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Comprehensive characterization of neutrophil genome topology

Genes & development, 2017-01, Vol.31 (2), p.141-153

2017

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Quantitative production of macrophages or neutrophils ex vivo using conditional Hoxb8

Nature methods, 2006-04, Vol.3 (4), p.287-293

2006

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Proteomics-based discovery of a novel, structurally unique, and developmentally regulated plasminogen receptor, Plg-RKT, a major regulator of cell surface plasminogen activation

Blood, 2010-02, Vol.115 (7), p.1319-1330

2010

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NSD1 PHD domains bind methylated H3K4 and H3K9 using interactions disrupted by point mutations in human sotos syndrome

Human mutation, 2011-03, Vol.32 (3), p.292-298

2011

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Persistent Transactivation by Meis1 Replaces Hox Function in Myeloid Leukemogenesis Models: Evidence for Co-Occupancy of Meis1-Pbx and Hox-Pbx Complexes on Promoters of Leukemia-Associated Genes

Molecular and Cellular Biology, 2006-05, Vol.26 (10), p.3902-3916

2006

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NUP98-NSD1 links H3K36 methylation to Hox-A gene activation and leukaemogenesis

Nature cell biology, 2007-07, Vol.9 (7), p.804-812

2007

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Specificity in Toll-like receptor signalling through distinct effector functions of TRAF3 and TRAF6

Nature, 2006-01, Vol.439 (7073), p.204-207

2006

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Meis1a Suppresses Differentiation by G-CSF and Promotes Proliferation by SCF: Potential Mechanisms of Cooperativity with Hoxa9 in Myeloid Leukemia

Proceedings of the National Academy of Sciences - PNAS, 2001-11, Vol.98 (23), p.13120-13125

2001

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Meis1 and pKnox1 Bind DNA Cooperatively with Pbx1 Utilizing an Interaction Surface Disrupted in Oncoprotein E2a-Pbx1

Proceedings of the National Academy of Sciences - PNAS, 1997-12, Vol.94 (26), p.14553-14558

1997

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An Induced Ets Repressor Complex Regulates Growth Arrest during Terminal Macrophage Differentiation

Cell, 2002-04, Vol.109 (2), p.169-180

2002

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Quantitative expansion of ES cell-derived myeloid progenitors capable of differentiating into macrophages

Journal of leukocyte biology, 2007-03, Vol.81 (3), p.711-719

2007

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TALE Homeodomain Proteins Regulate Gonadotropin-releasing Hormone Gene Expression Independently and via Interactions with Oct-1

The Journal of biological chemistry, 2004-07, Vol.279 (29), p.30287-30297

2004

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E2a/Pbx1 Induces the Rapid Proliferation of Stem Cell Factor-Dependent Murine Pro-T Cells That Cause Acute T-Lymphoid or Myeloid Leukemias in Mice

Molecular and Cellular Biology, 2004-02, Vol.24 (3), p.1256-1269

2004

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HoxB8 requires its Pbx-interaction motif to block differentiation of primary myeloid progenitors and of most cell line models of myeloid differentiation

Oncogene, 2001-09, Vol.20 (39), p.5440-5448

2001

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Meis1 programs transcription of FLT3 and cancer stem cell character, using a mechanism that requires interaction with Pbx and a novel function of the Meis1 C-terminus

Blood, 2005-07, Vol.106 (1), p.254-264

2005

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Nup98-HoxA9 immortalizes myeloid progenitors, enforces expression of Hoxa9, Hoxa7 and Meis1, and alters cytokine-specific responses in a manner similar to that induced by retroviral co-expression of Hoxa9 and Meis1

Oncogene, 2002-06, Vol.21 (27), p.4247-4256

2002

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Identification of key regulatory pathways of myeloid differentiation using an mESC-based karyotypically normal cell model

Blood, 2012-12, Vol.120 (24), p.4712-4719

2012

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Pre-B-Cell Transcription Factor 1 and Steroidogenic Factor 1 Synergistically Regulate Adrenocortical Growth and Steroidogenesis

Endocrinology (Philadelphia), 2007-02, Vol.148 (2), p.693-704

2007

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Hoxa9 Immortalizes a Granulocyte-Macrophage Colony-Stimulating Factor-Dependent Promyelocyte Capable of Biphenotypic Differentiation to Neutrophils or Macrophages, Independent of Enforced Meis Expression

Molecular and Cellular Biology, 2000-05, Vol.20 (9), p.3274-3285

2000

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E2a-Pbx1 induces aberrant expression of tissue-specific and developmentally regulated genes when expressed in NIH 3T3 fibroblasts

Molecular and Cellular Biology, 1997-03, Vol.17 (3), p.1503-1512

1997

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A conserved motif N-terminal to the DNA-binding domains of myogenic bHLH transcription factors mediates cooperative DNA binding with pbx-Meis1/Prep1

