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CDK4/6 and autophagy inhibitors synergistically induce senescence in Rb positive cytoplasmic cyclin E negative cancers

Nature communications, 2017-06, Vol.8 (1), p.15916-15916, Article 15916

2017

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Synthetic Lethality of PARP Inhibitors in Combination with MYC Blockade Is Independent of BRCA Status in Triple-Negative Breast Cancer

Cancer research (Chicago, Ill.), 2018-02, Vol.78 (3), p.742-757

2018

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Role of cell cycle in mediating sensitivity to radiotherapy

International Journal of Radiation Oncology, Biology, Physics, 2004-07, Vol.59 (4), p.928-942

2004

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Enhancer transcription reveals subtype-specific gene expression programs controlling breast cancer pathogenesis

Genome research, 2018-02, Vol.28 (2), p.159-170

2018

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Cell cycle arrest induces lipid droplet formation and confers ferroptosis resistance

Nature communications, 2024-01, Vol.15 (1), p.79-79, Article 79

2024

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CDK4/6 Inhibitors Sensitize Rb-positive Sarcoma Cells to Wee1 Kinase Inhibition through Reversible Cell-Cycle Arrest

Molecular cancer therapeutics, 2017-09, Vol.16 (9), p.1751-1764

2017

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Cyclin E Overexpression Sensitizes Triple-Negative Breast Cancer to Wee1 Kinase Inhibition

Clinical cancer research, 2018-12, Vol.24 (24), p.6594-6610

2018

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AXL Inhibition Suppresses the DNA Damage Response and Sensitizes Cells to PARP Inhibition in Multiple Cancers

Molecular cancer research, 2017-01, Vol.15 (1), p.45-58

2017

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Specific, reversible G1 arrest by UCN-01 in vivo provides cytostatic protection of normal cells against cytotoxic chemotherapy in breast cancer

British journal of cancer, 2020-03, Vol.122 (6), p.812-822

2020

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PAF-Wnt signaling-induced cell plasticity is required for maintenance of breast cancer cell stemness

Nature communications, 2016-02, Vol.7 (1), p.10633-10633, Article 10633

2016

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Cytoplasmic Cyclin E Mediates Resistance to Aromatase Inhibitors in Breast Cancer

Clinical cancer research, 2017-12, Vol.23 (23), p.7288-7300

2017

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PARP inhibitors as single agents and in combination therapy: the most promising treatment strategies in clinical trials for BRCA-mutant ovarian and triple-negative breast cancers

Expert opinion on investigational drugs, 2022-06, Vol.31 (6), p.607-631

2022

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Histone modification profiling in breast cancer cell lines highlights commonalities and differences among subtypes

BMC genomics, 2018-02, Vol.19 (1), p.150-11, Article 150

2018

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Cytoplasmic Cyclin E Predicts Recurrence in Patients with Breast Cancer

Clinical cancer research, 2017-06, Vol.23 (12), p.2991-3002

2017

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Synchronization of the cell cycle using Lovastatin

Cell cycle (Georgetown, Tex.), 2008-08, Vol.7 (15), p.2434-2440

2008

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LMW cyclin E and its novel catalytic partner CDK5 are therapeutic targets and prognostic biomarkers in salivary gland cancers

Oncogenesis (New York, NY), 2021-05, Vol.10 (5), p.40-40, Article 40

2021

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Autophagy : A Novel Mechanism of Synergistic Cytotoxicity between Doxorubicin and Roscovitine in a Sarcoma Model

Cancer research (Chicago, Ill.), 2008-10, Vol.68 (19), p.7966-7974

2008

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Integrative Analysis of Cyclin Protein Levels Identifies Cyclin B1 as a Classifier and Predictor of Outcomes in Breast Cancer

Clinical cancer research, 2009-06, Vol.15 (11), p.3654-3662

2009

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EVI1 splice variants modulate functional responses in ovarian cancer cells

Molecular oncology, 2013-06, Vol.7 (3), p.647-668

2013

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Atypical PKCι Contributes to Poor Prognosis through Loss of Apical-Basal Polarity and Cyclin E Overexpression in Ovarian Cancer

