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Prevalence of the Congenital Long-QT Syndrome

Circulation (New York, N.Y.), 2009-11, Vol.120 (18), p.1761-1767

2009

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Prevalence of long-QT syndrome gene variants in sudden infant death syndrome

Circulation (New York, N.Y.), 2007-01, Vol.115 (3), p.361-367

2007

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Long QT syndrome-associated mutations in intrauterine fetal death

JAMA : the journal of the American Medical Association, 2013-04, Vol.309 (14), p.1473

2013

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Propranolol prevents life-threatening arrhythmias in LQT3 transgenic mice: Implications for the clinical management of LQT3 patients

Heart rhythm, 2014, Vol.11 (1), p.126-132

2014

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Cardiac sodium channel dysfunction in sudden infant death syndrome

Circulation (New York, N.Y.), 2007-01, Vol.115 (3), p.368-376

2007

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KCNH2-K897T is a genetic modifier of latent congenital long-QT syndrome

Circulation (New York, N.Y.), 2005-08, Vol.112 (9), p.1251-1258

2005

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Torsades de pointes following acute myocardial infarction: Evidence for a deadly link with a common genetic variant

Heart rhythm, 2012-07, Vol.9 (7), p.1104-1112

2012

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Long-QT Syndrome: From Genetics to Management

Circulation. Arrhythmia and electrophysiology, 2012-08, Vol.5 (4), p.868-877

2012

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The Role of the Cardiac Sodium Channel in Perinatal Early Infant Mortality

Cardiac electrophysiology clinics, 2014-12, Vol.6 (4), p.749-759

2014

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A KCNH2 branch point mutation causing aberrant splicing contributes to an explanation of genotype-negative long QT syndrome

Heart rhythm, 2009-02, Vol.6 (2), p.212-218

2009

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Genetic association study of QT interval highlights role for calcium signaling pathways in myocardial repolarization

Nature genetics, 2014-08, Vol.46 (8), p.826-836

2014

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NOS1AP Is a Genetic Modifier of the Long-QT Syndrome

Circulation (New York, N.Y.), 2009-10, Vol.120 (17), p.1657-1663

2009

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Sudden Infant Death Syndrome and Cardiac Channelopathies: From Mechanisms to Prevention of Avoidable Tragedies

Cardiogenetics, 2011-12, Vol.1 (11), p.e6-e6

2011

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Inherited Cardiac Arrhythmia Syndrome: Role of Potassium Channels

Cardiac electrophysiology clinics, 2011-03, Vol.3 (1), p.113-124

2011

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The Common Long-QT Syndrome Mutation KCNQ1/A341V Causes Unusually Severe Clinical Manifestations in Patients With Different Ethnic Backgrounds : Toward a Mutation-Specific Risk Stratification

Circulation (New York, N.Y.), 2007-11, Vol.116 (21), p.2366-2375

2007

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Cardiac potassium channel dysfunction in sudden infant death syndrome

Journal of molecular and cellular cardiology, 2008-03, Vol.44 (3), p.571-581

2008

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Abstract 2927: Long QT Syndrome as a Cause of Stillbirths

Circulation (New York, N.Y.), 2007-10, Vol.116 (suppl_16)

2007

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KCNH2 -K897T Polymorphism Increases the Risk of Life-Threatening Arrhythmias Following Acute Myocardial Infarction

Heart rhythm, 2010, Vol.7 (11), p.1720-1720

2010

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How many cases of sudden infant death syndrome are due to the long QT syndrome?

Heart rhythm, 2005-05, Vol.2 (5), p.S110-S111

2005

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AB1-5

Heart rhythm, 2006-05, Vol.3 (5), p.S2

2006

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AB1-5: Gain of function KCNQ1 mutation associated with sudden infant death syndrome

Heart rhythm, 2006, Vol.3 (5), p.S2-S2

2006

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Long QT syndrome with cardiac arrest and transient short QT due to a novel KCNH2 mutation causing both loss and gain of function

Heart rhythm, 2005-05, Vol.2 (5), p.S145-S145

2005

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Common KCNH2 Polymorphism (K897T) as a genetic modifier of congenital long QT syndrome

Heart rhythm, 2005-05, Vol.2 (5), p.S111-S111

2005

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PREDESTINATION: PRimary vEntricular fibrillation and suDden dEath during a firST myocardIal iNfArcTION: Genetic Basis

New Diagnostic, Therapeutic and Organizational Strategies for Acute Coronary Syndromes Patients, p.85-96

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Honey bee colony loss linked to parasites, pesticides and extreme weather across the United States

Scientific reports, 2022-12, Vol.12 (1), p.20787-13, Article 20787

2022

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Prevalence of the Congenital Long-QT Syndrome

Prevalence of long-QT syndrome gene variants in sudden infant death syndrome

Long QT syndrome-associated mutations in intrauterine fetal death

Propranolol prevents life-threatening arrhythmias in LQT3 transgenic mice: Implications for the clinical management of LQT3 patients

Cardiac sodium channel dysfunction in sudden infant death syndrome

KCNH2-K897T is a genetic modifier of latent congenital long-QT syndrome

Torsades de pointes following acute myocardial infarction: Evidence for a deadly link with a common genetic variant

Long-QT Syndrome: From Genetics to Management

The Role of the Cardiac Sodium Channel in Perinatal Early Infant Mortality

A KCNH2 branch point mutation causing aberrant splicing contributes to an explanation of genotype-negative long QT syndrome

Genetic association study of QT interval highlights role for calcium signaling pathways in myocardial repolarization

NOS1AP Is a Genetic Modifier of the Long-QT Syndrome

Sudden Infant Death Syndrome and Cardiac Channelopathies: From Mechanisms to Prevention of Avoidable Tragedies

Inherited Cardiac Arrhythmia Syndrome: Role of Potassium Channels

The Common Long-QT Syndrome Mutation KCNQ1/A341V Causes Unusually Severe Clinical Manifestations in Patients With Different Ethnic Backgrounds : Toward a Mutation-Specific Risk Stratification

Cardiac potassium channel dysfunction in sudden infant death syndrome

Abstract 2927: Long QT Syndrome as a Cause of Stillbirths

KCNH2 -K897T Polymorphism Increases the Risk of Life-Threatening Arrhythmias Following Acute Myocardial Infarction

How many cases of sudden infant death syndrome are due to the long QT syndrome?

AB1-5

AB1-5: Gain of function KCNQ1 mutation associated with sudden infant death syndrome

Long QT syndrome with cardiac arrest and transient short QT due to a novel KCNH2 mutation causing both loss and gain of function

Common KCNH2 Polymorphism (K897T) as a genetic modifier of congenital long QT syndrome

PREDESTINATION: PRimary vEntricular fibrillation and suDden dEath during a firST myocardIal iNfArcTION: Genetic Basis

Honey bee colony loss linked to parasites, pesticides and extreme weather across the United States

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