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Clustering and Functional Coupling of Diverse Ion Channels and Signaling Proteins Revealed by Super-resolution STORM Microscopy in Neurons

Neuron (Cambridge, Mass.), 2016-10, Vol.92 (2), p.461-478

2016

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Phosphatidylinositol 4,5-bisphosphate (PIP2) regulates KCNQ3 K+ channels by interacting with four cytoplasmic channel domains

The Journal of biological chemistry, 2018-12, Vol.293 (50), p.19411-19428

2018

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Antagonistic Effect of a Cytoplasmic Domain on the Basal Activity of Polymodal Potassium Channels

Frontiers in molecular neuroscience, 2018-09, Vol.11, p.301-301

2018

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The Role of the Carboxyl Terminus Helix C-D Linker in Regulating KCNQ3 K+ Current Amplitudes by Controlling Channel Trafficking

PloS one, 2015-12, Vol.10 (12), p.e0145367-e0145367

2015

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Convergence of Multiple Stimuli to a Single Gate in TREK1 and TRAAK Potassium Channels

Frontiers in pharmacology, 2021-09, Vol.12, p.755826-755826

2021

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Augmentation of M-type (KCNQ) potassium channels as a novel strategy to reduce stroke-induced brain injury

The Journal of neuroscience, 2015-02, Vol.35 (5), p.2101-2111

2015

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Corrigendum: Convergence of Multiple Stimuli to a Single Gate in TREK1 and TRAAK Potassium Channels

Frontiers in pharmacology, 2021-11, Vol.12, p.795920-795920

2021

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Astemizole Sensitizes Adrenocortical Carcinoma Cells to Doxorubicin by Inhibiting Patched Drug Efflux Activity

Biomedicines, 2020-07, Vol.8 (8), p.251

2020

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Pore Helix-S6 Interactions Are Critical in Governing Current Amplitudes of KCNQ3 K+ Channels

Biophysical journal, 2012-06, Vol.102 (11), p.2499-2509

2012

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Pore Determinants of KCNQ3 K+ Current Expression

Biophysical journal, 2012-06, Vol.102 (11), p.2489-2498

2012

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Kv7.1 (KCNQ1) properties and channelopathies

The Journal of physiology, 2008-04, Vol.586 (7), p.1785-1789

2008

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The S4-S5 Linker of KCNQ1 Channels Forms a Structural Scaffold with the S6 Segment Controlling Gate Closure

The Journal of biological chemistry, 2011-01, Vol.286 (1), p.717-725

2011

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KCNQ1 Channels Voltage Dependence through a Voltage-dependent Binding of the S4-S5 Linker to the Pore Domain

The Journal of biological chemistry, 2011-01, Vol.286 (1), p.707-716

2011

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KCNE1–KCNQ1 osmoregulation by interaction of phosphatidylinositol‐4,5‐bisphosphate with Mg2+ and polyamines

The Journal of physiology, 2010-09, Vol.588 (18), p.3471-3483

2010

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Regions of KCNQ K(+) channels controlling functional expression

Frontiers in physiology, 2012-01, Vol.3, p.397-397

2012

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Transfer of Rolf S3-S4 Linker to hERG Eliminates Activation Gating but Spares Inactivation

Biophysical journal, 2009-09, Vol.97 (5), p.1323-1334

2009

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Abstract P2082: Thrombosis In The Coronary Microvasculature May Be Responsible For Impaired Cardiac Relaxation And Diastolic Dysfunction

Circulation research, 2023-08, Vol.133 (Suppl_1)

2023

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Opposite Effects of the S4–S5 Linker and PIP2 on Voltage-Gated Channel Function: KCNQ1/KCNE1 and Other Channels

Frontiers in pharmacology, 2012, Vol.3

2012

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Thrombosis in the Coronary Microvasculature Impairs Cardiac Relaxation and Induces Diastolic Dysfunction

Arteriosclerosis, thrombosis, and vascular biology, 2024-01, Vol.44 (1), p.e1

2024

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Convergence of Multiple Stimuli to a Single Gate in TREK1 and TRAAK Potassium Channels

