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Targeting BCL2 With BH3 Mimetics: Basic Science and Clinical Application of Venetoclax in Chronic Lymphocytic Leukemia and Related B Cell Malignancies

Clinical pharmacology and therapeutics, 2017-01, Vol.101 (1), p.89-98

2017

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A Cluster of Interferon-γ-Inducible p65 GTPases Plays a Critical Role in Host Defense against Toxoplasma gondii

Immunity (Cambridge, Mass.), 2012-08, Vol.37 (2), p.302-313

2012

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BH3-Only Proteins—Essential Initiators of Apoptotic Cell Death

Cell, 2000-12, Vol.103 (6), p.839-842

2000

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The Bcl-2 family: roles in cell survival and oncogenesis

Oncogene, 2003-11, Vol.22 (53), p.8590-8607

2003

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Targeting of MCL-1 kills MYC-driven mouse and human lymphomas even when they bear mutations in p53

Genes & development, 2014-01, Vol.28 (1), p.58-70

2014

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Programmed Anuclear Cell Death Delimits Platelet Life Span

Cell, 2007-03, Vol.128 (6), p.1173-1186

2007

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How the Bcl-2 family of proteins interact to regulate apoptosis

Cell research, 2006-02, Vol.16 (2), p.203-213

2006

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Differential Targeting of Prosurvival Bcl-2 Proteins by Their BH3-Only Ligands Allows Complementary Apoptotic Function

Molecular cell, 2005-02, Vol.17 (3), p.393-403

2005

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Proapoptotic Bcl-2 Relative Bim Required for Certain Apoptotic Responses, Leukocyte Homeostasis, and to Preclude Autoimmunity

Science (American Association for the Advancement of Science), 1999-11, Vol.286 (5445), p.1735-1738

1999

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The BH3-Only Protein Bid Is Dispensable for DNA Damage- and Replicative Stress-Induced Apoptosis or Cell-Cycle Arrest

Cell, 2007-04, Vol.129 (2), p.423-433

2007

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Bmf: A Proapoptotic BH3-Only Protein Regulated by Interaction with the Myosin V Actin Motor Complex, Activated by Anoikis

Science (American Association for the Advancement of Science), 2001-09, Vol.293 (5536), p.1829-1832

2001

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Solution Structure of Prosurvival Mcl-1 and Characterization of Its Binding by Proapoptotic BH3-only Ligands

The Journal of biological chemistry, 2005-02, Vol.280 (6), p.4738-4744

2005

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The Proapoptotic Activity of the Bcl-2 Family Member Bim Is Regulated by Interaction with the Dynein Motor Complex

Molecular cell, 1999-03, Vol.3 (3), p.287-296

1999

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The BH3 mimetic compound, ABT-737, synergizes with a range of cytotoxic chemotherapy agents in chronic lymphocytic leukemia

Leukemia, 2009-11, Vol.23 (11), p.2034-2041

2009

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Apoptosis initiated by Bcl-2-regulated caspase activation independently of the cytochrome c/Apaf-1/caspase-9 apoptosome

Nature (London), 2002-10, Vol.419 (6907), p.634-637

2002

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The Bcl-2-regulated apoptotic pathway

Journal of cell science, 2003-10, Vol.116 (Pt 20), p.4053-4056

2003

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Targeting BCL-2–expressing basal-like breast cancer with BH3-mimetics

Hereditary cancer in clinical practice, 2012-04, Vol.10 (S2), p.A25-A25, Article A25

2012

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Determination of Cell Survival by RING-Mediated Regulation of Inhibitor of Apoptosis (IAP) Protein Abundance

Proceedings of the National Academy of Sciences - PNAS, 2005-11, Vol.102 (45), p.16182-16187

2005

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Prospects for targeting the Bcl-2 family of proteins to develop novel cytotoxic drugs

Biochemical pharmacology, 2002-09, Vol.64 (5-6), p.851-863

2002

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The structure of Bcl-w reveals a role for the C-terminal residues in modulating biological activity

