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The N550K/H Mutations in FGFR2 Confer Differential Resistance to PD173074, Dovitinib, and Ponatinib ATP-Competitive Inhibitors

Neoplasia (New York, N.Y.), 2013-08, Vol.15 (8), p.975-IN30

2013

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Mixed lineage kinases activate MEK independently of RAF to mediate resistance to RAF inhibitors

Nature communications, 2014-05, Vol.5 (1), p.3901, Article 3901

2014

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Inhibition of Activated Fibroblast Growth Factor Receptor 2 in Endometrial Cancer Cells Induces Cell Death Despite PTEN Abrogation

Cancer research (Chicago, Ill.), 2008-09, Vol.68 (17), p.6902-6907

2008

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Bcl‐2 inhibitors enhance FGFR inhibitor‐induced mitochondrial‐dependent cell death in FGFR2‐mutant endometrial cancer

Molecular oncology, 2019-04, Vol.13 (4), p.738-756

2019

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Loss-of-Function Fibroblast Growth Factor Receptor-2 Mutations in Melanoma

Molecular cancer research, 2009-01, Vol.7 (1), p.41-54

2009

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Conditional Inactivation of the Men1 Gene Leads to Pancreatic and Pituitary Tumorigenesis but Does Not Affect Normal Development of These Tissues

Molecular and Cellular Biology, 2004-04, Vol.24 (8), p.3125-3131

2004

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FGFR2 mutations are rare across histologic subtypes of ovarian cancer

Gynecologic oncology, 2010-04, Vol.117 (1), p.125-129

2010

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C-reactive protein is a prognostic biomarker in pancreatic ductal adenocarcinoma patients

Asia-Pacific journal of clinical oncology, 2023-07

2023

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Melanomas of unknown primary have a mutation profile consistent with cutaneous sun-exposed melanoma

Pigment cell and melanoma research, 2013-11, Vol.26 (6), p.852-860

2013

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Classification of esophageal adenocarcinoma into subgroups that are associated with patient survival using RNA-seq and immunohistochemistry

Journal of clinical oncology, 2022-06, Vol.40 (16_suppl), p.e16028-e16028

2022

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Abstract C86: FGFR inhibition potentiates the effect of conventional chemotherapeutic agents in endometrial cancer cells

Molecular cancer therapeutics, 2009-12, Vol.8 (12_Supplement), p.C86-C86

2009

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Abstract 20: POT1 mutations predispose to familial melanoma

Cancer research (Chicago, Ill.), 2014-12, Vol.74 (23_Supplement), p.20-20

2014

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The N550K/H Mutations in FGFR2 Confer Differential Resistance to PD173074, Dovitinib, and Ponatinib ATP-Competitive Inhibitors12

Neoplasia (New York, N.Y.), 2013-08, Vol.15 (8), p.975-988

2013

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Evidence for persistent dysfunction of wild-type aldosterone synthase gene in glucocorticoid-treated familial hyperaldosteronism type I

Journal of hypertension, 1997-12, Vol.15 (12), p.1729-1733

1997

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In familial hyperaldosteronism type I, hybrid gene-induced aldosterone production dominates that induced by Wild-type genes

The journal of clinical endocrinology and metabolism, 1997-11, Vol.82 (11), p.3670-3676

1997

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In Familial Hyperaldosteronism Type I, Hybrid Gene-Induced Aldosterone Production Dominates That Induced by Wild-Type Genes 1

The journal of clinical endocrinology and metabolism, 1997-11, Vol.82 (11), p.3670-3676

1997

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In Familial Hyperaldosteronism Type I, Hybrid Gene-Induced Aldosterone Production Dominates That Induced by Wild-Type Genes1

The journal of clinical endocrinology and metabolism, 1997-11, Vol.82 (11), p.3670-3676

1997

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Peripheral blood lymphocyte proliferative responses in cattle infected with or vaccinated against Anaplasma marginale

