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p38 MAPK in Glucose Metabolism of Skeletal Muscle: Beneficial or Harmful?

International journal of molecular sciences, 2020-09, Vol.21 (18), p.6480

2020

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Glycine-based treatment ameliorates NAFLD by modulating fatty acid oxidation, glutathione synthesis, and the gut microbiome

Science translational medicine, 2020-12, Vol.12 (572)

2020

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P38α MAPK Coordinates Mitochondrial Adaptation to Caloric Surplus in Skeletal Muscle

International journal of molecular sciences, 2024-07, Vol.25 (14), p.7789

2024

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Paraoxonase1 (PON1) reduces insulin resistance in mice fed a high-fat diet, and promotes GLUT4 overexpression in myocytes, via the IRS-1/Akt pathway

Atherosclerosis, 2013-07, Vol.229 (1), p.71-78

2013

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Atherogenicity of amino acids in the lipid-laden macrophage model system in vitro and in atherosclerotic mice: a key role for triglyceride metabolism

The Journal of nutritional biochemistry, 2017-07, Vol.45, p.24-38

2017

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A case of rapidly progressive upper limb ischemic necrosis in a patient with COVID-19

International journal of infectious diseases, 2021-05, Vol.106, p.401-404

2021

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Lyso-DGTS Lipid Derivatives Enhance PON1 Activities and Prevent Oxidation of LDL: A Structure–Activity Relationship Study

Antioxidants, 2022-10, Vol.11 (10), p.2058

2022

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Dysregulated oxalate metabolism is a driver and therapeutic target in atherosclerosis

Cell reports (Cambridge), 2021-07, Vol.36 (4), p.109420-109420, Article 109420

2021

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Pomegranate juice consumption reduces oxidative stress, atherogenic modifications to LDL, and platelet aggregation: studies in humans and in atherosclerotic apolipoprotein E-deficient mice

The American journal of clinical nutrition, 2000-05, Vol.71 (5), p.1062-1076

2000

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Mineralocorticoid Receptor Blocker Increases Angiotensin-Converting Enzyme 2 Activity in Congestive Heart Failure Patients

Circulation research, 2005-10, Vol.97 (9), p.946-953

2005

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Induction of glutathione biosynthesis by glycine-based treatment mitigates atherosclerosis

Redox biology, 2022-06, Vol.52, p.102313-102313, Article 102313

2022

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Paraoxonase 2 (PON2) decreases high glucose-induced macrophage triglycerides (TG) accumulation, via inhibition of NADPH-oxidase and DGAT1 activity: Studies in PON2-deficient mice

Atherosclerosis, 2010-02, Vol.208 (2), p.390-395

2010

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High glucose stimulates macrophage SR-BI expression and induces a switch in its activity from cholesterol efflux to cholesterol influx

Biochemical and biophysical research communications, 2010-01, Vol.391 (1), p.523-528

2010

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Pomegranate Juice Supplementation to Atherosclerotic Mice Reduces Macrophage Lipid Peroxidation, Cellular Cholesterol Accumulation and Development of Atherosclerosis

The Journal of nutrition, 2001-08, Vol.131 (8), p.2082-2089

2001

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Paraoxonase 2 Induces a Phenotypic Switch in Macrophage Polarization Favoring an M2 Anti-Inflammatory State

International Journal of Endocrinology, 2015-01, Vol.2015, p.915243-9

2015

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Ginger Extract Consumption Reduces Plasma Cholesterol, Inhibits LDL Oxidation and Attenuates Development of Atherosclerosis in Atherosclerotic, Apolipoprotein E-Deficient Mice

The Journal of nutrition, 2000-05, Vol.130 (5), p.1124-1131

2000

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High Glucose Concentration Increases Macrophage Cholesterol Biosynthesis in Diabetes Through Activation of the Sterol Regulatory Element Binding Protein 1 (SREBP1): Inhibitory Effect of Insulin

Journal of cardiovascular pharmacology, 2008-10, Vol.52 (4), p.324-332

2008

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Soybean Oil Modulates the Gut Microbiota Associated with Atherogenic Biomarkers

Microorganisms (Basel), 2020-03, Vol.8 (4), p.486

2020

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Macrophage NADPH oxidase activation, impaired cholesterol fluxes, and increased cholesterol biosynthesis in diabetic mice: A stimulatory role for d -glucose

Atherosclerosis, 2007-12, Vol.195 (2), p.277-286

2007

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VLDL triglycerides inhibit HDL-associated paraoxonase 1 (PON1) activity: in vitro and in vivo studies

