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Sarcolipin is a newly identified regulator of muscle-based thermogenesis in mammals

Nature medicine, 2012-10, Vol.18 (10), p.1575-1579

2012

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STIM1 elevation in the heart results in aberrant Ca2+ handling and cardiomyopathy

Journal of molecular and cellular cardiology, 2015-10, Vol.87, p.38-47

2015

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Calcium influx is sufficient to induce muscular dystrophy through a TRPC-dependent mechanism

Proceedings of the National Academy of Sciences - PNAS, 2009-11, Vol.106 (45), p.19023-19028

2009

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Unraveling the secrets of a double life: Contractile versus signaling Ca2+ in a cardiac myocyte

Journal of molecular and cellular cardiology, 2012-02, Vol.52 (2), p.317-322

2012

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Sarcolipin overexpression improves muscle energetics and reduces fatigue

Journal of applied physiology (1985), 2015-04, Vol.118 (8), p.1050-1058

2015

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Mitigation of muscular dystrophy in mice by SERCA overexpression in skeletal muscle

The Journal of clinical investigation, 2011-03, Vol.121 (3), p.1044-1052

2011

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Decreased cardiac L-type Ca²⁺ channel activity induces hypertrophy and heart failure in mice

The Journal of clinical investigation, 2012-01, Vol.122 (1), p.280-290

2012

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Identification of Cysteines Involved in S-Nitrosylation, S-Glutathionylation, and Oxidation to Disulfides in Ryanodine Receptor Type 1

The Journal of biological chemistry, 2006-12, Vol.281 (52), p.40354-40368

2006

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RhoA signaling in cardiomyocytes protects against stress-induced heart failure but facilitates cardiac fibrosis

Science signaling, 2014-10, Vol.7 (348), p.ra100

2014

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Heat- and anesthesia-induced malignant hyperthermia in an RyR1 knock-in mouse

The FASEB journal, 2006-02, Vol.20 (2), p.329-330

2006

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Mice with the R176Q Cardiac Ryanodine Receptor Mutation Exhibit Catecholamine-Induced Ventricular Tachycardia and Cardiomyopathy

Proceedings of the National Academy of Sciences - PNAS, 2006-08, Vol.103 (32), p.12179-12184

2006

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Triadin binding to the C-terminal luminal loop of the ryanodine receptor is important for skeletal muscle excitation contraction coupling

The Journal of general physiology, 2007-10, Vol.130 (4), p.365-378

2007

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Cyclophilin D controls mitochondrial pore-dependent Ca(2+) exchange, metabolic flexibility, and propensity for heart failure in mice

The Journal of clinical investigation, 2010-10, Vol.120 (10), p.3680-3687

2010

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Epac and Phospholipase Cϵ Regulate Ca2+ Release in the Heart by Activation of Protein Kinase Cϵ and Calcium-Calmodulin Kinase II

The Journal of biological chemistry, 2009-01, Vol.284 (3), p.1514-1522

2009

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RyR1 S-Nitrosylation Underlies Environmental Heat Stroke and Sudden Death in Y522S RyR1 Knockin Mice

Cell, 2008-04, Vol.133 (1), p.53-65

2008

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Enhanced Ca²⁺ influx from STIM1-Orai1 induces muscle pathology in mouse models of muscular dystrophy

Human molecular genetics, 2014-07, Vol.23 (14), p.3706-3715

2014

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Unrestrained p38 MAPK Activation in Dusp1/4 Double-Null Mice Induces Cardiomyopathy

Circulation research, 2013-01, Vol.112 (1), p.48-56

2013

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Na+ Dysregulation Coupled with Ca2+ Entry through NCX1 Promotes Muscular Dystrophy in Mice

Molecular and cellular biology, 2014-06, Vol.34 (11), p.1991-2002

2014

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Novel excitation-contraction uncoupled RYR1 mutations in patients with central core disease

Neuromuscular disorders : NMD, 2013-02, Vol.23 (2), p.120-132

2013

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Ryr1I⁴⁸⁹⁵T mutation abolishes Ca²⁺ release channel function and delays development in homozygous offspring of a mutant mouse line