The EMBO journal, 2003-04, Vol.22 (7), p.1497-1507

2003

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Bim: a novel member of the Bcl-2 family that promotes apoptosis

The EMBO journal, 1998-01, Vol.17 (2), p.384-395

1998

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Activation of Fas by FasL induces apoptosis by a mechanism that cannot be blocked by Bcl-2 or Bcl-x(L)

Proceedings of the National Academy of Sciences - PNAS, 1999-12, Vol.96 (26), p.14871-14876

1999

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The BH3-mimetic ABT-737 induces mast cell apoptosis in vitro and in vivo: potential for therapeutics

The Journal of immunology (1950), 2010-08, Vol.185 (4), p.2555-2562

2010

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The anti-apoptosis function of Bcl-2 can be genetically separated from its inhibitory effect on cell cycle entry

The EMBO journal, 1997-08, Vol.16 (15), p.4628-4638

1997

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Apoptosis Regulatory Proteins
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Proto-Oncogene Proteins C-Bcl-2 - Metabolism
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Targeting BCL2 With BH3 Mimetics: Basic Science and Clinical Application of Venetoclax in Chronic Lymphocytic Leukemia and Related B Cell Malignancies

A Cluster of Interferon-γ-Inducible p65 GTPases Plays a Critical Role in Host Defense against Toxoplasma gondii

BH3-Only Proteins—Essential Initiators of Apoptotic Cell Death

The Bcl-2 family: roles in cell survival and oncogenesis

Targeting of MCL-1 kills MYC-driven mouse and human lymphomas even when they bear mutations in p53

Programmed Anuclear Cell Death Delimits Platelet Life Span

How the Bcl-2 family of proteins interact to regulate apoptosis

Differential Targeting of Prosurvival Bcl-2 Proteins by Their BH3-Only Ligands Allows Complementary Apoptotic Function

Proapoptotic Bcl-2 Relative Bim Required for Certain Apoptotic Responses, Leukocyte Homeostasis, and to Preclude Autoimmunity

The BH3-Only Protein Bid Is Dispensable for DNA Damage- and Replicative Stress-Induced Apoptosis or Cell-Cycle Arrest

Bmf: A Proapoptotic BH3-Only Protein Regulated by Interaction with the Myosin V Actin Motor Complex, Activated by Anoikis

Solution Structure of Prosurvival Mcl-1 and Characterization of Its Binding by Proapoptotic BH3-only Ligands

The Proapoptotic Activity of the Bcl-2 Family Member Bim Is Regulated by Interaction with the Dynein Motor Complex

The BH3 mimetic compound, ABT-737, synergizes with a range of cytotoxic chemotherapy agents in chronic lymphocytic leukemia

Apoptosis initiated by Bcl-2-regulated caspase activation independently of the cytochrome c/Apaf-1/caspase-9 apoptosome

The Bcl-2-regulated apoptotic pathway

Targeting BCL-2–expressing basal-like breast cancer with BH3-mimetics

Determination of Cell Survival by RING-Mediated Regulation of Inhibitor of Apoptosis (IAP) Protein Abundance

BTK Leu528Trp - a Potential Secondary Resistance Mechanism Specific for Patients with Chronic Lymphocytic Leukemia Treated with the Next Generation BTK Inhibitor Zanubrutinib

Prospects for targeting the Bcl-2 family of proteins to develop novel cytotoxic drugs

The structure of Bcl-w reveals a role for the C-terminal residues in modulating biological activity

Bim: a novel member of the Bcl-2 family that promotes apoptosis

Activation of Fas by FasL induces apoptosis by a mechanism that cannot be blocked by Bcl-2 or Bcl-x(L)

The BH3-mimetic ABT-737 induces mast cell apoptosis in vitro and in vivo: potential for therapeutics

The anti-apoptosis function of Bcl-2 can be genetically separated from its inhibitory effect on cell cycle entry

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