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Selective PPAR modulators (SPPARs) may fill the need for treatment of the atherogenic dyslipidemia of insulin resistance and type 2 diabetes: Can they reduce the associated cardiac risk?

Atherosclerosis, 2016-06, Vol.249, p.224-225

Camejo, Germán

2016

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Phase 2 clinical trials with K-877 (pemafibrate): A promising selective PPAR-α modulator for treatment of combined dyslipidemia

Atherosclerosis, 2017-06, Vol.261, p.163-164

Camejo, Germán

2017

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Proteomics of apolipoproteins and associated proteins from plasma high-density lipoproteins

Arteriosclerosis, thrombosis, and vascular biology, 2010-02, Vol.30 (2), p.156

2010

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ApoB-100 Lipoprotein Complex Formation with Intima Proteoglycans as a Cause of Atherosclerosis and Its Possible Ex Vivo Evaluation as a Disease Biomarker

Journal of cardiovascular development and disease, 2018-07, Vol.5 (3), p.36

2018

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Lysophospholipids: Effectors mediating the contribution of dyslipidemia to calcification associated with atherosclerosis

Atherosclerosis, 2010-07, Vol.211 (1), p.36-37

Camejo, Germán

2010

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Macrophages, extracellular matrix, and lipoproteins in arterial cholesterol balance

Journal of lipid research, 2014-01, Vol.55 (1), p.1-3

2014

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Proteomics and lipids of lipoproteins isolated at low salt concentrations in D2O/sucrose or in KBr

Journal of lipid research, 2008-02, Vol.49 (2), p.481-490

2008

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HDL2 interferes with LDL association with arterial proteoglycans: A possible athero-protective effect

Atherosclerosis, 2012-11, Vol.225 (1), p.115-120

2012

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Macrophages exposed to hypoxia secrete proteoglycans for which LDL has higher affinity

Atherosclerosis, 2011-03, Vol.215 (1), p.77-81

2011

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proteomic study of the apolipoproteins in LDL subclasses in patients with the metabolic syndrome and type 2 diabetes

Journal of lipid research, 2005-09, Vol.46 (9), p.1999-2006

2005

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Vasculoprotective properties of plasma lipoproteins from brown bears (Ursus arctos)

Journal of lipid research, 2021-01, Vol.62, p.100065, Article 100065

2021

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Hypoxia increases macrophage motility, possibly by decreasing the heparan sulfate proteoglycan biosynthesis

Journal of leukocyte biology, 2009-08, Vol.86 (2), p.381-388

2009

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Hypoxic regulation of secreted proteoglycans in macrophages

Glycobiology (Oxford), 2010, Vol.20 (1), p.33-40

2010

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Postprandial Hypertriglyceridemia Is Associated with the Variant 54 Threonine FABP2 Gene

Journal of cardiovascular development and disease, 2018-09, Vol.5 (3), p.47

2018

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Cellular consequences of the association of apoB lipoproteins with proteoglycans. Potential contribution to atherogenesis

Arteriosclerosis, thrombosis, and vascular biology, 1997-06, Vol.17 (6), p.1011

1997

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AZ 242, a novel PPARα/γ agonist with beneficial effects on insulin resistance and carbohydrate and lipid metabolism in ob/ob mice and obese Zucker rats

Journal of lipid research, 2002-11, Vol.43 (11), p.1855-1863

2002

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Hydrolytic enzymes released from resident macrophages and located in the intima extracellular matrix as agents that modify retained apolipoprotein B lipoproteins

Arteriosclerosis, thrombosis, and vascular biology, 2003-08, Vol.23 (8), p.1312

Camejo, German

2003

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Fatty acids cause alterations of human arterial smooth muscle cell proteoglycans that increase the affinity for low-density lipoprotein

Arteriosclerosis, thrombosis, and vascular biology, 2006-01, Vol.26 (1), p.130

2006

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Phospholipase A2 in Vascular Disease

Circulation research, 2001-08, Vol.89 (4), p.298-304

2001

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Macrophages, extracellular matrix, and lipoproteins in arterial cholesterol balance1

