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The tsunami named CRISPR/Cas9

Revue neurologique, 2018-09, Vol.174 (7-8), p.487-488

Chneiweiss, H.

2018

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The miR 302-367 cluster drastically affects self-renewal and infiltration properties of glioma-initiating cells through CXCR4 repression and consequent disruption of the SHH-GLI-NANOG network

Cell death and differentiation, 2012-02, Vol.19 (2), p.232-244

2012

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A cell-penetrating peptide based on the interaction between c-Src and connexin43 reverses glioma stem cell phenotype

Cell death & disease, 2014-01, Vol.5 (1), p.e1023-e1023

2014

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Emerging ethical issues for practicing neurologists

Journal of the neurological sciences, 2019-10, Vol.405, p.46-46

Chneiweiss, H.

2019

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DOCK4 promotes loss of proliferation in glioblastoma progenitor cells through nuclear beta-catenin accumulation and subsequent miR-302-367 cluster expression

Oncogene, 2018-01, Vol.37 (2), p.241-254

2018

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Retinoblastoma protein regulates the crosstalk between autophagy and apoptosis, and favors glioblastoma resistance to etoposide

Cell death & disease, 2013-08, Vol.4 (8), p.e767-e767

2013

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Expanded polyglutamine peptides disrupt EGF receptor signaling and glutamate transporter expression in Drosophila

Human molecular genetics, 2005-03, Vol.14 (5), p.713-724

2005

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Glut3 addiction is a druggable vulnerability for a molecularly defined subpopulation of glioblastoma

Annals of oncology, 2017-10, Vol.28, p.vii24-vii24

2017

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The PEA-15 PED protein protects glioblastoma cells from glucose deprivation-induced apoptosis via the ERK MAP kinase pathway

Oncogene, 2008-02, Vol.27 (8), p.1155-1166

2008

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Transforming growth factor α promotes sequential conversion of mature astrocytes into neural progenitors and stem cells

Oncogene, 2007-04, Vol.26 (19), p.2695-2706

2007

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Transforming growth factor promotes sequential conversion of mature astrocytes into neural progenitors and stem cells

Oncogene, 2007-04, Vol.26 (19), p.2695-2706

2007

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Molecular and Clinical Correlations in Spinocerebellar Ataxia 2: A Study of 32 Families

Human molecular genetics, 1997-05, Vol.6 (5), p.709-715

1997

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The expression of PEA-15 (phosphoprotein enriched in astrocytes of 15 kDa) defines subpopulations of astrocytes and neurons throughout the adult mouse brain

Neuroscience, 2004, Vol.126 (2), p.263-275

2004

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Characterization of PEA-15, a major substrate for protein kinase C in astrocytes

The Journal of biological chemistry, 1993-03, Vol.268 (8), p.5911-5920

1993

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Transforming growth factor alpha acts as a gliatrophin for mouse and human astrocytes

Oncogene, 2006-07, Vol.25 (29), p.4076-4085

2006

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Chromosomal assignment of the second locus for autosomal dominant cerebellar ataxia (SCA2) to chromosome 12q23-24.1

Nature genetics, 1993-07, Vol.4 (3), p.295-299

1993

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Death effector domain protein PEA-15 potentiates Ras activation of extracellular signal receptor-activated kinase by an adhesion-independent mechanism

Molecular biology of the cell, 2000-09, Vol.11 (9), p.2863-2872

2000

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Intracellular substrates for extracellular signaling. Characterization of a ubiquitous, neuron-enriched phosphoprotein (stathmin)

The Journal of biological chemistry, 1989-03, Vol.264 (7), p.3765-3772

1989

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PEA-15 Mediates Cytoplasmic Sequestration of ERK MAP Kinase

Developmental cell, 2001-08, Vol.1 (2), p.239-250

2001

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Évaluation du traitement multimodal–témozolomide, inhibiteurs de PARP et hadronthérapie–des cellules souches cancéreuses de glioblastome

