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Differentiation of type 1 ILCs from a common progenitor to all helper-like innate lymphoid cell lineages

Cell, 2014-04, Vol.157 (2), p.340-356

2014

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The Transcription Factor GATA-3 Controls Cell Fate and Maintenance of Type 2 Innate Lymphoid Cells

Immunity (Cambridge, Mass.), 2012-10, Vol.37 (4), p.634-648

2012

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Innate lymphoid cells as regulators of immunity, inflammation and tissue homeostasis

Nature immunology, 2016-07, Vol.17 (7), p.765-774

2016

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The neuropeptide NMU amplifies ILC2-driven allergic lung inflammation

Nature (London), 2017-09, Vol.549 (7672), p.351-356

2017

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Interleukin-33 Induces the Enzyme Tryptophan Hydroxylase 1 to Promote Inflammatory Group 2 Innate Lymphoid Cell-Mediated Immunity

Immunity (Cambridge, Mass.), 2020-04, Vol.52 (4), p.606-619.e6

2020

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The Transcription Factor T-bet Is Induced by IL-15 and Thymic Agonist Selection and Controls CD8αα+ Intraepithelial Lymphocyte Development

Immunity (Cambridge, Mass.), 2014-08, Vol.41 (2), p.230-243

2014

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Anti-microbial Functions of Group 3 Innate Lymphoid Cells in Gut-Associated Lymphoid Tissues Are Regulated by G-Protein-Coupled Receptor 183

Cell reports (Cambridge), 2018-06, Vol.23 (13), p.3750-3758

2018

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Lack of Type 2 Innate Lymphoid Cells Promotes a Type I-Driven Enhanced Immune Response in Contact Hypersensitivity

Journal of investigative dermatology, 2018-09, Vol.138 (9), p.1962-1972

2018

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ILC3s restrict the dissemination of intestinal bacteria to safeguard liver regeneration after surgery

Cell reports (Cambridge), 2023-03, Vol.42 (3), p.112269-112269, Article 112269

2023

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Tissue microenvironment dictates the fate and tumor-suppressive function of type 3 ILCs

The Journal of experimental medicine, 2017-08, Vol.214 (8), p.2331-2347

2017

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Dynamic, Transient, and Robust Increase in the Innervation of the Inflamed Mucosa in Inflammatory Bowel Diseases

Cells (Basel, Switzerland), 2021-08, Vol.10 (9), p.2253

2021

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Innate lymphoid cells control signaling circuits to regulate tissue-specific immunity

Cell research, 2020-06, Vol.30 (6), p.475-491

2020

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Interleukin-22 protects intestinal stem cells against genotoxic stress

Nature (London), 2019-02, Vol.566 (7743), p.249-253

2019

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The transcription factor GATA3 controls cell fate and maintenance of type 2 innate lymphoid cells

Immunity (Cambridge, Mass.), 2012-10, Vol.37 (4), p.634-648

2012

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The neuropeptide neuromedin U stimulates innate lymphoid cells and type 2 inflammation

Nature (London), 2017-09, Vol.549 (7671), p.282-286

2017

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β2-adrenergic receptor–mediated negative regulation of group 2 innate lymphoid cell responses

Science (American Association for the Advancement of Science), 2018-03, Vol.359 (6379), p.1056-1061

2018

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A T-bet gradient controls the fate and function of CCR6-RORγt+ innate lymphoid cells

Nature (London), 2013-02, Vol.494 (7436), p.261-265

2013

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Neuroimmune interactions in peripheral tissues

European journal of immunology, 2021-07, Vol.51 (7), p.1602-1614

2021

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Neuro-Immune Circuits Regulate Immune Responses in Tissues and Organ Homeostasis

Frontiers in immunology, 2020-03, Vol.11, p.308

2020

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NK Cell Development in Times of Innate Lymphoid Cell Diversity

Frontiers in immunology, 2020-07, Vol.11, p.813-813

2020

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Neuro-immune-metabolism: The tripod system of homeostasis

Immunology letters, 2021-12, Vol.240, p.77-97

2021

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Type 2 Immunity Regulates Dermal White Adipose Tissue Function

Journal of investigative dermatology, 2023-12, Vol.143 (12), p.2456-2467.e5

2023

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Non-redundant functions of group 2 innate lymphoid cells

Nature (London), 2022-11, Vol.611 (7937), p.794-800

2022

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Cooperation of ILC2s and TH2 cells in the expulsion of intestinal helminth parasites

Nature reviews. Immunology, 2024-04, Vol.24 (4), p.294-302

2024

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Neuropeptide regulation of non-redundant ILC2 responses at barrier surfaces

Nature (London), 2022-11, Vol.611 (7937), p.787-793

2022

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Differentiation of type 1 ILCs from a common progenitor to all helper-like innate lymphoid cell lineages

The Transcription Factor GATA-3 Controls Cell Fate and Maintenance of Type 2 Innate Lymphoid Cells

Innate lymphoid cells as regulators of immunity, inflammation and tissue homeostasis

The neuropeptide NMU amplifies ILC2-driven allergic lung inflammation

Interleukin-33 Induces the Enzyme Tryptophan Hydroxylase 1 to Promote Inflammatory Group 2 Innate Lymphoid Cell-Mediated Immunity

The Transcription Factor T-bet Is Induced by IL-15 and Thymic Agonist Selection and Controls CD8αα+ Intraepithelial Lymphocyte Development

Anti-microbial Functions of Group 3 Innate Lymphoid Cells in Gut-Associated Lymphoid Tissues Are Regulated by G-Protein-Coupled Receptor 183

Lack of Type 2 Innate Lymphoid Cells Promotes a Type I-Driven Enhanced Immune Response in Contact Hypersensitivity

ILC3s restrict the dissemination of intestinal bacteria to safeguard liver regeneration after surgery

Tissue microenvironment dictates the fate and tumor-suppressive function of type 3 ILCs

Dynamic, Transient, and Robust Increase in the Innervation of the Inflamed Mucosa in Inflammatory Bowel Diseases

Innate lymphoid cells control signaling circuits to regulate tissue-specific immunity

Interleukin-22 protects intestinal stem cells against genotoxic stress

The transcription factor GATA3 controls cell fate and maintenance of type 2 innate lymphoid cells

The neuropeptide neuromedin U stimulates innate lymphoid cells and type 2 inflammation

β2-adrenergic receptor–mediated negative regulation of group 2 innate lymphoid cell responses

A T-bet gradient controls the fate and function of CCR6-RORγt+ innate lymphoid cells

Neuroimmune interactions in peripheral tissues

Neuro-Immune Circuits Regulate Immune Responses in Tissues and Organ Homeostasis

NK Cell Development in Times of Innate Lymphoid Cell Diversity

Neuro-immune-metabolism: The tripod system of homeostasis

Type 2 Immunity Regulates Dermal White Adipose Tissue Function

Non-redundant functions of group 2 innate lymphoid cells

Cooperation of ILC2s and TH2 cells in the expulsion of intestinal helminth parasites

Neuropeptide regulation of non-redundant ILC2 responses at barrier surfaces

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