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Loss of p53 induces epidermal growth factor receptor promoter activity in normal human keratinocytes

Oncogene, 2008-07, Vol.27 (31), p.4315-4323

2008

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Case-control study of candidate gene methylation and adenomatous polyp formation

International journal of colorectal disease, 2017-02, Vol.32 (2), p.183-192

2017

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Population-based, case-control study of HER2 genetic polymorphism and breast cancer risk

JNCI : Journal of the National Cancer Institute, 2000-03, Vol.92 (5), p.412-417

2000

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Dose-dependent benefits of quercetin on tumorigenesis in the C3(1)/SV40Tag transgenic mouse model of breast cancer

Cancer biology & therapy, 2014-01, Vol.15 (11), p.1456-1467

2014

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Human papillomavirus type 16 E6 and E7 cooperate to increase epidermal growth factor receptor (EGFR) mRNA levels, overcoming mechanisms by which excessive EGFR signaling shortens the life span of normal human keratinocytes

Cancer research (Baltimore), 2001-05, Vol.61 (9), p.3837-3843

2001

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Evaluation of a human bio-engineered skin equivalent for drug permeation studies

Pharmaceutical research, 2000-09, Vol.17 (9), p.1092-1097

2000

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Six1 promotes epithelial-mesenchymal transition and malignant conversion in human papillomavirus type 16-immortalized human keratinocytes

Carcinogenesis (New York), 2014-06, Vol.35 (6), p.1379-1388

2014

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Unique carboxyl-terminal sequences of wild type and alternatively spliced variant forms of transforming growth factor-α precursors mediate specific interactions with ErbB4 and ErbB2

Oncogene, 2000-06, Vol.19 (28), p.3172-3181

2000

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Human keratinocytes and tumor-derived cell lines express alternatively spliced forms of transforming growth factor-α mRNA, encoding precursors lacking carboxyl-terminal valine residues

Oncogene, 1999-09, Vol.18 (40), p.5554-5562

1999

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Continuous cell lines with altered growth and differentiation properties originate after transfection of human keratinocytes with human papillomavirus type 16 DNA

Carcinogenesis (New York), 1988-09, Vol.9 (9), p.1573-1579

1988

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MS for Identification of Single Nucleotide Polymorphisms and MS/MS for Discrimination of Isomeric PCR Products

Analytical chemistry (Washington), 2000-09, Vol.72 (17), p.4033-4040

2000

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Evaluation of Transdermal Penetration Enhancers Using A Novel Skin Alternative

Journal of pharmaceutical sciences, 1997-09, Vol.86 (9), p.1001-1005

1997

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Increased sensitivity of human keratinocytes immortalized by human papillomavirus type 16 DNA to growth control by retinoids

Cancer research (Chicago, Ill.), 1992, Vol.52 (1), p.187-193

1992

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Unique SNP in CD44 Intron 1 and its Role in Breast Cancer Development

Anticancer research, 2010-04, Vol.30 (4), p.1263-1272

2010

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Genotyping single nucleotide polymorphisms using intact polymerase chain reaction products by electrospray quadrupole mass spectrometry

Rapid communications in mass spectrometry, 2001-01, Vol.15 (18), p.1752-1759

2001

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Retinoic acid inhibition of human papillomavirus type 16-mediated transformation of human keratinocytes

Cancer research (Chicago, Ill.), 1993-02, Vol.53 (4), p.905-909

1993

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A comparison of the uptake, metabolism and biologic effects of retinol delivered to human keratinocytes either free or bound to serum retinol-binding protein

The Journal of nutrition, 1993-02, Vol.123 (2), p.356-361

1993

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Natural Gas Hydrates in Flow Assurance

1, 2011

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Retinoic Acid Resistance at Late Stages of Human Papillomavirus Type 16-Mediated Transformation of Human Keratinocytes Arises Despite Intact Retinoid Signaling and Is Due to a Loss of Sensitivity to Transforming Growth Factor-β

Virology (New York, N.Y.), 2000-05, Vol.270 (2), p.397-407

2000

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Progressive loss of sensitivity to growth control by retinoic acid and transforming growth factor-beta at late stages of human papillomavirus type 16-initiated transformation of human keratinocytes

Advances in experimental medicine and biology, 1995, Vol.375, p.117

1995

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Comparison of the Metabolism of Retinol Delivered to Human Keratinocytes either Bound to Serum Retinol-Binding Protein or Added Directly to the Culture Medium