Parasitology research (1987), 1996, Vol.82 (6), p.551-562

1996

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FGFR2 point mutations in 466 endometrioid endometrial tumors: relationship with MSI, KRAS, PIK3CA, CTNNB1 mutations and clinicopathological features

PloS one, 2012-02, Vol.7 (2), p.e30801-e30801

2012

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Nonsense mutations in the shelterin complex genes ACD and TERF2IP in familial melanoma

JNCI : Journal of the National Cancer Institute, 2015-02, Vol.107 (2), p.1

2015

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Complex structural rearrangements are present in high-grade dysplastic Barrett's oesophagus samples

BMC medical genomics, 2019-02, Vol.12 (1), p.31-31, Article 31

2019

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The Impact of Signet Ring Cell Differentiation on Outcome in Patients with Esophageal and Gastroesophageal Junction Adenocarcinoma

Annals of surgical oncology, 2019-08, Vol.26 (8), p.2375-2384

2019

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The N550K/H Mutations in FGFR2 Confer Differential Resistance to PD173074, Dovitinib, and Ponatinib ATP-Competitive Inhibitors

Frequent somatic mutations in MAP3K5 and MAP3K9 in metastatic melanoma identified by exome sequencing

Mixed lineage kinases activate MEK independently of RAF to mediate resistance to RAF inhibitors

Inhibition of Activated Fibroblast Growth Factor Receptor 2 in Endometrial Cancer Cells Induces Cell Death Despite PTEN Abrogation

Bcl‐2 inhibitors enhance FGFR inhibitor‐induced mitochondrial‐dependent cell death in FGFR2‐mutant endometrial cancer

Loss-of-Function Fibroblast Growth Factor Receptor-2 Mutations in Melanoma

Conditional Inactivation of the Men1 Gene Leads to Pancreatic and Pituitary Tumorigenesis but Does Not Affect Normal Development of These Tissues

FGFR2 mutations are rare across histologic subtypes of ovarian cancer

POT1 loss-of-function variants predispose to familial melanoma

C-reactive protein is a prognostic biomarker in pancreatic ductal adenocarcinoma patients

Melanomas of unknown primary have a mutation profile consistent with cutaneous sun-exposed melanoma

Classification of esophageal adenocarcinoma into subgroups that are associated with patient survival using RNA-seq and immunohistochemistry

Abstract C86: FGFR inhibition potentiates the effect of conventional chemotherapeutic agents in endometrial cancer cells

Abstract 20: POT1 mutations predispose to familial melanoma

The N550K/H Mutations in FGFR2 Confer Differential Resistance to PD173074, Dovitinib, and Ponatinib ATP-Competitive Inhibitors12

Evidence for persistent dysfunction of wild-type aldosterone synthase gene in glucocorticoid-treated familial hyperaldosteronism type I

Frequent somatic MAP3K5 and MAP3K9 mutations in metastatic melanoma identified by exome sequencing

In familial hyperaldosteronism type I, hybrid gene-induced aldosterone production dominates that induced by Wild-type genes

In Familial Hyperaldosteronism Type I, Hybrid Gene-Induced Aldosterone Production Dominates That Induced by Wild-Type Genes 1

In Familial Hyperaldosteronism Type I, Hybrid Gene-Induced Aldosterone Production Dominates That Induced by Wild-Type Genes1

Peripheral blood lymphocyte proliferative responses in cattle infected with or vaccinated against Anaplasma marginale

FGFR2 point mutations in 466 endometrioid endometrial tumors: relationship with MSI, KRAS, PIK3CA, CTNNB1 mutations and clinicopathological features

Nonsense mutations in the shelterin complex genes ACD and TERF2IP in familial melanoma

Complex structural rearrangements are present in high-grade dysplastic Barrett's oesophagus samples

The Impact of Signet Ring Cell Differentiation on Outcome in Patients with Esophageal and Gastroesophageal Junction Adenocarcinoma

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