Proceedings of the National Academy of Sciences - PNAS, 2007-11, Vol.104 (47), p.18537-18542

2007

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Sarcolipin is a newly identified regulator of muscle-based thermogenesis in mammals

Nature medicine, 2012-12, Vol.18 (12), p.1857

2012

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Dynamic alterations in myoplasmic Ca2+ in malignant hyperthermia and central core disease

Biochemical and biophysical research communications, 2004-10, Vol.322 (4), p.1256-1266

2004

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Cyclophilin D controls mitochondrial pore–dependent Ca2+ exchange, metabolic flexibility, and propensity for heart failure in mice

The Journal of clinical investigation, 2010-10, Vol.120 (10), p.3680-3687

2010

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Cyclophilin D controls mitochondrial pore–dependent Ca2+ exchange, metabolic flexibility, and propensity for heart failure in mice

The Journal of clinical investigation, 2010-10, Vol.120 (10), p.3680-3687

2010

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Abstract 186: Increased STIM1 Expression Results in Altered Calcium Handling and Heart Failure

Circulation research, 2015-07, Vol.117 (suppl_1)

2015

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Sarcolipin is a newly identified regulator of muscle-based thermogenesis in mammals

STIM1 elevation in the heart results in aberrant Ca2+ handling and cardiomyopathy

Calcium influx is sufficient to induce muscular dystrophy through a TRPC-dependent mechanism

Unraveling the secrets of a double life: Contractile versus signaling Ca2+ in a cardiac myocyte

Sarcolipin overexpression improves muscle energetics and reduces fatigue

Mitigation of muscular dystrophy in mice by SERCA overexpression in skeletal muscle

Decreased cardiac L-type Ca²⁺ channel activity induces hypertrophy and heart failure in mice

Identification of Cysteines Involved in S-Nitrosylation, S-Glutathionylation, and Oxidation to Disulfides in Ryanodine Receptor Type 1

RhoA signaling in cardiomyocytes protects against stress-induced heart failure but facilitates cardiac fibrosis

Heat- and anesthesia-induced malignant hyperthermia in an RyR1 knock-in mouse

Mice with the R176Q Cardiac Ryanodine Receptor Mutation Exhibit Catecholamine-Induced Ventricular Tachycardia and Cardiomyopathy

Triadin binding to the C-terminal luminal loop of the ryanodine receptor is important for skeletal muscle excitation contraction coupling

Cyclophilin D controls mitochondrial pore-dependent Ca(2+) exchange, metabolic flexibility, and propensity for heart failure in mice

Epac and Phospholipase Cϵ Regulate Ca2+ Release in the Heart by Activation of Protein Kinase Cϵ and Calcium-Calmodulin Kinase II

RyR1 S-Nitrosylation Underlies Environmental Heat Stroke and Sudden Death in Y522S RyR1 Knockin Mice

Enhanced Ca²⁺ influx from STIM1-Orai1 induces muscle pathology in mouse models of muscular dystrophy

Unrestrained p38 MAPK Activation in Dusp1/4 Double-Null Mice Induces Cardiomyopathy

Na+ Dysregulation Coupled with Ca2+ Entry through NCX1 Promotes Muscular Dystrophy in Mice

Novel excitation-contraction uncoupled RYR1 mutations in patients with central core disease

Ryr1I⁴⁸⁹⁵T mutation abolishes Ca²⁺ release channel function and delays development in homozygous offspring of a mutant mouse line

Sarcolipin is a newly identified regulator of muscle-based thermogenesis in mammals

Dynamic alterations in myoplasmic Ca2+ in malignant hyperthermia and central core disease

Cyclophilin D controls mitochondrial pore–dependent Ca2+ exchange, metabolic flexibility, and propensity for heart failure in mice

Cyclophilin D controls mitochondrial pore–dependent Ca2+ exchange, metabolic flexibility, and propensity for heart failure in mice

Abstract 186: Increased STIM1 Expression Results in Altered Calcium Handling and Heart Failure

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