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Understanding the role of the perivascular space in cerebral small vessel disease

Cardiovascular research, 2018-09, Vol.114 (11), p.1462-1473

2018

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Perivascular spaces in the brain: anatomy, physiology and pathology

Nature reviews. Neurology, 2020-03, Vol.16 (3), p.137-153

2020

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Perturbations of the cerebrovascular matrisome: A convergent mechanism in small vessel disease of the brain?

Journal of Cerebral Blood Flow & Metabolism, 2016-01, Vol.36 (1), p.143-157

2016

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Consensus statement for diagnosis of subcortical small vessel disease

Journal of Cerebral Blood Flow & Metabolism, 2016-01, Vol.36 (1), p.6-25

2016

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CADASIL and CARASIL

Brain pathology (Zurich, Switzerland), 2014-09, Vol.24 (5), p.525-544

2014

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Cerebrovascular dysfunction and microcirculation rarefaction precede white matter lesions in a mouse genetic model of cerebral ischemic small vessel disease

The Journal of clinical investigation, 2010-02, Vol.120 (2), p.433-445

2010

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Prospects for Diminishing the Impact of Nonamyloid Small-Vessel Diseases of the Brain

Annual review of pharmacology and toxicology, 2020-01, Vol.60 (1), p.437-456

Joutel, Anne

2020

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Neurovascular coupling and CO2 interrogate distinct vascular regulations

Nature communications, 2024-09, Vol.15 (1), p.7635-12, Article 7635

2024

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Reducing Timp3 or vitronectin ameliorates disease manifestations in CADASIL mice

Annals of neurology, 2016-03, Vol.79 (3), p.387-403

2016

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Pathogenesis of CADASIL

BioEssays, 2011-01, Vol.33 (1), p.73-80

Joutel, Anne

2011

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Reducing Hypermuscularization of the Transitional Segment Between Arterioles and Capillaries Protects Against Spontaneous Intracerebral Hemorrhage

Circulation (New York, N.Y.), 2020-06, Vol.141 (25), p.2078-2094

2020

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Pathogenesis of white matter changes in cerebral small vessel diseases: beyond vessel-intrinsic mechanisms

Clinical science (1979), 2017-04, Vol.131 (8), p.635-651

2017

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Pathogenesis of CADASIL: Transgenic and knock‐out mice to probe function and dysfunction of the mutated gene, Notch3, in the cerebrovasculature

BioEssays, 2011, Vol.33 (1), p.73-80

Joutel, Anne

2011

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Potassium channelopathy-like defect underlies early-stage cerebrovascular dysfunction in a genetic model of small vessel disease

Proceedings of the National Academy of Sciences - PNAS, 2015-02, Vol.112 (7), p.E796-E805

2015

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CADASIL mutations sensitize the brain to ischemia via spreading depolarizations and abnormal extracellular potassium homeostasis

The Journal of clinical investigation, 2022-04, Vol.132 (8), p.1-12

2022

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Congruence between NOTCH3 mutations and GOM in 131 CADASIL patients

Brain (London, England : 1878), 2009-04, Vol.132 (4), p.933-939

2009

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Notch3 is required for arterial identity and maturation of vascular smooth muscle cells

Genes & development, 2004-11, Vol.18 (22), p.2730-2735

2004

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Blood brain barrier leakage is not a consistent feature of white matter lesions in CADASIL

Acta neuropathologica communications, 2019-11, Vol.7 (1), p.187-187, Article 187

2019

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The NOTCH3ECD cascade hypothesis of cerebral autosomal‐dominant arteriopathy with subcortical infarcts and leukoencephalopathy disease

Neurology and clinical neuroscience, 2015-01, Vol.3 (1), p.1-6

Joutel, Anne

2015

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Prostaglandin E2 Dilates Intracerebral Arterioles When Applied to Capillaries: Implications for Small Vessel Diseases

Frontiers in aging neuroscience, 2021-08, Vol.13, p.695965-695965

2021

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Mechanistic insights into a TIMP3-sensitive pathway constitutively engaged in the regulation of cerebral hemodynamics

eLife, 2016-08, Vol.5

2016

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Vascular Cognitive Impairment and Dementia

Journal of the American College of Cardiology, 2019-07, Vol.73 (25), p.3326-3344

2019

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The ectodomain of the Notch3 receptor accumulates within the cerebrovasculature of CADASIL patients

The Journal of clinical investigation, 2000-03, Vol.105 (5), p.597-605

2000

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Notch3 Is Essential for Regulation of the Renal Vascular Tone

Hypertension (Dallas, Tex. 1979), 2011-06, Vol.57 (6), p.1176-1182

2011

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Transcriptome analysis for Notch3 target genes identifies Grip2 as a novel regulator of myogenic response in the cerebrovasculature

Arteriosclerosis, thrombosis, and vascular biology, 2013-01, Vol.33 (1), p.76-86

2013

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Understanding the role of the perivascular space in cerebral small vessel disease

Perivascular spaces in the brain: anatomy, physiology and pathology

Perturbations of the cerebrovascular matrisome: A convergent mechanism in small vessel disease of the brain?

Consensus statement for diagnosis of subcortical small vessel disease

CADASIL and CARASIL

Cerebrovascular dysfunction and microcirculation rarefaction precede white matter lesions in a mouse genetic model of cerebral ischemic small vessel disease

Prospects for Diminishing the Impact of Nonamyloid Small-Vessel Diseases of the Brain

Neurovascular coupling and CO2 interrogate distinct vascular regulations

Reducing Timp3 or vitronectin ameliorates disease manifestations in CADASIL mice

Pathogenesis of CADASIL

Reducing Hypermuscularization of the Transitional Segment Between Arterioles and Capillaries Protects Against Spontaneous Intracerebral Hemorrhage

Pathogenesis of white matter changes in cerebral small vessel diseases: beyond vessel-intrinsic mechanisms

Pathogenesis of CADASIL: Transgenic and knock‐out mice to probe function and dysfunction of the mutated gene, Notch3, in the cerebrovasculature

Potassium channelopathy-like defect underlies early-stage cerebrovascular dysfunction in a genetic model of small vessel disease

CADASIL mutations sensitize the brain to ischemia via spreading depolarizations and abnormal extracellular potassium homeostasis

Congruence between NOTCH3 mutations and GOM in 131 CADASIL patients

Notch3 is required for arterial identity and maturation of vascular smooth muscle cells

Blood brain barrier leakage is not a consistent feature of white matter lesions in CADASIL

The NOTCH3ECD cascade hypothesis of cerebral autosomal‐dominant arteriopathy with subcortical infarcts and leukoencephalopathy disease

Prostaglandin E2 Dilates Intracerebral Arterioles When Applied to Capillaries: Implications for Small Vessel Diseases

Mechanistic insights into a TIMP3-sensitive pathway constitutively engaged in the regulation of cerebral hemodynamics

Vascular Cognitive Impairment and Dementia

The ectodomain of the Notch3 receptor accumulates within the cerebrovasculature of CADASIL patients

Notch3 Is Essential for Regulation of the Renal Vascular Tone

Transcriptome analysis for Notch3 target genes identifies Grip2 as a novel regulator of myogenic response in the cerebrovasculature

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