3), p.e58634

2013

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Utility of Clostridium difficile toxin B for inducing anti-tumor immunity

PloS one, 2014-10, Vol.9 (10), p.e110826-e110826

2014

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Inhibition of apoptosis in chlamydia-infected cells: blockade of mitochondrial cytochrome c release and caspase activation

The Journal of experimental medicine, 1998-02, Vol.187 (4), p.487-496

1998

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Granzyme B Induces BID-mediated Cytochrome c Release and Mitochondrial Permeability Transition

The Journal of biological chemistry, 2001-03, Vol.276 (10), p.6974-6982

2001

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Nix and Nip3 Form a Subfamily of Pro-apoptotic Mitochondrial Proteins

The Journal of biological chemistry, 1999-01, Vol.274 (1), p.7-10

1999

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Granzyme A, which causes single-stranded DNA damage, targets the double-strand break repair protein Ku70

EMBO reports, 2006-04, Vol.7 (4), p.431-437

2006

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Mitochondria-dependent and -independent regulation of Granzyme B-induced apoptosis

The Journal of experimental medicine, 1999-01, Vol.189 (1), p.131-144

1999

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Inhibition of ICE family proteases by baculovirus antiapoptotic protein p35

Science (American Association for the Advancement of Science), 1995-09, Vol.269 (5232), p.1885-1888

1995

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Granzyme B (GraB) autonomously crosses the cell membrane and perforin initiates apoptosis and GraB nuclear localization

The Journal of experimental medicine, 1997-03, Vol.185 (5), p.855-866

1997

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Activation of Caspase-2 in Apoptosis

The Journal of biological chemistry, 1997-08, Vol.272 (34), p.21010-21017

1997

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Purification of three cytotoxic lymphocyte granule serine proteases that induce apoptosis through distinct substrate and target cell interactions

The Journal of experimental medicine, 1992-12, Vol.176 (6), p.1521-1529

1992

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A natural killer cell granule protein that induces DNA fragmentation and apoptosis

The Journal of experimental medicine, 1992-02, Vol.175 (2), p.553-566

1992

Volltextzugriff (PDF)

Single domain antibody coated gold nanoparticles as enhancer for Clostridium difficile toxin detection by electrochemical impedance immunosensors

Bioelectrochemistry (Amsterdam, Netherlands), 2015-02, Vol.101, p.153-158

2015

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Cytotoxicity of Clostridium difficile toxin B does not require cysteine protease-mediated autocleavage and release of the glucosyltransferase domain into the host cell cytosol

Pathogens and disease, 2013-02, Vol.67 (1), p.11-18

2013

Volltextzugriff (PDF)

Perforin Triggers a Plasma Membrane-Repair Response that Facilitates CTL Induction of Apoptosis

Immunity (Cambridge, Mass.), 2005-09, Vol.23 (3), p.249-262

2005

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Critical roles of Clostridium difficile toxin B enzymatic activities in pathogenesis

Infection and immunity, 2015-02, Vol.83 (2), p.502-513

2015

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Host S-nitrosylation inhibits clostridial small molecule-activated glucosylating toxins

Nature medicine, 2011-09, Vol.17 (9), p.1136-1141

2011

Volltextzugriff (PDF)

Intravenous adenovirus expressing a multi-specific, single-domain antibody neutralizing TcdA and TcdB protects mice from Clostridium difficile infection

Pathogens and disease, 2016-10, Vol.74 (7), p.ftw078

2016

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Interaction of Cryptosporidium parvum with mouse dendritic cells leads to their activation and parasite transportation to mesenteric lymph nodes

Pathogens and disease, 2014-02, Vol.70 (1), p.17-27

2014

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Cysteine Protease-Mediated Autocleavage of Clostridium difficile Toxins Regulates Their Proinflammatory Activity

Cellular and molecular gastroenterology and hepatology, 2018-01, Vol.5 (4), p.611-625

2018

Volltextzugriff (PDF)

Cysteine Protease-Mediated Autocleavage of Clostridium difficile Toxins Regulates Their Proinflammatory ActivitySummary

Cellular and molecular gastroenterology and hepatology, 2018-01, Vol.5 (4), p.611-625

2018

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Granzyme B Binds to Target Cells Mostly by Charge and Must Be Added at the Same Time as Perforin to Trigger Apoptosis

The Journal of immunology (1950), 2005-05, Vol.174 (9), p.5456-5461

2005

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Defects in regulation of apoptosis in caspase-2-deficient mice

Genes & development, 1998-05, Vol.12 (9), p.1304-1314

1998

Volltextzugriff (PDF)

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A Chimeric Toxin Vaccine Protects against Primary and Recurrent Clostridium difficile Infection

Identification of toxemia in patients with Clostridium difficile infection

A segment of 97 amino acids within the translocation domain of Clostridium difficile toxin B is essential for toxicity

Utility of Clostridium difficile toxin B for inducing anti-tumor immunity

Inhibition of apoptosis in chlamydia-infected cells: blockade of mitochondrial cytochrome c release and caspase activation

Granzyme B Induces BID-mediated Cytochrome c Release and Mitochondrial Permeability Transition

Nix and Nip3 Form a Subfamily of Pro-apoptotic Mitochondrial Proteins

Granzyme A, which causes single-stranded DNA damage, targets the double-strand break repair protein Ku70

Mitochondria-dependent and -independent regulation of Granzyme B-induced apoptosis

Inhibition of ICE family proteases by baculovirus antiapoptotic protein p35

Granzyme B (GraB) autonomously crosses the cell membrane and perforin initiates apoptosis and GraB nuclear localization

Activation of Caspase-2 in Apoptosis

Purification of three cytotoxic lymphocyte granule serine proteases that induce apoptosis through distinct substrate and target cell interactions

A natural killer cell granule protein that induces DNA fragmentation and apoptosis

Single domain antibody coated gold nanoparticles as enhancer for Clostridium difficile toxin detection by electrochemical impedance immunosensors

Cytotoxicity of Clostridium difficile toxin B does not require cysteine protease-mediated autocleavage and release of the glucosyltransferase domain into the host cell cytosol

Perforin Triggers a Plasma Membrane-Repair Response that Facilitates CTL Induction of Apoptosis

Critical roles of Clostridium difficile toxin B enzymatic activities in pathogenesis

Host S-nitrosylation inhibits clostridial small molecule-activated glucosylating toxins

Intravenous adenovirus expressing a multi-specific, single-domain antibody neutralizing TcdA and TcdB protects mice from Clostridium difficile infection

Interaction of Cryptosporidium parvum with mouse dendritic cells leads to their activation and parasite transportation to mesenteric lymph nodes

Cysteine Protease-Mediated Autocleavage of Clostridium difficile Toxins Regulates Their Proinflammatory Activity

Cysteine Protease-Mediated Autocleavage of Clostridium difficile Toxins Regulates Their Proinflammatory ActivitySummary

Granzyme B Binds to Target Cells Mostly by Charge and Must Be Added at the Same Time as Perforin to Trigger Apoptosis

Defects in regulation of apoptosis in caspase-2-deficient mice

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