Experimental cell research, 1998-01, Vol.238 (1), p.257-264

1998

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Loss of Transforming Growth Factor-β (TGF-β) Receptor Type I Mediates TGF-β Resistance in Human Papillomavirus Type 16-Transformed Human Keratinocytes at Late Stages of in Vitro Progression

Virology (New York, N.Y.), 2000-05, Vol.270 (2), p.408-416

2000

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Comparison of the Rate of Uptake and Biologic Effects of Retinol Added to Human Keratinocytes Either Directly to the Culture Medium or Bound to Serum Retinol-Binding Protein

Journal of investigative dermatology, 1991-08, Vol.97 (2), p.298-304

1991

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Uptake and metabolism of [ 3H]retinoic acid delivered to human foreskin keratinocytes either bound to serum albumin or added directly to the culture medium

Biochimica et biophysica acta, 1996-04, Vol.1311 (2), p.102-110

1996

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Comparison of the Uptake and Metabolism of Retinol Delivered to Primary Mouse Keratinocytes Either Free or Bound to Rat Serum Retinol-binding Protein

Journal of investigative dermatology, 1989-02, Vol.92 (2), p.283-289

1989

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Loss of p53 induces epidermal growth factor receptor promoter activity in normal human keratinocytes

Case-control study of candidate gene methylation and adenomatous polyp formation

Population-based, case-control study of HER2 genetic polymorphism and breast cancer risk

Dose-dependent benefits of quercetin on tumorigenesis in the C3(1)/SV40Tag transgenic mouse model of breast cancer

Human papillomavirus type 16 E6 and E7 cooperate to increase epidermal growth factor receptor (EGFR) mRNA levels, overcoming mechanisms by which excessive EGFR signaling shortens the life span of normal human keratinocytes

Evaluation of a human bio-engineered skin equivalent for drug permeation studies

Six1 promotes epithelial-mesenchymal transition and malignant conversion in human papillomavirus type 16-immortalized human keratinocytes

Unique carboxyl-terminal sequences of wild type and alternatively spliced variant forms of transforming growth factor-α precursors mediate specific interactions with ErbB4 and ErbB2

Human keratinocytes and tumor-derived cell lines express alternatively spliced forms of transforming growth factor-α mRNA, encoding precursors lacking carboxyl-terminal valine residues

Continuous cell lines with altered growth and differentiation properties originate after transfection of human keratinocytes with human papillomavirus type 16 DNA

MS for Identification of Single Nucleotide Polymorphisms and MS/MS for Discrimination of Isomeric PCR Products

Evaluation of Transdermal Penetration Enhancers Using A Novel Skin Alternative

Increased sensitivity of human keratinocytes immortalized by human papillomavirus type 16 DNA to growth control by retinoids

Unique SNP in CD44 Intron 1 and its Role in Breast Cancer Development

Genotyping single nucleotide polymorphisms using intact polymerase chain reaction products by electrospray quadrupole mass spectrometry

Retinoic acid inhibition of human papillomavirus type 16-mediated transformation of human keratinocytes

A comparison of the uptake, metabolism and biologic effects of retinol delivered to human keratinocytes either free or bound to serum retinol-binding protein

Natural Gas Hydrates in Flow Assurance

Retinoic Acid Resistance at Late Stages of Human Papillomavirus Type 16-Mediated Transformation of Human Keratinocytes Arises Despite Intact Retinoid Signaling and Is Due to a Loss of Sensitivity to Transforming Growth Factor-β

Progressive loss of sensitivity to growth control by retinoic acid and transforming growth factor-beta at late stages of human papillomavirus type 16-initiated transformation of human keratinocytes

Comparison of the Metabolism of Retinol Delivered to Human Keratinocytes either Bound to Serum Retinol-Binding Protein or Added Directly to the Culture Medium

Loss of Transforming Growth Factor-β (TGF-β) Receptor Type I Mediates TGF-β Resistance in Human Papillomavirus Type 16-Transformed Human Keratinocytes at Late Stages of in Vitro Progression

Comparison of the Rate of Uptake and Biologic Effects of Retinol Added to Human Keratinocytes Either Directly to the Culture Medium or Bound to Serum Retinol-Binding Protein

Uptake and metabolism of [ 3H]retinoic acid delivered to human foreskin keratinocytes either bound to serum albumin or added directly to the culture medium

Comparison of the Uptake and Metabolism of Retinol Delivered to Primary Mouse Keratinocytes Either Free or Bound to Rat Serum Retinol-binding Protein

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