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Tumor suppressors and cell metabolism: a recipe for cancer growth

Genes & development, 2009-03, Vol.23 (5), p.537-548

2009

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Metformin Antagonizes Cancer Cell Proliferation by Suppressing Mitochondrial-Dependent Biosynthesis

PLoS biology, 2015-12, Vol.13 (12), p.e1002309-e1002309

2015

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Metabolic Plasticity as a Determinant of Tumor Growth and Metastasis

Cancer research (Chicago, Ill.), 2016-09, Vol.76 (18), p.5201-5208

2016

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Posttranscriptional Control of T Cell Effector Function by Aerobic Glycolysis

Cell, 2013-06, Vol.153 (6), p.1239-1251

2013

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AMPK Is a Negative Regulator of the Warburg Effect and Suppresses Tumor Growth In Vivo

Cell metabolism, 2013-01, Vol.17 (1), p.113-124

2013

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The Energy Sensor AMPK Regulates T Cell Metabolic Adaptation and Effector Responses In Vivo

Immunity (Cambridge, Mass.), 2015-01, Vol.42 (1), p.41-54

2015

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The AMP-activated protein kinase (AMPK) and cancer: Many faces of a metabolic regulator

Cancer letters, 2015-01, Vol.356 (2), p.165-170

2015

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Itaconate Links Inhibition of Succinate Dehydrogenase with Macrophage Metabolic Remodeling and Regulation of Inflammation

Cell metabolism, 2016-07, Vol.24 (1), p.158-166

2016

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PDK1-Dependent Metabolic Reprogramming Dictates Metastatic Potential in Breast Cancer

Cell metabolism, 2015-10, Vol.22 (4), p.577-589

2015

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AMPK Maintains Cellular Metabolic Homeostasis through Regulation of Mitochondrial Reactive Oxygen Species

Cell reports (Cambridge), 2017-10, Vol.21 (1), p.1-9

2017

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Memory CD8+ T Cells Require Increased Concentrations of Acetate Induced by Stress for Optimal Function

Immunity (Cambridge, Mass.), 2016-06, Vol.44 (6), p.1312-1324

2016

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Glycolytic metabolism is essential for CCR7 oligomerization and dendritic cell migration

Nature communications, 2018-06, Vol.9 (1), p.2463-12, Article 2463

2018

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Toll-like receptor–induced changes in glycolytic metabolism regulate dendritic cell activation

Blood, 2010-06, Vol.115 (23), p.4742-4749

2010

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Immature Low-Density Neutrophils Exhibit Metabolic Flexibility that Facilitates Breast Cancer Liver Metastasis

Cell reports (Cambridge), 2019-06, Vol.27 (13), p.3902-3915.e6

2019

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Mitochondrial Phosphoenolpyruvate Carboxykinase Regulates Metabolic Adaptation and Enables Glucose-Independent Tumor Growth

Molecular cell, 2015-10, Vol.60 (2), p.195-207

2015

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Itaconate confers tolerance to late NLRP3 inflammasome activation

Cell reports (Cambridge), 2021-03, Vol.34 (10), p.108756-108756, Article 108756

2021

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Polarizing Macrophages through Reprogramming of Glucose Metabolism

Cell metabolism, 2012-06, Vol.15 (6), p.793-795

2012

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Loss of the tumor suppressor LKB1 promotes metabolic reprogramming of cancer cells via HIF-1α

Proceedings of the National Academy of Sciences - PNAS, 2014-02, Vol.111 (7), p.2554-2559

2014

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CD8 memory T cells have a bioenergetic advantage that underlies their rapid recall ability

Proceedings of the National Academy of Sciences - PNAS, 2013-08, Vol.110 (35), p.14336-14341

2013

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AMP-Activated Protein Kinase Induces a p53-Dependent Metabolic Checkpoint

Molecular cell, 2005-04, Vol.18 (3), p.283-293

2005

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The oncometabolite 2-hydroxyglutarate activates the mTOR signalling pathway

Nature communications, 2016-09, Vol.7 (1), p.12700-12, Article 12700

2016

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Signaling Kinase AMPK Activates Stress-Promoted Transcription via Histone H2B Phosphorylation

Science (American Association for the Advancement of Science), 2010-09, Vol.329 (5996), p.1201-1205

2010

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Amino Acids Fuel T Cell-Mediated Inflammation

Immunity (Cambridge, Mass.), 2014-05, Vol.40 (5), p.635-637

2014

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Tumor suppressors and cell metabolism: a recipe for cancer growth

Metformin Antagonizes Cancer Cell Proliferation by Suppressing Mitochondrial-Dependent Biosynthesis

Metabolic Plasticity as a Determinant of Tumor Growth and Metastasis

Posttranscriptional Control of T Cell Effector Function by Aerobic Glycolysis

AMPK Is a Negative Regulator of the Warburg Effect and Suppresses Tumor Growth In Vivo

The Energy Sensor AMPK Regulates T Cell Metabolic Adaptation and Effector Responses In Vivo

The AMP-activated protein kinase (AMPK) and cancer: Many faces of a metabolic regulator

Itaconate Links Inhibition of Succinate Dehydrogenase with Macrophage Metabolic Remodeling and Regulation of Inflammation

PDK1-Dependent Metabolic Reprogramming Dictates Metastatic Potential in Breast Cancer

AMPK Maintains Cellular Metabolic Homeostasis through Regulation of Mitochondrial Reactive Oxygen Species

Memory CD8+ T Cells Require Increased Concentrations of Acetate Induced by Stress for Optimal Function

Glycolytic metabolism is essential for CCR7 oligomerization and dendritic cell migration

Toll-like receptor–induced changes in glycolytic metabolism regulate dendritic cell activation

Immature Low-Density Neutrophils Exhibit Metabolic Flexibility that Facilitates Breast Cancer Liver Metastasis

Mitochondrial Phosphoenolpyruvate Carboxykinase Regulates Metabolic Adaptation and Enables Glucose-Independent Tumor Growth

Itaconate confers tolerance to late NLRP3 inflammasome activation

Polarizing Macrophages through Reprogramming of Glucose Metabolism

Loss of the tumor suppressor LKB1 promotes metabolic reprogramming of cancer cells via HIF-1α

CD8 memory T cells have a bioenergetic advantage that underlies their rapid recall ability

The eEF2 Kinase Confers Resistance to Nutrient Deprivation by Blocking Translation Elongation

AMP-Activated Protein Kinase Induces a p53-Dependent Metabolic Checkpoint

The oncometabolite 2-hydroxyglutarate activates the mTOR signalling pathway

Signaling Kinase AMPK Activates Stress-Promoted Transcription via Histone H2B Phosphorylation

Amino Acids Fuel T Cell-Mediated Inflammation

Carnitine palmitoyltransferase 1C promotes cell survival and tumor growth under conditions of metabolic stress

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