UB Paderborn / Katalog / Suche / in CDI

Sortiert nach

Einfacher Volltextzugriff von (fast) überall

Installieren Sie die LibKey Nomad-Browsererweiterung und erhalten Sie von vielen Webseiten einfachen Zugriff auf lizenzierte Volltexte oder Open Access Publikationen. Mehr erfahren...

New Insights into Cardiac Ion Channel Regulation 2.0

International journal of molecular sciences, 2023-03, Vol.24 (5), p.4999

2023

Volltextzugriff (PDF)

Time Restricted Feeding to the Light Cycle Dissociates Canonical Circadian Clocks and Physiological Rhythms in Heart Rate

Frontiers in pharmacology, 2022-05, Vol.13, p.910195-910195

2022

Volltextzugriff (PDF)

Potassium channels in the heart: structure, function and regulation

The Journal of physiology, 2017-04, Vol.595 (7), p.2209-2228

2017

Volltextzugriff (PDF)

Defining the age-dependent and tissue-specific circadian transcriptome in male mice

Cell reports (Cambridge), 2023-01, Vol.42 (1), p.111982-111982, Article 111982

2023

Volltextzugriff (PDF)

Editorial: Perturbations in Metabolic Cues: Implications for Adverse Cardiac Function Leading to Sudden Cardiac Death

Frontiers in physiology, 2021-11, Vol.12, p.788904-788904

2021

Volltextzugriff (PDF)

The cardiomyocyte molecular clock regulates the circadian expression of Kcnh2 and contributes to ventricular repolarization

Heart rhythm, 2015-06, Vol.12 (6), p.1306-1314

2015

Volltextzugriff (PDF)

The cardiomyocyte molecular clock, regulation of Scn5a, and arrhythmia susceptibility

American Journal of Physiology: Cell Physiology, 2013-05, Vol.304 (10), p.C954-C965

2013

Volltextzugriff (PDF)

Long QT syndrome-associated mutations in intrauterine fetal death

JAMA : the journal of the American Medical Association, 2013-04, Vol.309 (14), p.1473

2013

Volltextzugriff (PDF)

Most LQT2 mutations reduce Kv11.1 (hERG) current by a class 2 (trafficking-deficient) mechanism

Circulation (New York, N.Y.), 2006-01, Vol.113 (3), p.365-373

2006

Volltextzugriff (PDF)

Mutation-Specific Differences in Kv7.1 ( KCNQ1 ) and Kv11.1 ( KCNH2 ) Channel Dysfunction and Long QT Syndrome Phenotypes

International journal of molecular sciences, 2022-07, Vol.23 (13), p.7389

2022

Volltextzugriff (PDF)

Prediction of mortality from 12-lead electrocardiogram voltage data using a deep neural network

Nature medicine, 2020-06, Vol.26 (6), p.886-891

2020

Volltextzugriff (PDF)

Molecular pathogenesis of long QT syndrome type 2

Journal of arrhythmia, 2016-10, Vol.32 (5), p.373-380

2016

Volltextzugriff (PDF)

A Molecular Mechanism for Adrenergic-Induced Long QT Syndrome

Journal of the American College of Cardiology, 2014-03, Vol.63 (8), p.819-827

2014

Volltextzugriff (PDF)

Drug‐induced long QT syndrome: hERG K+ channel block and disruption of protein trafficking by fluoxetine and norfluoxetine

British journal of pharmacology, 2006-11, Vol.149 (5), p.481-489

2006

Volltextzugriff (PDF)

Long QT Syndrome Type 2: Emerging Strategies for Correcting Class 2 KCNH2 ( hERG ) Mutations and Identifying New Patients

Biomolecules (Basel, Switzerland), 2020-08, Vol.10 (8), p.1144

2020

Volltextzugriff (PDF)

Cardiomyocyte Deletion of Bmal1 Exacerbates QT- and RR-Interval Prolongation in Scn5a+/ΔKPQ Mice

Frontiers in physiology, 2021-06, Vol.12, p.681011-681011

2021

Volltextzugriff (PDF)

Dim light at night unmasks sex-specific differences in circadian and autonomic regulation of cardiovascular physiology

Communications biology, 2024-09, Vol.7 (1), p.1191-14, Article 1191

2024

Volltextzugriff (PDF)

How Functional Genomics Can Keep Pace With VUS Identification

Frontiers in cardiovascular medicine, 2022-07, Vol.9, p.900431

2022

Volltextzugriff (PDF)

Caution: merging ion channel traffic ahead

The Journal of physiology, 2023-07, Vol.601 (13), p.2541-2542

2023

Volltextzugriff (PDF)