Cancer radiothérapie, 2017-10, Vol.21 (6-7), p.713-713

2017

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Spinocerebellar ataxia 3 and machado-joseph disease: Clinical, molecular, and neuropathological features

Annals of neurology, 1996-04, Vol.39 (4), p.490-499

1996

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The multifunctional protein PEA-15 is involved in the control of apoptosis and cell cycle in astrocytes

Biochemical pharmacology, 2003-10, Vol.66 (8), p.1581-1588

2003

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IM-04 DELTA-24-RDG AS AN ALTERNATIVE THERAPEUTIC TOOL FOR pHGG/DIPG

Neuro-oncology (Charlottesville, Va.), 2015-06, Vol.17 (suppl 3), p.iii15-iii15

2015

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Proteomic analysis of oligodendrogliomas expressing a mutant isocitrate dehydrogenase-1

Proteomics (Weinheim), 2011-11, Vol.11 (21), p.4139-4154

2011

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Keeping TNF-induced apoptosis under control in astrocytes: PEA-15 as a ‘double key’ on caspase-dependent and MAP-kinase-dependent pathways

Progress in Brain Research, 2001, Vol.132, p.455-467

2001

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The tsunami named CRISPR/Cas9

The miR 302-367 cluster drastically affects self-renewal and infiltration properties of glioma-initiating cells through CXCR4 repression and consequent disruption of the SHH-GLI-NANOG network

A cell-penetrating peptide based on the interaction between c-Src and connexin43 reverses glioma stem cell phenotype

Emerging ethical issues for practicing neurologists

DOCK4 promotes loss of proliferation in glioblastoma progenitor cells through nuclear beta-catenin accumulation and subsequent miR-302-367 cluster expression

Retinoblastoma protein regulates the crosstalk between autophagy and apoptosis, and favors glioblastoma resistance to etoposide

Expanded polyglutamine peptides disrupt EGF receptor signaling and glutamate transporter expression in Drosophila

Glut3 addiction is a druggable vulnerability for a molecularly defined subpopulation of glioblastoma

The PEA-15 PED protein protects glioblastoma cells from glucose deprivation-induced apoptosis via the ERK MAP kinase pathway

Transforming growth factor α promotes sequential conversion of mature astrocytes into neural progenitors and stem cells

Transforming growth factor promotes sequential conversion of mature astrocytes into neural progenitors and stem cells

Molecular and Clinical Correlations in Spinocerebellar Ataxia 2: A Study of 32 Families

The expression of PEA-15 (phosphoprotein enriched in astrocytes of 15 kDa) defines subpopulations of astrocytes and neurons throughout the adult mouse brain

Characterization of PEA-15, a major substrate for protein kinase C in astrocytes

Transforming growth factor alpha acts as a gliatrophin for mouse and human astrocytes

Chromosomal assignment of the second locus for autosomal dominant cerebellar ataxia (SCA2) to chromosome 12q23-24.1

Death effector domain protein PEA-15 potentiates Ras activation of extracellular signal receptor-activated kinase by an adhesion-independent mechanism

Intracellular substrates for extracellular signaling. Characterization of a ubiquitous, neuron-enriched phosphoprotein (stathmin)

PEA-15 Mediates Cytoplasmic Sequestration of ERK MAP Kinase

Évaluation du traitement multimodal–témozolomide, inhibiteurs de PARP et hadronthérapie–des cellules souches cancéreuses de glioblastome

Spinocerebellar ataxia 3 and machado-joseph disease: Clinical, molecular, and neuropathological features

The multifunctional protein PEA-15 is involved in the control of apoptosis and cell cycle in astrocytes

IM-04 DELTA-24-RDG AS AN ALTERNATIVE THERAPEUTIC TOOL FOR pHGG/DIPG

Proteomic analysis of oligodendrogliomas expressing a mutant isocitrate dehydrogenase-1

Keeping TNF-induced apoptosis under control in astrocytes: PEA-15 as a ‘double key’ on caspase-dependent and MAP-kinase-dependent pathways

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