Nucleic acids research, 1999-09, Vol.27 (18), p.3752-3767

1999

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Estrogen-dependent E2a/Pbx1 myeloid cell lines exhibit conditional differentiation that can be arrested by other leukemic oncoproteins

Blood, 2001-10, Vol.98 (8), p.2308-2318

2001

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The Spt-Ada-Gcn5-acetyltransferase complex interaction motif of E2a is essential for a subset of transcriptional and oncogenic properties of E2a-Pbx1

Leukemia & lymphoma, 2009-01, Vol.50 (5), p.816-828

2009

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Heterodimerization of Hox proteins with Pbx1 and oncoprotein E2a-Pbx1 generates unique DNA-binding specificities at nucleotides predicted to contact the N-terminal arm of the Hox homeodomain - demonstration of Hox-dependent targeting of E2a-Pbx1 in vivo

Oncogene, 1997-01, Vol.14 (1), p.75-83

1997

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An inhibitory switch derepressed by pbx, hox, and Meis/Prep1 partners regulates DNA-binding by pbx1 and E2a-pbx1 and is dispensable for myeloid immortalization by E2a-pbx1

Oncogene, 1999-12, Vol.18 (56), p.8033-8043

1999

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Comprehensive characterization of neutrophil genome topology

Quantitative production of macrophages or neutrophils ex vivo using conditional Hoxb8

Proteomics-based discovery of a novel, structurally unique, and developmentally regulated plasminogen receptor, Plg-RKT, a major regulator of cell surface plasminogen activation

NSD1 PHD domains bind methylated H3K4 and H3K9 using interactions disrupted by point mutations in human sotos syndrome

Persistent Transactivation by Meis1 Replaces Hox Function in Myeloid Leukemogenesis Models: Evidence for Co-Occupancy of Meis1-Pbx and Hox-Pbx Complexes on Promoters of Leukemia-Associated Genes

NUP98-NSD1 links H3K36 methylation to Hox-A gene activation and leukaemogenesis

Specificity in Toll-like receptor signalling through distinct effector functions of TRAF3 and TRAF6

Meis1a Suppresses Differentiation by G-CSF and Promotes Proliferation by SCF: Potential Mechanisms of Cooperativity with Hoxa9 in Myeloid Leukemia

Meis1 and pKnox1 Bind DNA Cooperatively with Pbx1 Utilizing an Interaction Surface Disrupted in Oncoprotein E2a-Pbx1

An Induced Ets Repressor Complex Regulates Growth Arrest during Terminal Macrophage Differentiation

Quantitative expansion of ES cell-derived myeloid progenitors capable of differentiating into macrophages

TALE Homeodomain Proteins Regulate Gonadotropin-releasing Hormone Gene Expression Independently and via Interactions with Oct-1

E2a/Pbx1 Induces the Rapid Proliferation of Stem Cell Factor-Dependent Murine Pro-T Cells That Cause Acute T-Lymphoid or Myeloid Leukemias in Mice

HoxB8 requires its Pbx-interaction motif to block differentiation of primary myeloid progenitors and of most cell line models of myeloid differentiation

Meis1 programs transcription of FLT3 and cancer stem cell character, using a mechanism that requires interaction with Pbx and a novel function of the Meis1 C-terminus

Nup98-HoxA9 immortalizes myeloid progenitors, enforces expression of Hoxa9, Hoxa7 and Meis1, and alters cytokine-specific responses in a manner similar to that induced by retroviral co-expression of Hoxa9 and Meis1

Identification of key regulatory pathways of myeloid differentiation using an mESC-based karyotypically normal cell model

Pre-B-Cell Transcription Factor 1 and Steroidogenic Factor 1 Synergistically Regulate Adrenocortical Growth and Steroidogenesis

Hoxa9 Immortalizes a Granulocyte-Macrophage Colony-Stimulating Factor-Dependent Promyelocyte Capable of Biphenotypic Differentiation to Neutrophils or Macrophages, Independent of Enforced Meis Expression

E2a-Pbx1 induces aberrant expression of tissue-specific and developmentally regulated genes when expressed in NIH 3T3 fibroblasts

A conserved motif N-terminal to the DNA-binding domains of myogenic bHLH transcription factors mediates cooperative DNA binding with pbx-Meis1/Prep1

Estrogen-dependent E2a/Pbx1 myeloid cell lines exhibit conditional differentiation that can be arrested by other leukemic oncoproteins

The Spt-Ada-Gcn5-acetyltransferase complex interaction motif of E2a is essential for a subset of transcriptional and oncogenic properties of E2a-Pbx1

Heterodimerization of Hox proteins with Pbx1 and oncoprotein E2a-Pbx1 generates unique DNA-binding specificities at nucleotides predicted to contact the N-terminal arm of the Hox homeodomain - demonstration of Hox-dependent targeting of E2a-Pbx1 in vivo

An inhibitory switch derepressed by pbx, hox, and Meis/Prep1 partners regulates DNA-binding by pbx1 and E2a-pbx1 and is dispensable for myeloid immortalization by E2a-pbx1

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