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Nucleotide Excision Repair and Transcriptional Regulation: TFIIH and Beyond

Annual review of biochemistry, 2016-06, Vol.85 (1), p.265-290

2016

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TFIIE orchestrates the recruitment of the TFIIH kinase module at promoter before release during transcription

Nature communications, 2019-05, Vol.10 (1), p.2084-2084, Article 2084

2019

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Lurbinectedin Specifically Triggers the Degradation of Phosphorylated RNA Polymerase II and the Formation of DNA Breaks in Cancer Cells

Molecular cancer therapeutics, 2016-10, Vol.15 (10), p.2399-2412

2016

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HR-Bac, a toolbox based on homologous recombination for expression, screening and production of multiprotein complexes using the baculovirus expression system

Scientific reports, 2022-02, Vol.12 (1), p.2030-2030, Article 2030

2022

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Author Correction: HR-Bac, a toolbox based on homologous recombination for expression, screening and production of multiprotein complexes using the baculovirus expression system

Scientific reports, 2022-02, Vol.12 (1), p.3443-3443, Article 3443

2022

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The phosphorylation of the androgen receptor by TFIIH directs the ubiquitin/proteasome process

The EMBO journal, 2011-02, Vol.30 (3), p.468-479

2011

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Active mRNA degradation by EXD2 nuclease elicits recovery of transcription after genotoxic stress

Nature communications, 2023-01, Vol.14 (1), p.341-341, Article 341

2023

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Dysregulation of LXR responsive genes contribute to ichthyosis in trichothiodystrophy

Journal of dermatological science, 2020-03, Vol.97 (3), p.201-207

2020

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Super-enhancer-driven expression of BAHCC1 promotes melanoma cell proliferation and genome stability

Cell reports (Cambridge), 2023-11, Vol.42 (11), p.113363-113363, Article 113363

2023

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A PKD-MFF signaling axis couples mitochondrial fission to mitotic progression

Cell reports (Cambridge), 2021-05, Vol.35 (7), p.109129-109129, Article 109129

2021

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Promoters of ASCL1‐ and NEUROD1‐dependent genes are specific targets of lurbinectedin in SCLC cells

EMBO molecular medicine, 2022-04, Vol.14 (4), p.e14841-n/a

2022

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XPG Stabilizes TFIIH, Allowing Transactivation of Nuclear Receptors: Implications for Cockayne Syndrome in XP-G/CS Patients

Molecular cell, 2007-04, Vol.26 (2), p.231-243

2007

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TFIIH-dependent MMP-1 overexpression in trichothiodystrophy leads to extracellular matrix alterations in patient skin

Proceedings of the National Academy of Sciences - PNAS, 2015-02, Vol.112 (5), p.1499-1504

2015

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Dynamic partnership between TFIIH, PGC-1α and SIRT1 is impaired in trichothiodystrophy

PLoS genetics, 2014-10, Vol.10 (10), p.e1004732-e1004732

2014

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Neurological defects in trichothiodystrophy reveal a coactivator function of TFIIH

Nature neuroscience, 2007-11, Vol.10 (11), p.1414-1422

2007

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Dysregulation of the Peroxisome Proliferator-Activated Receptor Target Genes by XPD Mutations

Molecular and Cellular Biology, 2005-07, Vol.25 (14), p.6065-6076

2005

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TFIIH: when transcription met DNA repair

Nature reviews. Molecular cell biology, 2012-06, Vol.13 (6), p.343

2012

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Abnormal XPD-induced nuclear receptor transactivation in DNA repair disorders: trichothiodystrophy and xeroderma pigmentosum

European journal of human genetics : EJHG, 2013-08, Vol.21 (8), p.831-837

2013

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Selective Regulation of Vitamin D Receptor-Responsive Genes by TFIIH

Molecular cell, 2004-10, Vol.16 (2), p.187-197

2004

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The Long Road to Understanding RNAPII Transcription Initiation and Related Syndromes

Annual review of biochemistry, 2021-06, Vol.90, p.193

2021

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A combined approach identifies a limited number of new thyroid hormone target genes in post-natal mouse cerebellum

Journal of molecular endocrinology, 2007-07, Vol.39 (1), p.17-28

2007

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Reduced levels of prostaglandin I 2 synthase: a distinctive feature of the cancer-free trichothiodystrophy

Proceedings of the National Academy of Sciences - PNAS, 2021-06, Vol.118 (26)

2021

Volltextzugriff (PDF)

Reduced levels of prostaglandin I2 synthase: a distinctive feature of the cancer-free trichothiodystrophy

Proceedings of the National Academy of Sciences - PNAS, 2021-06, Vol.118 (26)

2021

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CDK7 and MITF repress a transcription program involved in survival and drug tolerance in melanoma

EMBO reports, 2021-09, Vol.22 (9), p.e51683-n/a

2021

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Both XPD alleles contribute to the phenotype of compound heterozygote xeroderma pigmentosum patients

The Journal of experimental medicine, 2009-12, Vol.206 (13), p.3031-3046

2009

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Nucleotide Excision Repair and Transcriptional Regulation: TFIIH and Beyond

TFIIE orchestrates the recruitment of the TFIIH kinase module at promoter before release during transcription

Lurbinectedin Specifically Triggers the Degradation of Phosphorylated RNA Polymerase II and the Formation of DNA Breaks in Cancer Cells

HR-Bac, a toolbox based on homologous recombination for expression, screening and production of multiprotein complexes using the baculovirus expression system

Author Correction: HR-Bac, a toolbox based on homologous recombination for expression, screening and production of multiprotein complexes using the baculovirus expression system

The phosphorylation of the androgen receptor by TFIIH directs the ubiquitin/proteasome process

Active mRNA degradation by EXD2 nuclease elicits recovery of transcription after genotoxic stress

Dysregulation of LXR responsive genes contribute to ichthyosis in trichothiodystrophy

Super-enhancer-driven expression of BAHCC1 promotes melanoma cell proliferation and genome stability

A PKD-MFF signaling axis couples mitochondrial fission to mitotic progression

Promoters of ASCL1‐ and NEUROD1‐dependent genes are specific targets of lurbinectedin in SCLC cells

XPG Stabilizes TFIIH, Allowing Transactivation of Nuclear Receptors: Implications for Cockayne Syndrome in XP-G/CS Patients

TFIIH-dependent MMP-1 overexpression in trichothiodystrophy leads to extracellular matrix alterations in patient skin

Dynamic partnership between TFIIH, PGC-1α and SIRT1 is impaired in trichothiodystrophy

Neurological defects in trichothiodystrophy reveal a coactivator function of TFIIH

Dysregulation of the Peroxisome Proliferator-Activated Receptor Target Genes by XPD Mutations

TFIIH: when transcription met DNA repair

Abnormal XPD-induced nuclear receptor transactivation in DNA repair disorders: trichothiodystrophy and xeroderma pigmentosum

Selective Regulation of Vitamin D Receptor-Responsive Genes by TFIIH

The Long Road to Understanding RNAPII Transcription Initiation and Related Syndromes

A combined approach identifies a limited number of new thyroid hormone target genes in post-natal mouse cerebellum

Reduced levels of prostaglandin I 2 synthase: a distinctive feature of the cancer-free trichothiodystrophy

Reduced levels of prostaglandin I2 synthase: a distinctive feature of the cancer-free trichothiodystrophy

CDK7 and MITF repress a transcription program involved in survival and drug tolerance in melanoma

Both XPD alleles contribute to the phenotype of compound heterozygote xeroderma pigmentosum patients

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