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Mineralocorticoid and angiotensin II type 1 receptors in the subfornical organ mediate angiotensin II – Induced hypothalamic reactive oxygen species and hypertension

Neuroscience, 2016-08, Vol.329, p.112-121

2016

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Central neuronal activation and pressor responses induced by circulating ANG II: role of the brain aldosterone-"ouabain" pathway

American journal of physiology. Heart and circulatory physiology, 2010-08, Vol.299 (2), p.H422-H430

2010

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Knockdown of mineralocorticoid or angiotensin II type 1 receptor gene expression in the paraventricular nucleus prevents angiotensin II hypertension in rats

The Journal of physiology, 2014-08, Vol.592 (16), p.3523-3536

2014

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Neuroendocrine humoral and vascular components in the pressor pathway for brain angiotensin II: a new axis in long term blood pressure control

PloS one, 2014-10, Vol.9 (9), p.e108916-e108916

2014

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Role of central nervous system aldosterone synthase and mineralocorticoid receptors in salt-induced hypertension in Dahl salt-sensitive rats

American journal of physiology. Regulatory, integrative and comparative physiology, 2009-04, Vol.296 (4), p.R994-R1000

2009

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Regulation of hypothalamic renin–angiotensin system and oxidative stress by aldosterone

Experimental physiology, 2011-10, Vol.96 (10), p.1028-1038

2011

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Effects of central sodium on epithelial sodium channels in rat brain

American journal of physiology. Regulatory, integrative and comparative physiology, 2010-07, Vol.299 (1), p.R222-R233

2010

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Central infusion of aliskiren prevents sympathetic hyperactivity and hypertension in Dahl salt-sensitive rats on high salt intake

American journal of physiology. Regulatory, integrative and comparative physiology, 2012-04, Vol.302 (7), p.R825-R832

2012

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Mineralocorticoid Actions in the Brain and Hypertension

Current hypertension reports, 2011-06, Vol.13 (3), p.214-220

2011

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Chronic central versus systemic blockade of AT1 receptors and cardiac dysfunction in rats post-myocardial infarction

American journal of physiology. Heart and circulatory physiology, 2009-09, Vol.297 (3), p.H968

2009

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Possible role of brain salt-inducible kinase 1 in responses to central sodium in Dahl rats

American journal of physiology. Regulatory, integrative and comparative physiology, 2012-07, Vol.303 (2), p.R236-R245

2012

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Central infusion of aldosterone synthase inhibitor prevents sympathetic hyperactivity and hypertension by central Na+ in Wistar rats

American journal of physiology. Regulatory, integrative and comparative physiology, 2008-07, Vol.295 (1), p.R166-R172

2008

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The central role of the brain in salt-sensitive hypertension

Current opinion in cardiology, 2006-07, Vol.21 (4), p.295-304

2006

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Prevention of Sympathetic and Cardiac Dysfunction After Myocardial Infarction in Transgenic Rats Deficient in Brain Angiotensinogen

Circulation research, 2004-04, Vol.94 (6), p.843-849

2004

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Brain sodium channels and ouabainlike compounds mediate central aldosterone-induced hypertension

American journal of physiology. Heart and circulatory physiology, 2003-12, Vol.285 (6), p.H2516-H2523

2003

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Blockade of brain mineralocorticoid receptors or Na+ channels prevents sympathetic hyperactivity and improves cardiac function in rats post-MI

American journal of physiology. Heart and circulatory physiology, 2005-05, Vol.288 (5), p.H2491-H2497

2005

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Activation of brain renin-angiotensin-aldosterone system by central sodium in Wistar rats

American journal of physiology. Heart and circulatory physiology, 2006-09, Vol.291 (3), p.H1109-H1117

2006

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The brain renin-angiotensin-aldosterone system: A major mechanism for sympathetic hyperactivity and left ventricular remodeling and dysfunction after myocardial infarction

Current heart failure reports, 2009-06, Vol.6 (2), p.81-88

2009

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Sympathetic hyperactivity and cardiac dysfunction post-MI: Different impact of specific CNS versus general AT1 receptor blockade

Journal of molecular and cellular cardiology, 2007-10, Vol.43 (4), p.479-486

2007

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Enhanced sympathoexcitatory and pressor responses to central Na+ in Dahl salt-sensitive vs. -resistant rats

