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Methods Used to Evaluate Pain Behaviors in Rodents

Frontiers in molecular neuroscience, 2017-09, Vol.10, p.284-284

2017

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An animal model of oxaliplatin-induced cold allodynia reveals a crucial role for Nav1.6 in peripheral pain pathways

Pain (Amsterdam), 2013-09, Vol.154 (9), p.1749-1757

2013

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Development of a high-throughput fluorescent no-wash sodium influx assay

PloS one, 2019-03, Vol.14 (3), p.e0213751-e0213751

2019

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Ant venoms contain vertebrate-selective pain-causing sodium channel toxins

Nature communications, 2023-05, Vol.14 (1), p.2977-2977, Article 2977

2023

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Natural Compounds Purified from the Leaves of Aristotelia chilensis: Makomakinol, a New Alkaloid and the Effect of Aristoteline and Hobartine on NaV Channels

International journal of molecular sciences, 2023-11, Vol.24 (21), p.15504

2023

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The structure, dynamics and selectivity profile of a NaV1.7 potency-optimised huwentoxin-IV variant

PloS one, 2017-03, Vol.12 (3), p.e0173551

2017

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Analgesic Effects of GpTx-1, PF-04856264 and CNV1014802 in a Mouse Model of NaV1.7-Mediated Pain

Toxins, 2016-03, Vol.8 (3), p.78

2016

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Venom composition and pain-causing toxins of the Australian great carpenter bee Xylocopa aruana

Scientific reports, 2022-12, Vol.12 (1), p.22168-22168, Article 22168

2022

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Discovery and mode of action of a novel analgesic β-toxin from the African spider Ceratogyrus darlingi

PloS one, 2017, Vol.12 (9), p.e0182848-e0182848

2017

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Towards a generic prototyping approach for therapeutically-relevant peptides and proteins in a cell-free translation system

Nature communications, 2022-01, Vol.13 (1), p.260-260, Article 260

2022

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µ-Conotoxins Targeting the Human Voltage-Gated Sodium Channel Subtype NaV1.7

Toxins, 2022-08, Vol.14 (9), p.600

2022

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The ‘dispanins’ and related proteins in physiology and neurological disease

Trends in neurosciences (Regular ed.), 2024-08, Vol.47 (8), p.622-634

2024

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The pharmacology of voltage-gated sodium channel activators

Neuropharmacology, 2017-12, Vol.127, p.87-108

2017

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NaV1.7 as a pain target – From gene to pharmacology

Pharmacology & therapeutics (Oxford), 2017-04, Vol.172, p.73-100

2017

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The Tarantula Venom Peptide Eo1a Binds to the Domain II S3-S4 Extracellular Loop of Voltage-Gated Sodium Channel NaV1.8 to Enhance Activation

Frontiers in pharmacology, 2022-01, Vol.12, p.789570-789570

2022

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Discovery, Pharmacological Characterisation and NMR Structure of the Novel µ-Conotoxin SxIIIC, a Potent and Irreversible NaV Channel Inhibitor

Biomedicines, 2020-10, Vol.8 (10), p.391

2020

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Inflammatory and Neuropathic Gene Expression Signatures of Chemotherapy-Induced Neuropathy Induced by Vincristine, Cisplatin, and Oxaliplatin in C57BL/6J Mice

The journal of pain, 2020-01, Vol.21 (1-2), p.182-194

2020

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A venom peptide-induced NaV channel modulation mechanism involving the interplay between fixed channel charges and ionic gradients

The Journal of biological chemistry, 2024-10, Vol.300 (10), p.107757, Article 107757

2024

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Venom-derived pain-causing toxins: insights into sensory neuron function and pain mechanisms

Pain (Amsterdam), 2022-11, Vol.163 (S1), p.S46-S56

2022

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Characterization of Synthetic Tf2 as a NaV1.3 Selective Pharmacological Probe

Biomedicines, 2020-06, Vol.8 (6), p.155

2020

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Sodium Channels and Venom Peptide Pharmacology

Advances in pharmacology (1990), 2017, Vol.79, p.67-116

2017

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Erythromelalgia caused by the missense mutation p.Arg220Pro in an alternatively spliced exon of SCN9A (NaV1.7)

Human molecular genetics, 2024-01, Vol.33 (2), p.103-109

2024

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Recombinant production, bioconjugation and membrane binding studies ofPn3a, a selective NaV1.7 inhibitor

Biochemical pharmacology, 2020-11, Vol.181, p.114148-114148, Article 114148

2020

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Methods Used to Evaluate Pain Behaviors in Rodents

An animal model of oxaliplatin-induced cold allodynia reveals a crucial role for Nav1.6 in peripheral pain pathways

Development of a high-throughput fluorescent no-wash sodium influx assay

Ant venoms contain vertebrate-selective pain-causing sodium channel toxins

Natural Compounds Purified from the Leaves of Aristotelia chilensis: Makomakinol, a New Alkaloid and the Effect of Aristoteline and Hobartine on NaV Channels

The structure, dynamics and selectivity profile of a NaV1.7 potency-optimised huwentoxin-IV variant

Analgesic Effects of GpTx-1, PF-04856264 and CNV1014802 in a Mouse Model of NaV1.7-Mediated Pain

Venom composition and pain-causing toxins of the Australian great carpenter bee Xylocopa aruana

Pain-causing stinging nettle toxins target TMEM233 to modulate NaV1.7 function

Discovery and mode of action of a novel analgesic β-toxin from the African spider Ceratogyrus darlingi

Towards a generic prototyping approach for therapeutically-relevant peptides and proteins in a cell-free translation system

µ-Conotoxins Targeting the Human Voltage-Gated Sodium Channel Subtype NaV1.7

The Snake with the Scorpion's Sting: Novel Three-Finger Toxin Sodium Channel Activators from the Venom of the Long-Glanded Blue Coral Snake (Calliophis bivirgatus)

The ‘dispanins’ and related proteins in physiology and neurological disease

The pharmacology of voltage-gated sodium channel activators

NaV1.7 as a pain target – From gene to pharmacology

The Tarantula Venom Peptide Eo1a Binds to the Domain II S3-S4 Extracellular Loop of Voltage-Gated Sodium Channel NaV1.8 to Enhance Activation

Discovery, Pharmacological Characterisation and NMR Structure of the Novel µ-Conotoxin SxIIIC, a Potent and Irreversible NaV Channel Inhibitor

Inflammatory and Neuropathic Gene Expression Signatures of Chemotherapy-Induced Neuropathy Induced by Vincristine, Cisplatin, and Oxaliplatin in C57BL/6J Mice

A venom peptide-induced NaV channel modulation mechanism involving the interplay between fixed channel charges and ionic gradients

Venom-derived pain-causing toxins: insights into sensory neuron function and pain mechanisms

Characterization of Synthetic Tf2 as a NaV1.3 Selective Pharmacological Probe

Sodium Channels and Venom Peptide Pharmacology

Erythromelalgia caused by the missense mutation p.Arg220Pro in an alternatively spliced exon of SCN9A (NaV1.7)

Recombinant production, bioconjugation and membrane binding studies ofPn3a, a selective NaV1.7 inhibitor

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