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p38 MAPK in Glucose Metabolism of Skeletal Muscle: Beneficial or Harmful?

International journal of molecular sciences, 2020-09, Vol.21 (18), p.6480

2020

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Glycine-based treatment ameliorates NAFLD by modulating fatty acid oxidation, glutathione synthesis, and the gut microbiome

Science translational medicine, 2020-12, Vol.12 (572)

2020

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Paraoxonase1 (PON1) reduces insulin resistance in mice fed a high-fat diet, and promotes GLUT4 overexpression in myocytes, via the IRS-1/Akt pathway

Atherosclerosis, 2013-07, Vol.229 (1), p.71-78

2013

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Atherogenicity of amino acids in the lipid-laden macrophage model system in vitro and in atherosclerotic mice: a key role for triglyceride metabolism

The Journal of nutritional biochemistry, 2017-07, Vol.45, p.24-38

2017

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A case of rapidly progressive upper limb ischemic necrosis in a patient with COVID-19

International journal of infectious diseases, 2021-05, Vol.106, p.401-404

2021

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Lyso-DGTS Lipid Derivatives Enhance PON1 Activities and Prevent Oxidation of LDL: A Structure–Activity Relationship Study

Antioxidants, 2022-10, Vol.11 (10), p.2058

2022

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Dysregulated oxalate metabolism is a driver and therapeutic target in atherosclerosis

Cell reports (Cambridge), 2021-07, Vol.36 (4), p.109420-109420, Article 109420

2021

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Pomegranate juice consumption reduces oxidative stress, atherogenic modifications to LDL, and platelet aggregation: studies in humans and in atherosclerotic apolipoprotein E-deficient mice

The American journal of clinical nutrition, 2000-05, Vol.71 (5), p.1062-1076

2000

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Mineralocorticoid Receptor Blocker Increases Angiotensin-Converting Enzyme 2 Activity in Congestive Heart Failure Patients

Circulation research, 2005-10, Vol.97 (9), p.946-953

2005

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Induction of glutathione biosynthesis by glycine-based treatment mitigates atherosclerosis

Redox biology, 2022-06, Vol.52, p.102313-102313, Article 102313

2022

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Paraoxonase 2 (PON2) decreases high glucose-induced macrophage triglycerides (TG) accumulation, via inhibition of NADPH-oxidase and DGAT1 activity: Studies in PON2-deficient mice

Atherosclerosis, 2010-02, Vol.208 (2), p.390-395

2010

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High glucose stimulates macrophage SR-BI expression and induces a switch in its activity from cholesterol efflux to cholesterol influx

Biochemical and biophysical research communications, 2010-01, Vol.391 (1), p.523-528

2010

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Pomegranate Juice Supplementation to Atherosclerotic Mice Reduces Macrophage Lipid Peroxidation, Cellular Cholesterol Accumulation and Development of Atherosclerosis

The Journal of nutrition, 2001-08, Vol.131 (8), p.2082-2089

2001

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Paraoxonase 2 Induces a Phenotypic Switch in Macrophage Polarization Favoring an M2 Anti-Inflammatory State

International Journal of Endocrinology, 2015-01, Vol.2015, p.915243-9

2015

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Ginger Extract Consumption Reduces Plasma Cholesterol, Inhibits LDL Oxidation and Attenuates Development of Atherosclerosis in Atherosclerotic, Apolipoprotein E-Deficient Mice

The Journal of nutrition, 2000-05, Vol.130 (5), p.1124-1131

2000

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High Glucose Concentration Increases Macrophage Cholesterol Biosynthesis in Diabetes Through Activation of the Sterol Regulatory Element Binding Protein 1 (SREBP1): Inhibitory Effect of Insulin

Journal of cardiovascular pharmacology, 2008-10, Vol.52 (4), p.324-332

2008

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Soybean Oil Modulates the Gut Microbiota Associated with Atherogenic Biomarkers

Microorganisms (Basel), 2020-03, Vol.8 (4), p.486

2020

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Macrophage NADPH oxidase activation, impaired cholesterol fluxes, and increased cholesterol biosynthesis in diabetic mice: A stimulatory role for d -glucose

Atherosclerosis, 2007-12, Vol.195 (2), p.277-286

2007

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VLDL triglycerides inhibit HDL-associated paraoxonase 1 (PON1) activity: in vitro and in vivo studies

BioFactors (Oxford), 2012-07, Vol.38 (4), p.292-299

2012

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Niacin Administration Significantly Reduces Oxidative Stress in Patients With Hypercholesterolemia and Low Levels of High-Density Lipoprotein Cholesterol

