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Tumor suppressors and cell metabolism: a recipe for cancer growth

Genes & development, 2009-03, Vol.23 (5), p.537-548

2009

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Metformin Antagonizes Cancer Cell Proliferation by Suppressing Mitochondrial-Dependent Biosynthesis

PLoS biology, 2015-12, Vol.13 (12), p.e1002309-e1002309

2015

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Posttranscriptional Control of T Cell Effector Function by Aerobic Glycolysis

Cell, 2013-06, Vol.153 (6), p.1239-1251

2013

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AMPK Is a Negative Regulator of the Warburg Effect and Suppresses Tumor Growth In Vivo

Cell metabolism, 2013-01, Vol.17 (1), p.113-124

2013

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Metabolic Plasticity as a Determinant of Tumor Growth and Metastasis

Cancer research (Chicago, Ill.), 2016-09, Vol.76 (18), p.5201-5208

2016

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The Energy Sensor AMPK Regulates T Cell Metabolic Adaptation and Effector Responses In Vivo

Immunity (Cambridge, Mass.), 2015-01, Vol.42 (1), p.41-54

2015

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The AMP-activated protein kinase (AMPK) and cancer: Many faces of a metabolic regulator

Cancer letters, 2015-01, Vol.356 (2), p.165-170

2015

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Itaconate Links Inhibition of Succinate Dehydrogenase with Macrophage Metabolic Remodeling and Regulation of Inflammation

Cell metabolism, 2016-07, Vol.24 (1), p.158-166

2016

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PDK1-Dependent Metabolic Reprogramming Dictates Metastatic Potential in Breast Cancer

Cell metabolism, 2015-10, Vol.22 (4), p.577-589

2015

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AMPK Maintains Cellular Metabolic Homeostasis through Regulation of Mitochondrial Reactive Oxygen Species

Cell reports (Cambridge), 2017-10, Vol.21 (1), p.1-9

2017

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Memory CD8+ T Cells Require Increased Concentrations of Acetate Induced by Stress for Optimal Function

Immunity (Cambridge, Mass.), 2016-06, Vol.44 (6), p.1312-1324

2016

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Toll-like receptor–induced changes in glycolytic metabolism regulate dendritic cell activation

Blood, 2010-06, Vol.115 (23), p.4742-4749

2010

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Glycolytic metabolism is essential for CCR7 oligomerization and dendritic cell migration

Nature communications, 2018-06, Vol.9 (1), p.2463-12, Article 2463

2018

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Immature Low-Density Neutrophils Exhibit Metabolic Flexibility that Facilitates Breast Cancer Liver Metastasis

Cell reports (Cambridge), 2019-06, Vol.27 (13), p.3902-3915.e6

2019

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Mitochondrial Phosphoenolpyruvate Carboxykinase Regulates Metabolic Adaptation and Enables Glucose-Independent Tumor Growth

Molecular cell, 2015-10, Vol.60 (2), p.195-207

2015

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Itaconate confers tolerance to late NLRP3 inflammasome activation

Cell reports (Cambridge), 2021-03, Vol.34 (10), p.108756-108756, Article 108756

2021

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CD8 memory T cells have a bioenergetic advantage that underlies their rapid recall ability

Proceedings of the National Academy of Sciences - PNAS, 2013-08, Vol.110 (35), p.14336-14341

2013

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Loss of the tumor suppressor LKB1 promotes metabolic reprogramming of cancer cells via HIF-1α

Proceedings of the National Academy of Sciences - PNAS, 2014-02, Vol.111 (7), p.2554-2559

2014

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Polarizing Macrophages through Reprogramming of Glucose Metabolism

Cell metabolism, 2012-06, Vol.15 (6), p.793-795

2012

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AMP-Activated Protein Kinase Induces a p53-Dependent Metabolic Checkpoint

Molecular cell, 2005-04, Vol.18 (3), p.283-293

2005

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The oncometabolite 2-hydroxyglutarate activates the mTOR signalling pathway

Nature communications, 2016-09, Vol.7 (1), p.12700-12, Article 12700

2016

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Signaling Kinase AMPK Activates Stress-Promoted Transcription via Histone H2B Phosphorylation

Science (American Association for the Advancement of Science), 2010-09, Vol.329 (5996), p.1201-1205

2010

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Amino Acids Fuel T Cell-Mediated Inflammation

Immunity (Cambridge, Mass.), 2014-05, Vol.40 (5), p.635-637

2014

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Tumor suppressors and cell metabolism: a recipe for cancer growth

Metformin Antagonizes Cancer Cell Proliferation by Suppressing Mitochondrial-Dependent Biosynthesis

Posttranscriptional Control of T Cell Effector Function by Aerobic Glycolysis

AMPK Is a Negative Regulator of the Warburg Effect and Suppresses Tumor Growth In Vivo

Metabolic Plasticity as a Determinant of Tumor Growth and Metastasis

The Energy Sensor AMPK Regulates T Cell Metabolic Adaptation and Effector Responses In Vivo

The AMP-activated protein kinase (AMPK) and cancer: Many faces of a metabolic regulator

Itaconate Links Inhibition of Succinate Dehydrogenase with Macrophage Metabolic Remodeling and Regulation of Inflammation

PDK1-Dependent Metabolic Reprogramming Dictates Metastatic Potential in Breast Cancer

AMPK Maintains Cellular Metabolic Homeostasis through Regulation of Mitochondrial Reactive Oxygen Species

Memory CD8+ T Cells Require Increased Concentrations of Acetate Induced by Stress for Optimal Function

Toll-like receptor–induced changes in glycolytic metabolism regulate dendritic cell activation

Glycolytic metabolism is essential for CCR7 oligomerization and dendritic cell migration

Immature Low-Density Neutrophils Exhibit Metabolic Flexibility that Facilitates Breast Cancer Liver Metastasis

Mitochondrial Phosphoenolpyruvate Carboxykinase Regulates Metabolic Adaptation and Enables Glucose-Independent Tumor Growth

Itaconate confers tolerance to late NLRP3 inflammasome activation

CD8 memory T cells have a bioenergetic advantage that underlies their rapid recall ability

Loss of the tumor suppressor LKB1 promotes metabolic reprogramming of cancer cells via HIF-1α

Polarizing Macrophages through Reprogramming of Glucose Metabolism

The eEF2 Kinase Confers Resistance to Nutrient Deprivation by Blocking Translation Elongation

AMP-Activated Protein Kinase Induces a p53-Dependent Metabolic Checkpoint

The oncometabolite 2-hydroxyglutarate activates the mTOR signalling pathway

Signaling Kinase AMPK Activates Stress-Promoted Transcription via Histone H2B Phosphorylation

Amino Acids Fuel T Cell-Mediated Inflammation

Carnitine palmitoyltransferase 1C promotes cell survival and tumor growth under conditions of metabolic stress

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