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IL-6 activated JAK/STAT3 pathway and sensitivity to Hsp90 inhibitors in multiple myeloma

Current medicinal chemistry, 2014-01, Vol.21 (26), p.3042-3047

2014

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Irreversible TrxR1 inhibitors block STAT3 activity and induce cancer cell death

Science advances, 2020-03, Vol.6 (12), p.eaax7945-eaax7945

2020

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Therapeutic implications of tumor interstitial acidification

Seminars in cancer biology, 2017-04, Vol.43, p.119-133

2017

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Polo-like kinases and acute leukemia

Oncogene, 2019, Vol.38 (1), p.1-16

2019

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Thermal proteome profiling identifies PIP4K2A and ZADH2 as off‐targets of Polo‐like kinase 1 inhibitor volasertib

The FASEB journal, 2021-07, Vol.35 (7), p.e21741-n/a

2021

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Targeting Plk1 with siRNNs in primary cells from pediatric B-cell acute lymphoblastic leukemia patients

Scientific reports, 2020-02, Vol.10 (1), p.2688, Article 2688

2020

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RNAi prodrugs decrease elevated mRNA levels of Polo‐like kinase 1 in ex vivo cultured primary cells from pediatric acute myeloid leukemia patients

The FASEB journal, 2021-05, Vol.35 (5), p.e21476-n/a

2021

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miR-185 and miR-133b deregulation is associated with overall survival and metastasis in colorectal cancer

International journal of oncology, 2011-08, Vol.39 (2), p.311-318

2011

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CORACLE (COVID-19 liteRAture CompiLEr): A platform for efficient tracking and extraction of SARS-CoV-2 and COVID-19 literature, with examples from post-COVID with respiratory involvement

Computational and structural biotechnology journal, 2024-12, Vol.23, p.2661-2668

2024

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Designing siRNA and Evaluating Its Effect on RNA Targets Using qPCR and Western Blot

Methods in molecular biology (Clifton, N.J.), 2019, Vol.2036, p.53

2019

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STAT3 is activated in multicellular spheroids of colon carcinoma cells and mediates expression of IRF9 and interferon stimulated genes

Scientific reports, 2019-01, Vol.9 (1), p.536-536, Article 536

2019

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STAT3 differential scanning fluorimetry and differential scanning light scattering assays: Addressing a missing link in the characterization of STAT3 inhibitor interactions

Journal of pharmaceutical and biomedical analysis, 2018-10, Vol.160, p.80-88

2018

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Identification of novel small molecules that inhibit STAT3-dependent transcription and function

PloS one, 2017-06, Vol.12 (6), p.e0178844

2017

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RNAi prodrugs targeting Plk1 induce specific gene silencing in primary cells from pediatric T-acute lymphoblastic leukemia patients

Journal of controlled release, 2017-09, Vol.261, p.199-206

2017

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PLK1 as a cooperating partner for BCL2-mediated antiapoptotic program in leukemia

Blood cancer journal (New York), 2023-09, Vol.13 (1), p.139-139, Article 139

2023

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A drug screening assay on cancer cells chronically adapted to acidosis

Cancer cell international, 2018-09, Vol.18 (1), p.147-147, Article 147

2018

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Cell crowding induces interferon regulatory factor 9, which confers resistance to chemotherapeutic drugs

International journal of cancer, 2015-02, Vol.136 (4), p.E51-E61

2015

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An activated JAK/STAT3 pathway and CD45 expression are associated with sensitivity to Hsp90 inhibitors in multiple myeloma

Experimental cell research, 2013-03, Vol.319 (5), p.600-611

2013

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Novel RNAi Prodrugs Targeting Plk1 Induce Specific mRNA Cleavage in Pediatric Acute B-Cell Lymphoblastic Leukemia

CLINICAL LYMPHOMA MYELOMA & LEUKEMIA, 2020, Vol.20, p.S163

2020

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Activation of STAT1 is required for interferon-alpha-mediated cell death