American journal of physiology. Heart and circulatory physiology, 2001-11, Vol.281 (5), p.H1881-H1889

2001

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Optimal dietary methionine requirement for juvenile sea cucumber Apostichopus japonicus selenka

Aquaculture research, 2021-04, Vol.52 (4), p.1348-1358

2021

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Brain Amiloride-Sensitive Phe-Met-Arg-Phe-NH2–Gated Na+ Channels and Na+-Induced Sympathoexcitation and Hypertension

Hypertension (Dallas, Tex. 1979), 2002-02, Vol.39 (2, Part 2 Suppl), p.557-561

2002

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Brain renin–angiotensin–aldosterone system and ventricular remodeling after myocardial infarct: a reviewThis article is one of a selection of papers published in a special issue on Advances in Cardiovascular Research

Canadian journal of physiology and pharmacology, 2009-12, Vol.87 (12), p.979-988

2009

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Inhibition of brain angiotensin III attenuates sympathetic hyperactivity and cardiac dysfunction in rats post-myocardial infarction

Cardiovascular research, 2013-03, Vol.97 (3), p.424-431

2013

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Mineralocorticoid and AT1 receptors in the paraventricular nucleus contribute to sympathetic hyperactivity and cardiac dysfunction in rats post myocardial infarct

The Journal of physiology, 2014-08, Vol.592 (15), p.3273-3286

2014

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Mineralocorticoid and angiotensin II type 1 receptors in the subfornical organ mediate angiotensin II – Induced hypothalamic reactive oxygen species and hypertension

Central neuronal activation and pressor responses induced by circulating ANG II: role of the brain aldosterone-"ouabain" pathway

Knockdown of mineralocorticoid or angiotensin II type 1 receptor gene expression in the paraventricular nucleus prevents angiotensin II hypertension in rats

Neuroendocrine humoral and vascular components in the pressor pathway for brain angiotensin II: a new axis in long term blood pressure control

Role of central nervous system aldosterone synthase and mineralocorticoid receptors in salt-induced hypertension in Dahl salt-sensitive rats

Regulation of hypothalamic renin–angiotensin system and oxidative stress by aldosterone

Effects of central sodium on epithelial sodium channels in rat brain

Central infusion of aliskiren prevents sympathetic hyperactivity and hypertension in Dahl salt-sensitive rats on high salt intake

Mineralocorticoid Actions in the Brain and Hypertension

Chronic central versus systemic blockade of AT1 receptors and cardiac dysfunction in rats post-myocardial infarction

Possible role of brain salt-inducible kinase 1 in responses to central sodium in Dahl rats

Central infusion of aldosterone synthase inhibitor prevents sympathetic hyperactivity and hypertension by central Na+ in Wistar rats

The central role of the brain in salt-sensitive hypertension

Prevention of Sympathetic and Cardiac Dysfunction After Myocardial Infarction in Transgenic Rats Deficient in Brain Angiotensinogen

Brain sodium channels and ouabainlike compounds mediate central aldosterone-induced hypertension

Blockade of brain mineralocorticoid receptors or Na+ channels prevents sympathetic hyperactivity and improves cardiac function in rats post-MI

Activation of brain renin-angiotensin-aldosterone system by central sodium in Wistar rats

The brain renin-angiotensin-aldosterone system: A major mechanism for sympathetic hyperactivity and left ventricular remodeling and dysfunction after myocardial infarction

Sympathetic hyperactivity and cardiac dysfunction post-MI: Different impact of specific CNS versus general AT1 receptor blockade

Enhanced sympathoexcitatory and pressor responses to central Na+ in Dahl salt-sensitive vs. -resistant rats

Optimal dietary methionine requirement for juvenile sea cucumber Apostichopus japonicus selenka

Brain Amiloride-Sensitive Phe-Met-Arg-Phe-NH2–Gated Na+ Channels and Na+-Induced Sympathoexcitation and Hypertension

Brain renin–angiotensin–aldosterone system and ventricular remodeling after myocardial infarct: a reviewThis article is one of a selection of papers published in a special issue on Advances in Cardiovascular Research

Inhibition of brain angiotensin III attenuates sympathetic hyperactivity and cardiac dysfunction in rats post-myocardial infarction

Mineralocorticoid and AT1 receptors in the paraventricular nucleus contribute to sympathetic hyperactivity and cardiac dysfunction in rats post myocardial infarct

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