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Fifty years of gating currents and channel gating

The Journal of general physiology, 2023-08, Vol.155 (8), p.1

2023

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Inhibition of Bax Channel-Forming Activity by Bcl-2

Science (American Association for the Advancement of Science), 1997-07, Vol.277 (5324), p.370-372

1997

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Endogenous expression of the β1A sodium channel subunit in HEK-293 cells

FEBS letters, 2000-05, Vol.473 (2), p.132-134

2000

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Activation and Inactivation of Homomeric KvLQT1 Potassium Channels

Biophysical journal, 1998-08, Vol.75 (2), p.785-792

1998

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Structural parts involved in activation and inactivation of the sodium channel

Nature (London), 1989-06, Vol.339 (6226), p.597-603

1989

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Mapping the site of block by tetrodotoxin and saxitoxin of sodium channel II

FEBS letters, 1991-11, Vol.293 (1), p.93-96

1991

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[7] Nonstationary noise analysis and application to patch clamp recordings

Methods in Enzymology, 1992, Vol.207, p.131-148

1992

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The muscle chloride channel ClC-1 has a double-barreled appearance that is differentially affected in dominant and recessive myotonia

The Journal of general physiology, 1999-03, Vol.113 (3), p.457-468

1999

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Modulation of Sodium Current in Mammalian Cells by an Epilepsy-Correlated β1-Subunit Mutation

Biochemical and biophysical research communications, 2002, Vol.291 (4), p.1095-1101

2002

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Sodium channel heterologous expression in mammalian cells and the role of the endogenous β1-subunits

Neuroscience letters, 2003, Vol.336 (3), p.175-179

2003

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Modulation of sodium current in mammalian cells by an epilepsy-correlated beta 1-subunit mutation

Biochemical and biophysical research communications, 2002-03, Vol.291 (4), p.1095-1101

2002

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Tonic and Phasic Guanidinium Toxin-Block of Skeletal Muscle Na Channels Expressed in Mammalian Cells

Biophysical journal, 2003-05, Vol.84 (5), p.2999-3006

2003

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Pursuing the structure and function of voltage-gated channels

Trends in neurosciences (Regular ed.), 1990-06, Vol.13 (6), p.201-206

1990

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Gating and Flickery Block Differentially Affected by Rubidium in Homomeric KCNQ1 and Heteromeric KCNQ1/KCNE1 Potassium Channels

Biophysical journal, 2000, Vol.78 (1), p.211-226

2000

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The Binding of κ-Conotoxin PVIIA and Fast C-Type Inactivation of Shaker K + Channels are Mutually Exclusive

Biophysical journal, 2004, Vol.86 (1), p.191-209

2004

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Skeletal Muscle Sodium Channel Is Affected by an Epileptogenic β1 Subunit Mutation

Biochemical and biophysical research communications, 2001-03, Vol.282 (1), p.55-59

2001

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Sodium channel heterologous expression in mammalian cells and the role of the endogenous beta1-subunits

Neuroscience letters, 2003-01, Vol.336 (3), p.175-179

2003

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The Binding of [kappa]-Conotoxin PVIIA and Fast C-Type Inactivation of Shaker K^sup +^ Channels are Mutually Exclusive

Biophysical journal, 2004-01, Vol.86 (1), p.191

2004

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Binding of kappa-conotoxin PVIIA to Shaker K+ channels reveals different K+ and Rb+ occupancies within the ion channel pore

The Journal of general physiology, 2004-07, Vol.124 (1), p.71-81

2004

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Disposition of metformin (N,N‐dimethylbiguanide) in man

Clinical pharmacology and therapeutics, 1978-12, Vol.24 (6), p.683-693

1978

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The block of Shaker K+ channels by kappa-conotoxin PVIIA is state dependent

The Journal of general physiology, 1999-07, Vol.114 (1), p.125-140

1999

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Mechanism of block of single protopores of the Torpedo chloride channel ClC-0 by 2-(p-chlorophenoxy)butyric acid (CPB)

The Journal of general physiology, 2001-07, Vol.118 (1), p.45-62

2001

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Mechanisms of block of muscle type CLC chloride channels (Review)

Molecular membrane biology, 2002-10, Vol.19 (4), p.285-292

2002

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Tonic and Phasic Tetrodotoxin Block of Sodium Channels with Point Mutations in the Outer Pore Region

Biophysical journal, 1999-07, Vol.77 (1), p.229-240

1999

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Functional Properties of Sodium Channels Do Not Depend on the Cytoskeleton Integrity

Biochemical and biophysical research communications, 2000-09, Vol.276 (1), p.204-209

2000

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Fifty years of gating currents and channel gating

Inhibition of Bax Channel-Forming Activity by Bcl-2

Endogenous expression of the β1A sodium channel subunit in HEK-293 cells

Activation and Inactivation of Homomeric KvLQT1 Potassium Channels

Structural parts involved in activation and inactivation of the sodium channel

Mapping the site of block by tetrodotoxin and saxitoxin of sodium channel II

[7] Nonstationary noise analysis and application to patch clamp recordings

The muscle chloride channel ClC-1 has a double-barreled appearance that is differentially affected in dominant and recessive myotonia

Modulation of Sodium Current in Mammalian Cells by an Epilepsy-Correlated β1-Subunit Mutation

Sodium channel heterologous expression in mammalian cells and the role of the endogenous β1-subunits

Modulation of sodium current in mammalian cells by an epilepsy-correlated beta 1-subunit mutation

Tonic and Phasic Guanidinium Toxin-Block of Skeletal Muscle Na Channels Expressed in Mammalian Cells

Pursuing the structure and function of voltage-gated channels

Gating and Flickery Block Differentially Affected by Rubidium in Homomeric KCNQ1 and Heteromeric KCNQ1/KCNE1 Potassium Channels

The Binding of κ-Conotoxin PVIIA and Fast C-Type Inactivation of Shaker K + Channels are Mutually Exclusive

Skeletal Muscle Sodium Channel Is Affected by an Epileptogenic β1 Subunit Mutation

Sodium channel heterologous expression in mammalian cells and the role of the endogenous beta1-subunits

The Binding of [kappa]-Conotoxin PVIIA and Fast C-Type Inactivation of Shaker K^sup +^ Channels are Mutually Exclusive

Binding of kappa-conotoxin PVIIA to Shaker K+ channels reveals different K+ and Rb+ occupancies within the ion channel pore

Disposition of metformin (N,N‐dimethylbiguanide) in man

The block of Shaker K+ channels by kappa-conotoxin PVIIA is state dependent

Mechanism of block of single protopores of the Torpedo chloride channel ClC-0 by 2-(p-chlorophenoxy)butyric acid (CPB)

Mechanisms of block of muscle type CLC chloride channels (Review)

Tonic and Phasic Tetrodotoxin Block of Sodium Channels with Point Mutations in the Outer Pore Region

Functional Properties of Sodium Channels Do Not Depend on the Cytoskeleton Integrity

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