Journal of lipid research, 2014-01, Vol.55 (1), p.1-3

2014

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Changes in Matrix Proteoglycans Induced by Insulin and Fatty Acids in Hepatic Cells May Contribute to Dyslipidemia of Insulin Resistance

Diabetes (New York, N.Y.), 2001-09, Vol.50 (9), p.2126-2132

2001

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Fatty acids modulate the composition of extracellular matrix in cultured human arterial smooth muscle cells by altering the expression of genes for proteoglycan core proteins

Diabetes (New York, N.Y.), 1999-03, Vol.48 (3), p.616

1999

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Proteoglycans contribution to association of Lp(a) and LDL with smooth muscle cell extracellular matrix

Arteriosclerosis, thrombosis, and vascular biology, 1999-05, Vol.19 (5), p.1162

1999

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Ezetimibe in Combination With Simvastatin Reduces Remnant Cholesterol Without Affecting Biliary Lipid Concentrations in Gallstone Patients

Journal of the American Heart Association, 2018-12, Vol.7 (24), p.e009876

2018

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Possible functional interactions of apolipoprotein B-100 segments that associate with cell proteoglycans and the ApoB/E receptor

Arteriosclerosis, thrombosis, and vascular biology, 1997-01, Vol.17 (1), p.149

1997

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Selective PPAR modulators (SPPARs) may fill the need for treatment of the atherogenic dyslipidemia of insulin resistance and type 2 diabetes: Can they reduce the associated cardiac risk?

Phase 2 clinical trials with K-877 (pemafibrate): A promising selective PPAR-α modulator for treatment of combined dyslipidemia

Proteomics of apolipoproteins and associated proteins from plasma high-density lipoproteins

ApoB-100 Lipoprotein Complex Formation with Intima Proteoglycans as a Cause of Atherosclerosis and Its Possible Ex Vivo Evaluation as a Disease Biomarker

Lysophospholipids: Effectors mediating the contribution of dyslipidemia to calcification associated with atherosclerosis

Macrophages, extracellular matrix, and lipoproteins in arterial cholesterol balance

Proteomics and lipids of lipoproteins isolated at low salt concentrations in D2O/sucrose or in KBr

HDL2 interferes with LDL association with arterial proteoglycans: A possible athero-protective effect

Macrophages exposed to hypoxia secrete proteoglycans for which LDL has higher affinity

proteomic study of the apolipoproteins in LDL subclasses in patients with the metabolic syndrome and type 2 diabetes

Vasculoprotective properties of plasma lipoproteins from brown bears (Ursus arctos)

Hypoxia increases macrophage motility, possibly by decreasing the heparan sulfate proteoglycan biosynthesis

Hypoxic regulation of secreted proteoglycans in macrophages

Postprandial Hypertriglyceridemia Is Associated with the Variant 54 Threonine FABP2 Gene

Cellular consequences of the association of apoB lipoproteins with proteoglycans. Potential contribution to atherogenesis

AZ 242, a novel PPARα/γ agonist with beneficial effects on insulin resistance and carbohydrate and lipid metabolism in ob/ob mice and obese Zucker rats

Hydrolytic enzymes released from resident macrophages and located in the intima extracellular matrix as agents that modify retained apolipoprotein B lipoproteins

Fatty acids cause alterations of human arterial smooth muscle cell proteoglycans that increase the affinity for low-density lipoprotein

Phospholipase A2 in Vascular Disease

Macrophages, extracellular matrix, and lipoproteins in arterial cholesterol balance1

Changes in Matrix Proteoglycans Induced by Insulin and Fatty Acids in Hepatic Cells May Contribute to Dyslipidemia of Insulin Resistance

Fatty acids modulate the composition of extracellular matrix in cultured human arterial smooth muscle cells by altering the expression of genes for proteoglycan core proteins

Proteoglycans contribution to association of Lp(a) and LDL with smooth muscle cell extracellular matrix

Ezetimibe in Combination With Simvastatin Reduces Remnant Cholesterol Without Affecting Biliary Lipid Concentrations in Gallstone Patients

Possible functional interactions of apolipoprotein B-100 segments that associate with cell proteoglycans and the ApoB/E receptor

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