Experimental cell research, 2011, Vol.317 (1), p.9-19

2011

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Cell-to-cell contact triggers the expression of the interferon-stimulated genes and mediates therapy resistance in cancer cell lines through a soluble factor

CYTOKINE, 2015, Vol.76 (1), p.105

2015

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Cell crowding results in accumulation of interferon-stimulated genes independently of STAT1

CYTOKINE, 2013, Vol.63 (3), p.278

2013

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Association of interferon stimulated genes and IRF9 upregulation with drug resistance in multicellular spheroids and monolayer cells upon crowding

CYTOKINE, 2013, Vol.63 (3), p.303

2013

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Spontaneous time-dependent accumulation of interferon-regulatory factor 9 induces chemo-resistance in colon carcinoma cells independently of STAT1 signaling

CANCER RESEARCH, 2013, Vol.73 (8)

2013

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Activated JAK-STAT3 pathway is associated with sensitivity to Hsp90 inhibitors in multiple myeloma

CYTOKINE, 2011, Vol.56 (1), p.41

2011

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IL-6 activated JAK/STAT3 pathway and sensitivity to Hsp90 inhibitors in multiple myeloma

Irreversible TrxR1 inhibitors block STAT3 activity and induce cancer cell death

Therapeutic implications of tumor interstitial acidification

Polo-like kinases and acute leukemia

Thermal proteome profiling identifies PIP4K2A and ZADH2 as off‐targets of Polo‐like kinase 1 inhibitor volasertib

Targeting Plk1 with siRNNs in primary cells from pediatric B-cell acute lymphoblastic leukemia patients

RNAi prodrugs decrease elevated mRNA levels of Polo‐like kinase 1 in ex vivo cultured primary cells from pediatric acute myeloid leukemia patients

miR-185 and miR-133b deregulation is associated with overall survival and metastasis in colorectal cancer

CORACLE (COVID-19 liteRAture CompiLEr): A platform for efficient tracking and extraction of SARS-CoV-2 and COVID-19 literature, with examples from post-COVID with respiratory involvement

Designing siRNA and Evaluating Its Effect on RNA Targets Using qPCR and Western Blot

STAT3 is activated in multicellular spheroids of colon carcinoma cells and mediates expression of IRF9 and interferon stimulated genes

STAT3 differential scanning fluorimetry and differential scanning light scattering assays: Addressing a missing link in the characterization of STAT3 inhibitor interactions

Identification of novel small molecules that inhibit STAT3-dependent transcription and function

RNAi prodrugs targeting Plk1 induce specific gene silencing in primary cells from pediatric T-acute lymphoblastic leukemia patients

PLK1 as a cooperating partner for BCL2-mediated antiapoptotic program in leukemia

A drug screening assay on cancer cells chronically adapted to acidosis

Cell crowding induces interferon regulatory factor 9, which confers resistance to chemotherapeutic drugs

An activated JAK/STAT3 pathway and CD45 expression are associated with sensitivity to Hsp90 inhibitors in multiple myeloma

Novel RNAi Prodrugs Targeting Plk1 Induce Specific mRNA Cleavage in Pediatric Acute B-Cell Lymphoblastic Leukemia

Activation of STAT1 is required for interferon-alpha-mediated cell death

Cell-to-cell contact triggers the expression of the interferon-stimulated genes and mediates therapy resistance in cancer cell lines through a soluble factor

Cell crowding results in accumulation of interferon-stimulated genes independently of STAT1

Association of interferon stimulated genes and IRF9 upregulation with drug resistance in multicellular spheroids and monolayer cells upon crowding

Spontaneous time-dependent accumulation of interferon-regulatory factor 9 induces chemo-resistance in colon carcinoma cells independently of STAT1 signaling

Activated JAK-STAT3 pathway is associated with sensitivity to Hsp90 inhibitors in multiple myeloma

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