A KCNQ1 Mutation Causes a High Penetrance for Familial Atrial Fibrillation

Journal of cardiovascular electrophysiology, 2013-05, Vol.24 (5), p.562-569

2013

Volltextzugriff (PDF)

R231C mutation in KCNQ1 causes long QT syndrome type 1 and familial atrial fibrillation

Heart rhythm, 2011-01, Vol.8 (1), p.48-55

2011

Volltextzugriff (PDF)

Editorial: Ion Channel Trafficking and Cardiac Arrhythmias

Frontiers in physiology, 2018-09, Vol.9, p.1254-1254

2018

Volltextzugriff (PDF)

Detecting organelle-specific activity of potassium channels with a DNA nanodevice

Nature biotechnology, 2024-07, Vol.42 (7), p.1065-1074

2024

Volltextzugriff (PDF)

Biology of Cardiac Arrhythmias: Ion Channel Protein Trafficking

Circulation research, 2004-06, Vol.94 (11), p.1418-1428

2004

Volltextzugriff (PDF)

Pharmacological correction of long QT-linked mutations in KCNH2 (hERG) increases the trafficking of Kv11.1 channels stored in the transitional endoplasmic reticulum

American Journal of Physiology: Cell Physiology, 2013-11, Vol.305 (9), p.C919-C930

2013

Volltextzugriff (PDF)

1 – 25 von 727

Suche als RSS-Feed

Suchergebnisse filtern

Filter anzeigen

Nur zeigen

Geprüft (Peer-Review)
(624)
Mit Volltextzugriff
(536)

Publikationsform

4 weitere anzeigen...

Weniger anzeigen...

Sprache

Erscheinungsjahr

n.n

Thema

15 weitere anzeigen...

Weniger anzeigen...

Zeitschrift / Serie

5 weitere anzeigen...

Weniger anzeigen...

Neu hinzugefügt

... seit 30 Tagen
(2)
... seit 90 Tagen
(5)

Menü

Weitere Dienste

Einstellungen

New Insights into Cardiac Ion Channel Regulation 2.0

Time Restricted Feeding to the Light Cycle Dissociates Canonical Circadian Clocks and Physiological Rhythms in Heart Rate

Potassium channels in the heart: structure, function and regulation

Defining the age-dependent and tissue-specific circadian transcriptome in male mice

Editorial: Perturbations in Metabolic Cues: Implications for Adverse Cardiac Function Leading to Sudden Cardiac Death

The cardiomyocyte molecular clock regulates the circadian expression of Kcnh2 and contributes to ventricular repolarization

The cardiomyocyte molecular clock, regulation of Scn5a, and arrhythmia susceptibility

Long QT syndrome-associated mutations in intrauterine fetal death

Most LQT2 mutations reduce Kv11.1 (hERG) current by a class 2 (trafficking-deficient) mechanism

Mutation-Specific Differences in Kv7.1 ( KCNQ1 ) and Kv11.1 ( KCNH2 ) Channel Dysfunction and Long QT Syndrome Phenotypes

Prediction of mortality from 12-lead electrocardiogram voltage data using a deep neural network

Molecular pathogenesis of long QT syndrome type 2

A Molecular Mechanism for Adrenergic-Induced Long QT Syndrome

Drug‐induced long QT syndrome: hERG K+ channel block and disruption of protein trafficking by fluoxetine and norfluoxetine

Long QT Syndrome Type 2: Emerging Strategies for Correcting Class 2 KCNH2 ( hERG ) Mutations and Identifying New Patients

Cardiomyocyte Deletion of Bmal1 Exacerbates QT- and RR-Interval Prolongation in Scn5a+/ΔKPQ Mice

Dim light at night unmasks sex-specific differences in circadian and autonomic regulation of cardiovascular physiology

How Functional Genomics Can Keep Pace With VUS Identification

Caution: merging ion channel traffic ahead

A KCNQ1 Mutation Causes a High Penetrance for Familial Atrial Fibrillation

R231C mutation in KCNQ1 causes long QT syndrome type 1 and familial atrial fibrillation

Editorial: Ion Channel Trafficking and Cardiac Arrhythmias

Detecting organelle-specific activity of potassium channels with a DNA nanodevice

Biology of Cardiac Arrhythmias: Ion Channel Protein Trafficking

Pharmacological correction of long QT-linked mutations in KCNH2 (hERG) increases the trafficking of Kv11.1 channels stored in the transitional endoplasmic reticulum

Suchergebnisse filtern