The American journal of the medical sciences, 2013-03, Vol.345 (3), p.195-199

2013

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A mouse model for human atherosclerosis: Long-term histopathological study of lesion development in the aortic arch of apolipoprotein E-deficient (E 0) mice

Acta histochemica, 2006-01, Vol.108 (6), p.415-424

2006

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Atorvastatin therapy in hypercholesterolemic patients suppresses cellular uptake of oxidized-LDL by differentiating monocytes

Atherosclerosis, 2002-09, Vol.164 (1), p.179-185

2002

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Anti-oxidative effects of pomegranate juice (PJ) consumption by diabetic patients on serum and on macrophages

Atherosclerosis, 2006-08, Vol.187 (2), p.363-371

2006

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Oxidative stress and macrophage foam cell formation during diabetes mellitus-induced atherogenesis: role of insulin therapy

Pharmacology & therapeutics (Oxford), 2012-11, Vol.136 (2), p.175-185

2012

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Novel bioactive lipids enhanced HDL-mediated cholesterol efflux from macrophages through the ABCA1 receptor pathway

Chemistry and physics of lipids, 2024-01, Vol.258, p.105367-105367, Article 105367

2024

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p38 MAPK in Glucose Metabolism of Skeletal Muscle: Beneficial or Harmful?

Glycine-based treatment ameliorates NAFLD by modulating fatty acid oxidation, glutathione synthesis, and the gut microbiome

Paraoxonase1 (PON1) reduces insulin resistance in mice fed a high-fat diet, and promotes GLUT4 overexpression in myocytes, via the IRS-1/Akt pathway

Atherogenicity of amino acids in the lipid-laden macrophage model system in vitro and in atherosclerotic mice: a key role for triglyceride metabolism

A case of rapidly progressive upper limb ischemic necrosis in a patient with COVID-19

Lyso-DGTS Lipid Derivatives Enhance PON1 Activities and Prevent Oxidation of LDL: A Structure–Activity Relationship Study

Dysregulated oxalate metabolism is a driver and therapeutic target in atherosclerosis

Pomegranate juice consumption reduces oxidative stress, atherogenic modifications to LDL, and platelet aggregation: studies in humans and in atherosclerotic apolipoprotein E-deficient mice

Mineralocorticoid Receptor Blocker Increases Angiotensin-Converting Enzyme 2 Activity in Congestive Heart Failure Patients

Induction of glutathione biosynthesis by glycine-based treatment mitigates atherosclerosis

Paraoxonase 2 (PON2) decreases high glucose-induced macrophage triglycerides (TG) accumulation, via inhibition of NADPH-oxidase and DGAT1 activity: Studies in PON2-deficient mice

High glucose stimulates macrophage SR-BI expression and induces a switch in its activity from cholesterol efflux to cholesterol influx

Pomegranate Juice Supplementation to Atherosclerotic Mice Reduces Macrophage Lipid Peroxidation, Cellular Cholesterol Accumulation and Development of Atherosclerosis

Paraoxonase 2 Induces a Phenotypic Switch in Macrophage Polarization Favoring an M2 Anti-Inflammatory State

Ginger Extract Consumption Reduces Plasma Cholesterol, Inhibits LDL Oxidation and Attenuates Development of Atherosclerosis in Atherosclerotic, Apolipoprotein E-Deficient Mice

High Glucose Concentration Increases Macrophage Cholesterol Biosynthesis in Diabetes Through Activation of the Sterol Regulatory Element Binding Protein 1 (SREBP1): Inhibitory Effect of Insulin

Soybean Oil Modulates the Gut Microbiota Associated with Atherogenic Biomarkers

Macrophage NADPH oxidase activation, impaired cholesterol fluxes, and increased cholesterol biosynthesis in diabetic mice: A stimulatory role for d -glucose

VLDL triglycerides inhibit HDL-associated paraoxonase 1 (PON1) activity: in vitro and in vivo studies

Niacin Administration Significantly Reduces Oxidative Stress in Patients With Hypercholesterolemia and Low Levels of High-Density Lipoprotein Cholesterol

A mouse model for human atherosclerosis: Long-term histopathological study of lesion development in the aortic arch of apolipoprotein E-deficient (E 0) mice

Atorvastatin therapy in hypercholesterolemic patients suppresses cellular uptake of oxidized-LDL by differentiating monocytes

Anti-oxidative effects of pomegranate juice (PJ) consumption by diabetic patients on serum and on macrophages

Oxidative stress and macrophage foam cell formation during diabetes mellitus-induced atherogenesis: role of insulin therapy

Novel bioactive lipids enhanced HDL-mediated cholesterol efflux from macrophages through the ABCA1 receptor pathway

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