Proceedings of the National Academy of Sciences - PNAS, 2005-08, Vol.102 (35), p.12519-12524

2005

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LMW-E/CDK2 deregulates acinar morphogenesis, induces tumorigenesis, and associates with the activated b-Raf-ERK1/2-mTOR pathway in breast cancer patients

PLoS genetics, 2012-03, Vol.8 (3), p.e1002538

2012

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Farnesyl and geranylgeranyl transferase inhibitors induce G1 arrest by targeting the proteasome

Cancer research (Chicago, Ill.), 2006-01, Vol.66 (2), p.1040-1051

2006

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Lovastatin mediated G1 arrest in normal and tumor breast cells is through inhibition of CDK2 activity and redistribution of p21 and p27, independent of p53

Oncogene, 1998-11, Vol.17 (18), p.2393-2402

1998

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Cyclin E deregulation alters the biologic properties of ovarian cancer cells

Oncogene, 2004-04, Vol.23 (15), p.2648-2657

2004

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Lovastatin-Mediated G1 Arrest Is through Inhibition of the Proteasome, Independent of Hydroxymethyl Glutaryl-CoA Reductase

Proceedings of the National Academy of Sciences - PNAS, 1999-07, Vol.96 (14), p.7797-7802

1999

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CDK4/6 and autophagy inhibitors synergistically induce senescence in Rb positive cytoplasmic cyclin E negative cancers

Synthetic Lethality of PARP Inhibitors in Combination with MYC Blockade Is Independent of BRCA Status in Triple-Negative Breast Cancer

Role of cell cycle in mediating sensitivity to radiotherapy

Enhancer transcription reveals subtype-specific gene expression programs controlling breast cancer pathogenesis

Cell cycle arrest induces lipid droplet formation and confers ferroptosis resistance

CDK4/6 Inhibitors Sensitize Rb-positive Sarcoma Cells to Wee1 Kinase Inhibition through Reversible Cell-Cycle Arrest

Cyclin E Overexpression Sensitizes Triple-Negative Breast Cancer to Wee1 Kinase Inhibition

AXL Inhibition Suppresses the DNA Damage Response and Sensitizes Cells to PARP Inhibition in Multiple Cancers

Specific, reversible G1 arrest by UCN-01 in vivo provides cytostatic protection of normal cells against cytotoxic chemotherapy in breast cancer

PAF-Wnt signaling-induced cell plasticity is required for maintenance of breast cancer cell stemness

Cytoplasmic Cyclin E Mediates Resistance to Aromatase Inhibitors in Breast Cancer

PARP inhibitors as single agents and in combination therapy: the most promising treatment strategies in clinical trials for BRCA-mutant ovarian and triple-negative breast cancers

Histone modification profiling in breast cancer cell lines highlights commonalities and differences among subtypes

Cytoplasmic Cyclin E Predicts Recurrence in Patients with Breast Cancer

Synchronization of the cell cycle using Lovastatin

LMW cyclin E and its novel catalytic partner CDK5 are therapeutic targets and prognostic biomarkers in salivary gland cancers

Autophagy : A Novel Mechanism of Synergistic Cytotoxicity between Doxorubicin and Roscovitine in a Sarcoma Model

Integrative Analysis of Cyclin Protein Levels Identifies Cyclin B1 as a Classifier and Predictor of Outcomes in Breast Cancer

EVI1 splice variants modulate functional responses in ovarian cancer cells

Atypical PKCι Contributes to Poor Prognosis through Loss of Apical-Basal Polarity and Cyclin E Overexpression in Ovarian Cancer

LMW-E/CDK2 deregulates acinar morphogenesis, induces tumorigenesis, and associates with the activated b-Raf-ERK1/2-mTOR pathway in breast cancer patients

Farnesyl and geranylgeranyl transferase inhibitors induce G1 arrest by targeting the proteasome

Lovastatin mediated G1 arrest in normal and tumor breast cells is through inhibition of CDK2 activity and redistribution of p21 and p27, independent of p53

Cyclin E deregulation alters the biologic properties of ovarian cancer cells

Lovastatin-Mediated G1 Arrest Is through Inhibition of the Proteasome, Independent of Hydroxymethyl Glutaryl-CoA Reductase

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