BioFactors (Oxford), 2012-07, Vol.38 (4), p.292-299

2012

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Niacin Administration Significantly Reduces Oxidative Stress in Patients With Hypercholesterolemia and Low Levels of High-Density Lipoprotein Cholesterol

The American journal of the medical sciences, 2013-03, Vol.345 (3), p.195-199

2013

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A mouse model for human atherosclerosis: Long-term histopathological study of lesion development in the aortic arch of apolipoprotein E-deficient (E 0) mice

Acta histochemica, 2006-01, Vol.108 (6), p.415-424

2006

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Atorvastatin therapy in hypercholesterolemic patients suppresses cellular uptake of oxidized-LDL by differentiating monocytes

Atherosclerosis, 2002-09, Vol.164 (1), p.179-185

2002

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Anti-oxidative effects of pomegranate juice (PJ) consumption by diabetic patients on serum and on macrophages

Atherosclerosis, 2006-08, Vol.187 (2), p.363-371

2006

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Oxidative stress and macrophage foam cell formation during diabetes mellitus-induced atherogenesis: role of insulin therapy

Pharmacology & therapeutics (Oxford), 2012-11, Vol.136 (2), p.175-185

2012

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p38 MAPK in Glucose Metabolism of Skeletal Muscle: Beneficial or Harmful?

Glycine-based treatment ameliorates NAFLD by modulating fatty acid oxidation, glutathione synthesis, and the gut microbiome

P38α MAPK Coordinates Mitochondrial Adaptation to Caloric Surplus in Skeletal Muscle

Paraoxonase1 (PON1) reduces insulin resistance in mice fed a high-fat diet, and promotes GLUT4 overexpression in myocytes, via the IRS-1/Akt pathway

Atherogenicity of amino acids in the lipid-laden macrophage model system in vitro and in atherosclerotic mice: a key role for triglyceride metabolism

A case of rapidly progressive upper limb ischemic necrosis in a patient with COVID-19

Lyso-DGTS Lipid Derivatives Enhance PON1 Activities and Prevent Oxidation of LDL: A Structure–Activity Relationship Study

Dysregulated oxalate metabolism is a driver and therapeutic target in atherosclerosis

Pomegranate juice consumption reduces oxidative stress, atherogenic modifications to LDL, and platelet aggregation: studies in humans and in atherosclerotic apolipoprotein E-deficient mice

Mineralocorticoid Receptor Blocker Increases Angiotensin-Converting Enzyme 2 Activity in Congestive Heart Failure Patients

Induction of glutathione biosynthesis by glycine-based treatment mitigates atherosclerosis

Paraoxonase 2 (PON2) decreases high glucose-induced macrophage triglycerides (TG) accumulation, via inhibition of NADPH-oxidase and DGAT1 activity: Studies in PON2-deficient mice

High glucose stimulates macrophage SR-BI expression and induces a switch in its activity from cholesterol efflux to cholesterol influx

Pomegranate Juice Supplementation to Atherosclerotic Mice Reduces Macrophage Lipid Peroxidation, Cellular Cholesterol Accumulation and Development of Atherosclerosis

Paraoxonase 2 Induces a Phenotypic Switch in Macrophage Polarization Favoring an M2 Anti-Inflammatory State

Ginger Extract Consumption Reduces Plasma Cholesterol, Inhibits LDL Oxidation and Attenuates Development of Atherosclerosis in Atherosclerotic, Apolipoprotein E-Deficient Mice

High Glucose Concentration Increases Macrophage Cholesterol Biosynthesis in Diabetes Through Activation of the Sterol Regulatory Element Binding Protein 1 (SREBP1): Inhibitory Effect of Insulin

Soybean Oil Modulates the Gut Microbiota Associated with Atherogenic Biomarkers

Macrophage NADPH oxidase activation, impaired cholesterol fluxes, and increased cholesterol biosynthesis in diabetic mice: A stimulatory role for d -glucose

VLDL triglycerides inhibit HDL-associated paraoxonase 1 (PON1) activity: in vitro and in vivo studies

Niacin Administration Significantly Reduces Oxidative Stress in Patients With Hypercholesterolemia and Low Levels of High-Density Lipoprotein Cholesterol

A mouse model for human atherosclerosis: Long-term histopathological study of lesion development in the aortic arch of apolipoprotein E-deficient (E 0) mice

Atorvastatin therapy in hypercholesterolemic patients suppresses cellular uptake of oxidized-LDL by differentiating monocytes

Anti-oxidative effects of pomegranate juice (PJ) consumption by diabetic patients on serum and on macrophages

Oxidative stress and macrophage foam cell formation during diabetes mellitus-induced atherogenesis: role of insulin therapy

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