UB Paderborn / Katalog / Suche / in CDI

Sortiert nach

Einfacher Volltextzugriff von (fast) überall

Installieren Sie die LibKey Nomad-Browsererweiterung und erhalten Sie von vielen Webseiten einfachen Zugriff auf lizenzierte Volltexte oder Open Access Publikationen. Mehr erfahren...

Potential roles of gut microbiome and metabolites in modulating ALS in mice

Nature (London), 2019-08, Vol.572 (7770), p.474-480

2019

Volltextzugriff (PDF)

Macrophage migration inhibitory factor: A multifaceted cytokine implicated in multiple neurological diseases

Experimental neurology, 2018-03, Vol.301 (Pt B), p.83-91

2018

Volltextzugriff (PDF)

AAV2/9-mediated overexpression of MIF inhibits SOD1 misfolding, delays disease onset, and extends survival in mouse models of ALS

Proceedings of the National Academy of Sciences - PNAS, 2019-07, Vol.116 (29), p.14755-14760

2019

Volltextzugriff (PDF)

MIF homolog d-dopachrome tautomerase (D-DT/MIF-2) does not inhibit accumulation and toxicity of misfolded SOD1

Scientific reports, 2022-06, Vol.12 (1), p.9570-9570, Article 9570

2022

Volltextzugriff (PDF)

Misfolded Mutant SOD1 Directly Inhibits VDAC1 Conductance in a Mouse Model of Inherited ALS

Neuron (Cambridge, Mass.), 2010-08, Vol.67 (4), p.575-587

2010

Volltextzugriff (PDF)

CD4 T-cell aging exacerbates neuroinflammation in a late-onset mouse model of amyotrophic lateral sclerosis

Journal of neuroinflammation, 2024-01, Vol.21 (1), p.17-17, Article 17

2024

Volltextzugriff (PDF)

Cu/Zn-superoxide dismutase and wild-type like fALS SOD1 mutants produce cytotoxic quantities of H2O2 via cysteine-dependent redox short-circuit

Scientific reports, 2019-07, Vol.9 (1), p.1-13, Article 10826

2019

Volltextzugriff (PDF)

All Roads Lead to Rome: Different Molecular Players Converge to Common Toxic Pathways in Neurodegeneration

Cells (Basel, Switzerland), 2021-09, Vol.10 (9), p.2438

2021

Volltextzugriff (PDF)

Endogenous macrophage migration inhibitory factor reduces the accumulation and toxicity of misfolded SOD1 in a mouse model of ALS

Proceedings of the National Academy of Sciences - PNAS, 2016-09, Vol.113 (36), p.10198-10203

2016

Volltextzugriff (PDF)

Targeting the Mitochondrial Protein VDAC1 as a Potential Therapeutic Strategy in ALS

International journal of molecular sciences, 2022-09, Vol.23 (17), p.9946

2022

Volltextzugriff (PDF)

Early upregulation of cytosolic phospholipase A2α in motor neurons is induced by misfolded SOD1 in a mouse model of amyotrophic lateral sclerosis

Journal of neuroinflammation, 2021-11, Vol.18 (1), p.1-274, Article 274

2021

Volltextzugriff (PDF)

Macrophage Migration Inhibitory Factor as a Chaperone Inhibiting Accumulation of Misfolded SOD1

Neuron (Cambridge, Mass.), 2015-04, Vol.86 (1), p.218-232

2015

Volltextzugriff (PDF)

VDAC1 N-terminus is essential both for apoptosis and the protective effect of anti-apoptotic proteins

Journal of cell science, 2009-06, Vol.122 (11), p.1906-1916

2009

Volltextzugriff (PDF)

Empty mesoporous silica particles significantly delay disease progression and extend survival in a mouse model of ALS

Scientific reports, 2020-11, Vol.10 (1), p.20675-20675, Article 20675

2020

Volltextzugriff (PDF)

MIF inhibits the formation and toxicity of misfolded SOD1 amyloid aggregates: implications for familial ALS

Cell death & disease, 2018-01, Vol.9 (2), p.107-14, Article 107

2018

Volltextzugriff (PDF)

4-Phenylbutyric Acid (4-PBA) Derivatives Prevent SOD1 Amyloid Aggregation In Vitro with No Effect on Disease Progression in SOD1-ALS Mice

International journal of molecular sciences, 2022-08, Vol.23 (16), p.9403

2022

Volltextzugriff (PDF)

ALS-linked mutant superoxide dismutase 1 (SOD1) alters mitochondrial protein composition and decreases protein import

Proceedings of the National Academy of Sciences - PNAS, 2010-12, Vol.107 (49), p.21146-21151

2010

Volltextzugriff (PDF)

A VDAC1-Derived N-Terminal Peptide Inhibits Mutant SOD1-VDAC1 Interactions and Toxicity in the SOD1 Model of ALS

Frontiers in cellular neuroscience, 2019-08, Vol.13, p.346

2019

Volltextzugriff (PDF)

Therapeutic Potential of Phytocannabinoid Cannabigerol for Multiple Sclerosis: Modulation of Microglial Activation In Vitro and In Vivo

Biomolecules (Basel, Switzerland), 2023-02, Vol.13 (2), p.376

2023

Volltextzugriff (PDF)

Oligomeric states of the voltage-dependent anion channel and cytochrome c release from mitochondria

Biochemical journal, 2005-02, Vol.386 (Pt 1), p.73-83

2005

Volltextzugriff (PDF)

