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Optimized Sleeping Beauty transposons rapidly generate stable transgenic cell lines

Biotechnology journal, 2015-04, Vol.10 (4), p.647-653

2015

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Vaccine-induced COVID-19 mimicry syndrome

eLife, 2022-01, Vol.11

2022

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Image-Based Annotation of Chemogenomic Libraries for Phenotypic Screening

Molecules (Basel, Switzerland), 2022-02, Vol.27 (4), p.1439

2022

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Generation of a Sleeping Beauty Transposon-Based Cellular System for Rapid and Sensitive Screening for Compounds and Cellular Factors Limiting SARS-CoV-2 Replication

Frontiers in microbiology, 2021-07, Vol.12, p.701198-701198

2021

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MLL-AF4 and a murinized pSer-variant thereof are turning on the nucleolar stress pathway

Cell & bioscience, 2022-04, Vol.12 (1), p.47-47, Article 47

2022

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How chromosomal translocations arise to cause cancer: Gene proximity, trans-splicing, and DNA end joining

iScience, 2023-06, Vol.26 (6), p.106900-106900, Article 106900

2023

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Distinct and targetable role of calcium-sensing receptor in leukaemia

Nature communications, 2023-10, Vol.14 (1), p.6242-6242, Article 6242

2023

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The insecticides permethrin and chlorpyrifos show limited genotoxicity and no leukemogenic potential in human and murine hematopoietic stem progenitor cells

Haematologica (Roma), 2022-02, Vol.107 (2), p.544-549

2022

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The MLL recombinome of adult CD10-negative B-cell precursor acute lymphoblastic leukemia: results from the GMALL study group

Blood, 2009-04, Vol.113 (17), p.4011-4015

2009

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The role of reciprocal fusions in MLL-r acute leukemia: studying the chromosomal translocation t(6;11)

Oncogene, 2021-10, Vol.40 (40), p.5902-5912

2021

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Unraveling the Activation Mechanism of Taspase1 which Controls the Oncogenic AF4–MLL Fusion Protein

EBioMedicine, 2015-05, Vol.2 (5), p.386-395

2015

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Biology of t(6;11) Fusion Proteins and Their Role in MLL-Rearranged Acute Leukemia Lineage Determination

Blood, 2019-11, Vol.134 (Supplement_1), p.5033-5033

2019

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Model System to Analyze RNA-Mediated DNA Repair in Mammalian Cells

CRISPR journal, 2023-06, Vol.6 (3), p.289-301

2023

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Distinct and Targetable Role of Calcium and Calcium Sensing Receptor in Leukaemia

Blood, 2023-11, Vol.142 (Supplement 1), p.5709-5709

2023

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Genomic DNA of leukemic patients: Target for clinical diagnosis of MLL rearrangements

Biotechnology journal, 2006-06, Vol.1 (6), p.656-663

2006

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Do non-genomically encoded fusion transcripts cause recurrent chromosomal translocations?

Cancers, 2012-10, Vol.4 (4), p.1036-1049

2012

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Functional analysis of the two reciprocal fusion genes MLL-NEBL and NEBL-MLL reveal their oncogenic potential

Cancer letters, 2013-05, Vol.332 (1), p.30-34

2013

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The differential role of the lipid raft-associated protein flotillin 2 for progression of myeloid leukemia

Blood advances, 2022-06, Vol.6 (12), p.3611-3624

2022

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AF4 and AF4N protein complexes: recruitment of P-TEFb kinase, their interactome and potential functions

American journal of blood research, 2015-01, Vol.5 (1), p.10-24

2015

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Premature transcript termination, trans-splicing and DNA repair: a vicious path to cancer

American journal of blood research, 2011-01, Vol.1 (1), p.1-12

2011

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Three-Way Translocations and Insertions Are the Molecular Basis for Complex MLL Rearrangements

Blood, 2007-11, Vol.110 (11), p.979-979

2007

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The MLL Recombinome of Adult ALL - Results from the GMALL Study Group

Blood, 2007-11, Vol.110 (11), p.977-977

2007

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New Insights to the MLL Recombinome Including 8 Novel Partner Genes ACTN4, C2CD3, DCP1A, FLNA, LAMC3, LOC100128568, NRIP3, and TNRC18

Blood, 2008-11, Vol.112 (11), p.2047-2047

2008

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A complex MLL rearrangement identified five years after initial MDS diagnosis results in out-of-frame fusions without progression to acute leukemia

Cancer genetics, 2011-10, Vol.204 (10), p.557-562

2011

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Novel Spliced MLL Fusions Have Been Identified Involving the MLL Partner Genes ELL, EPS15, MLLT3, and SEPT5

Blood, 2007-11, Vol.110 (11), p.978-978

2007

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Optimized Sleeping Beauty transposons rapidly generate stable transgenic cell lines

Vaccine-induced COVID-19 mimicry syndrome

Image-Based Annotation of Chemogenomic Libraries for Phenotypic Screening

Generation of a Sleeping Beauty Transposon-Based Cellular System for Rapid and Sensitive Screening for Compounds and Cellular Factors Limiting SARS-CoV-2 Replication

MLL-AF4 and a murinized pSer-variant thereof are turning on the nucleolar stress pathway

How chromosomal translocations arise to cause cancer: Gene proximity, trans-splicing, and DNA end joining

Distinct and targetable role of calcium-sensing receptor in leukaemia

The insecticides permethrin and chlorpyrifos show limited genotoxicity and no leukemogenic potential in human and murine hematopoietic stem progenitor cells

The MLL recombinome of adult CD10-negative B-cell precursor acute lymphoblastic leukemia: results from the GMALL study group

The role of reciprocal fusions in MLL-r acute leukemia: studying the chromosomal translocation t(6;11)

Unraveling the Activation Mechanism of Taspase1 which Controls the Oncogenic AF4–MLL Fusion Protein

Biology of t(6;11) Fusion Proteins and Their Role in MLL-Rearranged Acute Leukemia Lineage Determination

Model System to Analyze RNA-Mediated DNA Repair in Mammalian Cells

Distinct and Targetable Role of Calcium and Calcium Sensing Receptor in Leukaemia

Genomic DNA of leukemic patients: Target for clinical diagnosis of MLL rearrangements

Do non-genomically encoded fusion transcripts cause recurrent chromosomal translocations?

Functional analysis of the two reciprocal fusion genes MLL-NEBL and NEBL-MLL reveal their oncogenic potential

The differential role of the lipid raft-associated protein flotillin 2 for progression of myeloid leukemia

AF4 and AF4N protein complexes: recruitment of P-TEFb kinase, their interactome and potential functions

Premature transcript termination, trans-splicing and DNA repair: a vicious path to cancer

Three-Way Translocations and Insertions Are the Molecular Basis for Complex MLL Rearrangements

The MLL Recombinome of Adult ALL - Results from the GMALL Study Group

New Insights to the MLL Recombinome Including 8 Novel Partner Genes ACTN4, C2CD3, DCP1A, FLNA, LAMC3, LOC100128568, NRIP3, and TNRC18

A complex MLL rearrangement identified five years after initial MDS diagnosis results in out-of-frame fusions without progression to acute leukemia

Novel Spliced MLL Fusions Have Been Identified Involving the MLL Partner Genes ELL, EPS15, MLLT3, and SEPT5

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