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Group 2 innate lymphoid cells mediate ozone-induced airway inflammation and hyperresponsiveness in mice

Journal of allergy and clinical immunology, 2016-02, Vol.137 (2), p.571-578

2016

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mTORC2 regulates multiple aspects of NKT‐cell development and function

European journal of immunology, 2017-03, Vol.47 (3), p.516-526

2017

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Oxidative damage of SP‐D abolishes control of eosinophil extracellular DNA trap formation

Journal of leukocyte biology, 2018-07, Vol.104 (1), p.205-214

2018

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Ozone Inhalation Attenuated the Effects of Budesonide on Aspergillus fumigatus -Induced Airway Inflammation and Hyperreactivity in Mice

Frontiers in immunology, 2019-09, Vol.10, p.2173

2019

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Diacylglycerol kinase ζ promotes allergic airway inflammation and airway hyperresponsiveness through distinct mechanisms

Science signaling, 2019-09, Vol.12 (597)

2019

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Adeno-associated virus 9-mediated airway expression of antibody protects old and immunodeficient mice against influenza virus

Clinical and vaccine immunology, 2014-11, Vol.21 (11), p.1528-1533

2014

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Surfactant Protein-D (SP-D) Is A Lung Specific Regulator of Group 2 Innate Lymphoid Cells (ILC2)

Journal of allergy and clinical immunology, 2017-02, Vol.139 (2), p.AB15-AB15

2017

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Antagonistic Effects Of Ozone (O3) Exposure and Glucocorticoid Treatment On Airway Hyperresponsiveness (AHR) and Surfactant Protein D (SP-D) Production In Mice

Journal of allergy and clinical immunology, 2014-02, Vol.133 (2), p.AB161-AB161

2014

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Circulating T cells, natural killer (NK) cells and group 2 innate lymphoid cells (ILC2) of severe asthmapatients from the University of California Asthma Network (UCANTM ) clinic, express increased levels of both IL-13 and IL-17

Journal of allergy and clinical immunology, 2017-02, Vol.139 (2), p.AB267-AB267

2017

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Inhalational exposure to statins and drug vehicle induces transient immunological changes in both the airways and peripheral blood of non-human primates

Journal of allergy and clinical immunology, 2017-02, Vol.139 (2), p.AB266-AB266

2017

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Role of Natural Killer (NK) Cell Surface Receptor –NKp46– in Primary Influenza A Infection

Journal of allergy and clinical immunology, 2015-02, Vol.135 (2), p.AB164-AB164

2015

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169 Off-the-shelf, engineered iPSC-derived NK cells mediate potent cytotoxic activity against primary glioblastoma cells and promote durable long-term survival in vivo

Journal for immunotherapy of cancer, 2021-11, Vol.9 (Suppl 2), p.A180-A180

2021

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Group-2 Innate Lymphoid Cells Promote Air-Pollutant Induced Airway Inflammation and Hyperresponsiveness (AHR)

Journal of allergy and clinical immunology, 2014-02, Vol.133 (2), p.AB232-AB232

2014

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Epithelial IL-33 and TSLP Elicit Innate Lymphoid Cell Responses to Mediate Ozone-Induced Airway Inflammation and Hyperresponsiveness

Journal of allergy and clinical immunology, 2015-02, Vol.135 (2), p.AB82-AB82

2015

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155 iPSC-derived NK cells mediate robust anti-tumor activity against glioblastoma

Journal for immunotherapy of cancer, 2020-11, Vol.8 (Suppl 3), p.A93-A94

2020

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Interactions of Natural Killer (NK) Cells and Surfactant Protein D (SP-D) in Regulation of Ozone Induced Airway Inflammation: Involvement of NKp46

Journal of allergy and clinical immunology, 2013-02, Vol.131 (2), p.AB197-AB197

2013

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Antagonistic Roles of Thymic Stromal Lymphopoietin (TSLP) and Surfactant Protein-D (SP-D) in Dendritic Cell Regulation During Ozone-Induced Exacerbation of Allergic Airway Inflammation in Mice

Journal of allergy and clinical immunology, 2013-02, Vol.131 (2), p.AB61-AB61

2013

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Ozone Inhalation Induces Epithelial IL-33 and Thymic Stromal Lymphopoietin (TSLP) and Leads To Eosinophilic Airway Inflammation

Journal of allergy and clinical immunology, 2014-02, Vol.133 (2), p.AB145-AB145

2014

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iPSC-derived NK cells maintain high cytotoxicity and enhance in vivo tumor control in concert with T cells and anti-PD-1 therapy

Science translational medicine, 2020-11, Vol.12 (568)

2020

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Deficiency of Melanoma Differentiation-associated Protein 5 Results in Exacerbated Chronic Postviral Lung Inflammation