Localization of the voltage-dependent anion channel-1 Ca2+ -binding sites

Cell calcium (Edinburgh), 2007-03, Vol.41 (3), p.235-244

2007

Volltextzugriff (PDF)

Mapping the ruthenium red-binding site of the voltage-dependent anion channel-1

Cell calcium (Edinburgh), 2008-02, Vol.43 (2), p.196-204

2008

Volltextzugriff (PDF)

Translational challenges in amyotrophic lateral sclerosis therapy with macrophage migration inhibitory factor

Neural regeneration research, 2024-09

2024

Volltextzugriff (PDF)

Blocking an epitope of misfolded SOD1 ameliorates disease phenotype in a model of amyotrophic lateral sclerosis

Brain (London, England : 1878), 2023-11, Vol.146 (11), p.4594-4607

2023

Volltextzugriff (PDF)

Hexokinase-I Protection against Apoptotic Cell Death Is Mediated via Interaction with the Voltage-dependent Anion Channel-1: MAPPING THE SITE OF BINDING

The Journal of biological chemistry, 2008-05, Vol.283 (19), p.13482-13490

2008

Volltextzugriff (PDF)

1 – 25 von 46

Suche als RSS-Feed

Suchergebnisse filtern

Filter anzeigen

Nur zeigen

Geprüft (Peer-Review)
(46)
Mit Volltextzugriff
(44)

Publikationsform

Artikel
(46)

Sprache

Englisch
(46)

Erscheinungsjahr

n.n

Thema

Science & Technology
(34)
Life Sciences & Biomedicine
(26)
Animals
(24)
Amyotrophic Lateral Sclerosis
(22)
Humans
(19)
Mice
(18)
Spinal Cord
(14)
Amyotrophic Lateral Sclerosis - Metabolism
(13)
Protein Folding
(13)
Als
(12)
Biochemistry & Molecular Biology
(12)
Mitochondria
(12)
Mutation
(12)
Neurodegenerative Diseases
(12)
Superoxide Dismutase
(12)
Superoxide Dismutase-1 - Metabolism
(12)
Motor Neurons
(11)
Disease Models, Animal
(10)
Proteins
(10)
Neurosciences
(9)

15 weitere anzeigen...

Weniger anzeigen...

Neu hinzugefügt

... seit 30 Tagen
(1)

Menü

Weitere Dienste

Einstellungen

Potential roles of gut microbiome and metabolites in modulating ALS in mice

Macrophage migration inhibitory factor: A multifaceted cytokine implicated in multiple neurological diseases

AAV2/9-mediated overexpression of MIF inhibits SOD1 misfolding, delays disease onset, and extends survival in mouse models of ALS

MIF homolog d-dopachrome tautomerase (D-DT/MIF-2) does not inhibit accumulation and toxicity of misfolded SOD1

Misfolded Mutant SOD1 Directly Inhibits VDAC1 Conductance in a Mouse Model of Inherited ALS

CD4 T-cell aging exacerbates neuroinflammation in a late-onset mouse model of amyotrophic lateral sclerosis

Cu/Zn-superoxide dismutase and wild-type like fALS SOD1 mutants produce cytotoxic quantities of H2O2 via cysteine-dependent redox short-circuit

All Roads Lead to Rome: Different Molecular Players Converge to Common Toxic Pathways in Neurodegeneration

Endogenous macrophage migration inhibitory factor reduces the accumulation and toxicity of misfolded SOD1 in a mouse model of ALS

Targeting the Mitochondrial Protein VDAC1 as a Potential Therapeutic Strategy in ALS

Early upregulation of cytosolic phospholipase A2α in motor neurons is induced by misfolded SOD1 in a mouse model of amyotrophic lateral sclerosis

Macrophage Migration Inhibitory Factor as a Chaperone Inhibiting Accumulation of Misfolded SOD1

VDAC1 N-terminus is essential both for apoptosis and the protective effect of anti-apoptotic proteins

Empty mesoporous silica particles significantly delay disease progression and extend survival in a mouse model of ALS

MIF inhibits the formation and toxicity of misfolded SOD1 amyloid aggregates: implications for familial ALS

4-Phenylbutyric Acid (4-PBA) Derivatives Prevent SOD1 Amyloid Aggregation In Vitro with No Effect on Disease Progression in SOD1-ALS Mice

ALS-linked mutant superoxide dismutase 1 (SOD1) alters mitochondrial protein composition and decreases protein import

A VDAC1-Derived N-Terminal Peptide Inhibits Mutant SOD1-VDAC1 Interactions and Toxicity in the SOD1 Model of ALS

Therapeutic Potential of Phytocannabinoid Cannabigerol for Multiple Sclerosis: Modulation of Microglial Activation In Vitro and In Vivo

Oligomeric states of the voltage-dependent anion channel and cytochrome c release from mitochondria

Localization of the voltage-dependent anion channel-1 Ca2+ -binding sites

Mapping the ruthenium red-binding site of the voltage-dependent anion channel-1

Translational challenges in amyotrophic lateral sclerosis therapy with macrophage migration inhibitory factor

Blocking an epitope of misfolded SOD1 ameliorates disease phenotype in a model of amyotrophic lateral sclerosis

Hexokinase-I Protection against Apoptotic Cell Death Is Mediated via Interaction with the Voltage-dependent Anion Channel-1: MAPPING THE SITE OF BINDING

Suchergebnisse filtern