Frontiers in pharmacology, 2021-09, Vol.12

2021

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Convergence of Multiple Stimuli to a Single Gate in TREK1 and TRAAK Potassium Channels

Frontiers in pharmacology, 2021-09, Vol.12

2021

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Use of a Voltage-Sensitive Phosphatase Highlights Two PiP2-Binding Sites in the C-Terminus of KCNQ3 Channels

Biophysical journal, 2014-01, Vol.106 (2), p.750a-750a

2014

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Use of Voltage-Sensitive Phosphatase to Investigate the Location of the PIP2-Binding Site on KCNQ2 Channels

Biophysical journal, 2013-01, Vol.104 (2), p.270a-270a

2013

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The Role of the Carboxyl Terminus Helix C-D Linker in Regulating KCNQ3 K.sup.+ Current Amplitudes by Controlling Channel Trafficking

PloS one, 2015-12, Vol.10 (12)

2015

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Thrombosis in the coronary microvasculature may be responsible for impaired cardiac relaxation and diastolic dysfunction

Archives of cardiovascular diseases supplements, 2023-05, Vol.15 (2), p.207-208

2023

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Clustering and Functional Coupling of Diverse Ion Channels and Signaling Proteins Revealed by Super-resolution STORM Microscopy in Neurons

Phosphatidylinositol 4,5-bisphosphate (PIP2) regulates KCNQ3 K+ channels by interacting with four cytoplasmic channel domains

Antagonistic Effect of a Cytoplasmic Domain on the Basal Activity of Polymodal Potassium Channels

The Role of the Carboxyl Terminus Helix C-D Linker in Regulating KCNQ3 K+ Current Amplitudes by Controlling Channel Trafficking

Convergence of Multiple Stimuli to a Single Gate in TREK1 and TRAAK Potassium Channels

Augmentation of M-type (KCNQ) potassium channels as a novel strategy to reduce stroke-induced brain injury

Corrigendum: Convergence of Multiple Stimuli to a Single Gate in TREK1 and TRAAK Potassium Channels

Astemizole Sensitizes Adrenocortical Carcinoma Cells to Doxorubicin by Inhibiting Patched Drug Efflux Activity

Pore Helix-S6 Interactions Are Critical in Governing Current Amplitudes of KCNQ3 K+ Channels

Pore Determinants of KCNQ3 K+ Current Expression

Kv7.1 (KCNQ1) properties and channelopathies

The S4-S5 Linker of KCNQ1 Channels Forms a Structural Scaffold with the S6 Segment Controlling Gate Closure

KCNQ1 Channels Voltage Dependence through a Voltage-dependent Binding of the S4-S5 Linker to the Pore Domain

KCNE1–KCNQ1 osmoregulation by interaction of phosphatidylinositol‐4,5‐bisphosphate with Mg2+ and polyamines

Regions of KCNQ K(+) channels controlling functional expression

Transfer of Rolf S3-S4 Linker to hERG Eliminates Activation Gating but Spares Inactivation

Abstract P2082: Thrombosis In The Coronary Microvasculature May Be Responsible For Impaired Cardiac Relaxation And Diastolic Dysfunction

Opposite Effects of the S4–S5 Linker and PIP2 on Voltage-Gated Channel Function: KCNQ1/KCNE1 and Other Channels

Thrombosis in the Coronary Microvasculature Impairs Cardiac Relaxation and Induces Diastolic Dysfunction

Convergence of Multiple Stimuli to a Single Gate in TREK1 and TRAAK Potassium Channels

Convergence of Multiple Stimuli to a Single Gate in TREK1 and TRAAK Potassium Channels

Use of a Voltage-Sensitive Phosphatase Highlights Two PiP2-Binding Sites in the C-Terminus of KCNQ3 Channels

Use of Voltage-Sensitive Phosphatase to Investigate the Location of the PIP2-Binding Site on KCNQ2 Channels

The Role of the Carboxyl Terminus Helix C-D Linker in Regulating KCNQ3 K.sup.+ Current Amplitudes by Controlling Channel Trafficking

Thrombosis in the coronary microvasculature may be responsible for impaired cardiac relaxation and diastolic dysfunction

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