American journal of respiratory and critical care medicine, 2014-02, Vol.189 (4), p.437-448

2014

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Harnessing features of adaptive NK cells to generate iPSC-derived NK cells for enhanced immunotherapy

Cell stem cell, 2021-12, Vol.28 (12), p.2062-2075.e5

2021

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Lung CD103+ dendritic cells efficiently transport influenza virus to the lymph node and load viral antigen onto MHC class I for presentation to CD8 T cells

The Journal of immunology (1950), 2011-12, Vol.187 (11), p.6011-6021

2011

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iPSC-Derived NK Cells Synergize with T Cells and Anti-PD-1 Antibody to Mediate Durable Anti-Tumor Responses In Vivo

Blood, 2019-11, Vol.134 (Supplement_1), p.1933-1933

2019

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Triple Gene-Modified iPSC-Derived NK Cells Combined with Daratumumab for Targeted Immunotherapy Against AML

Blood, 2020-11, Vol.136 (Supplement 1), p.57-58

2020

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NK cells regulate CD8+ T cell priming and dendritic cell migration during influenza A infection by IFN-γ and perforin-dependent mechanisms

The Journal of immunology (1950), 2012-09, Vol.189 (5), p.2099-2109

2012

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Group 2 innate lymphoid cells mediate ozone-induced airway inflammation and hyperresponsiveness in mice

mTORC2 regulates multiple aspects of NKT‐cell development and function

Oxidative damage of SP‐D abolishes control of eosinophil extracellular DNA trap formation

Ozone Inhalation Attenuated the Effects of Budesonide on Aspergillus fumigatus -Induced Airway Inflammation and Hyperreactivity in Mice

Diacylglycerol kinase ζ promotes allergic airway inflammation and airway hyperresponsiveness through distinct mechanisms

Adeno-associated virus 9-mediated airway expression of antibody protects old and immunodeficient mice against influenza virus

Surfactant Protein-D (SP-D) Is A Lung Specific Regulator of Group 2 Innate Lymphoid Cells (ILC2)

Antagonistic Effects Of Ozone (O3) Exposure and Glucocorticoid Treatment On Airway Hyperresponsiveness (AHR) and Surfactant Protein D (SP-D) Production In Mice

Circulating T cells, natural killer (NK) cells and group 2 innate lymphoid cells (ILC2) of severe asthmapatients from the University of California Asthma Network (UCANTM ) clinic, express increased levels of both IL-13 and IL-17

Inhalational exposure to statins and drug vehicle induces transient immunological changes in both the airways and peripheral blood of non-human primates

Role of Natural Killer (NK) Cell Surface Receptor –NKp46– in Primary Influenza A Infection

169 Off-the-shelf, engineered iPSC-derived NK cells mediate potent cytotoxic activity against primary glioblastoma cells and promote durable long-term survival in vivo

Group-2 Innate Lymphoid Cells Promote Air-Pollutant Induced Airway Inflammation and Hyperresponsiveness (AHR)

Epithelial IL-33 and TSLP Elicit Innate Lymphoid Cell Responses to Mediate Ozone-Induced Airway Inflammation and Hyperresponsiveness

155 iPSC-derived NK cells mediate robust anti-tumor activity against glioblastoma

Interactions of Natural Killer (NK) Cells and Surfactant Protein D (SP-D) in Regulation of Ozone Induced Airway Inflammation: Involvement of NKp46

Antagonistic Roles of Thymic Stromal Lymphopoietin (TSLP) and Surfactant Protein-D (SP-D) in Dendritic Cell Regulation During Ozone-Induced Exacerbation of Allergic Airway Inflammation in Mice

Ozone Inhalation Induces Epithelial IL-33 and Thymic Stromal Lymphopoietin (TSLP) and Leads To Eosinophilic Airway Inflammation

iPSC-derived NK cells maintain high cytotoxicity and enhance in vivo tumor control in concert with T cells and anti-PD-1 therapy

Deficiency of Melanoma Differentiation-associated Protein 5 Results in Exacerbated Chronic Postviral Lung Inflammation

Harnessing features of adaptive NK cells to generate iPSC-derived NK cells for enhanced immunotherapy

Lung CD103+ dendritic cells efficiently transport influenza virus to the lymph node and load viral antigen onto MHC class I for presentation to CD8 T cells

iPSC-Derived NK Cells Synergize with T Cells and Anti-PD-1 Antibody to Mediate Durable Anti-Tumor Responses In Vivo

Triple Gene-Modified iPSC-Derived NK Cells Combined with Daratumumab for Targeted Immunotherapy Against AML

NK cells regulate CD8+ T cell priming and dendritic cell migration during influenza A infection by IFN-γ and perforin-dependent mechanisms

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