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Widespread expression of the ancient HERV-K (HML-2) provirus group in normal human tissues

PLoS biology, 2022-10, Vol.20 (10), p.e3001826

2022

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discovery of HTLV-1, the first pathogenic human retrovirus

Proceedings of the National Academy of Sciences - PNAS, 2015-12, Vol.112 (51), p.15525-15529

Coffin, John M

2015

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Effects of retroviruses on host genome function

Annual review of genetics, 2008-01, Vol.42 (1), p.709-732

2008

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Lack of detectable HIV-1 molecular evolution during suppressive antiretroviral therapy

PLoS pathogens, 2014-03, Vol.10 (3), p.e1004010

2014

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Proviruses with identical sequences comprise a large fraction of the replication-competent HIV reservoir

PLoS pathogens, 2017-03, Vol.13 (3), p.e1006283-e1006283

2017

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What Integration Sites Tell Us about HIV Persistence

Cell host & microbe, 2016-05, Vol.19 (5), p.588-598

2016

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Discovery of unfixed endogenous retrovirus insertions in diverse human populations

Proceedings of the National Academy of Sciences - PNAS, 2016-04, Vol.113 (16), p.E2326-E2334

2016

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Identification, characterization, and comparative genomic distribution of the HERV-K (HML-2) group of human endogenous retroviruses

Retrovirology, 2011-11, Vol.8 (1), p.90-90, Article 90

2011

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Clinical Trial of the Anti-PD-L1 Antibody BMS-936559 in HIV-1 Infected Participants on Suppressive Antiretroviral Therapy

The Journal of infectious diseases, 2017-06, Vol.215 (11), p.1725-1733

2017

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Quantification of HIV-1 latency reversal in resting CD4⁺ T cells from patients on suppressive antiretroviral therapy

Proceedings of the National Academy of Sciences - PNAS, 2014-05, Vol.111 (19), p.7078-7083

2014

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Single-cell analysis of HIV-1 transcriptional activity reveals expression of proviruses in expanded clones during ART

Proceedings of the National Academy of Sciences - PNAS, 2017-05, Vol.114 (18), p.E3659-E3668

2017

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Nomenclature for endogenous retrovirus (ERV) loci

Retrovirology, 2018-08, Vol.15 (1), p.59-59, Article 59

2018

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Virions at the gates: receptors and the host-virus arms race

PLoS biology, 2013-05, Vol.11 (5), p.e1001574-e1001574

Coffin, John M

2013

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HIV-1 in lymph nodes is maintained by cellular proliferation during antiretroviral therapy

The Journal of clinical investigation, 2019-11, Vol.129 (11), p.4629-4642

2019

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Flexible Use of Nuclear Import Pathways by HIV-1

Cell host & microbe, 2010-03, Vol.7 (3), p.221-233

2010

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Antiretroviral intensification and valproic acid lack sustained effect on residual HIV-1 viremia or resting CD4+ cell infection

PloS one, 2010-02, Vol.5 (2), p.e9390-e9390

2010

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The effect of raltegravir intensification on low-level residual viremia in HIV-infected patients on antiretroviral therapy: a randomized controlled trial

PLoS medicine, 2010-08, Vol.7 (8), p.e1000321

2010

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Clonal Expansion of Infected CD4+ T Cells in People Living with HIV

Viruses, 2021-10, Vol.13 (10), p.2078

2021

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Integration in oncogenes plays only a minor role in determining the in vivo distribution of HIV integration sites before or during suppressive antiretroviral therapy

PLoS pathogens, 2021-04, Vol.17 (4), p.e1009141-e1009141

2021

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Low-Level Viremia Persists for at Least 7 Years in Patients on Suppressive Antiretroviral Therapy

Proceedings of the National Academy of Sciences - PNAS, 2008-03, Vol.105 (10), p.3879-3884

2008

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Immediate antiviral therapy appears to restrict resting CD4⁺ cell HIV-1 infection without accelerating the decay of latent infection

Proceedings of the National Academy of Sciences - PNAS, 2012-06, Vol.109 (24), p.9523-9528

2012

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Single cell analysis of lymph node tissue from HIV-1 infected patients reveals that the majority of CD4+ T-cells contain one HIV-1 DNA molecule

PLoS pathogens, 2013, Vol.9 (6), p.e1003432-e1003432

2013

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Likely role of APOBEC3G-mediated G-to-A mutations in HIV-1 evolution and drug resistance

PLoS pathogens, 2009-04, Vol.5 (4), p.e1000367-e1000367

2009

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Majority of CD4+T cells from peripheral blood of HIV-1—infected individuals contain only one HIV DNA molecule

Proceedings of the National Academy of Sciences - PNAS, 2011-07, Vol.108 (27), p.11199-11204

2011

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An analytical pipeline for identifying and mapping the integration sites of HIV and other retroviruses

BMC genomics, 2020-03, Vol.21 (1), p.216-216, Article 216

2020

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Widespread expression of the ancient HERV-K (HML-2) provirus group in normal human tissues

discovery of HTLV-1, the first pathogenic human retrovirus

Effects of retroviruses on host genome function

Lack of detectable HIV-1 molecular evolution during suppressive antiretroviral therapy

Proviruses with identical sequences comprise a large fraction of the replication-competent HIV reservoir

What Integration Sites Tell Us about HIV Persistence

Discovery of unfixed endogenous retrovirus insertions in diverse human populations

Identification, characterization, and comparative genomic distribution of the HERV-K (HML-2) group of human endogenous retroviruses

Clinical Trial of the Anti-PD-L1 Antibody BMS-936559 in HIV-1 Infected Participants on Suppressive Antiretroviral Therapy

Quantification of HIV-1 latency reversal in resting CD4⁺ T cells from patients on suppressive antiretroviral therapy

Single-cell analysis of HIV-1 transcriptional activity reveals expression of proviruses in expanded clones during ART

Nomenclature for endogenous retrovirus (ERV) loci

Virions at the gates: receptors and the host-virus arms race

HIV-1 in lymph nodes is maintained by cellular proliferation during antiretroviral therapy

Flexible Use of Nuclear Import Pathways by HIV-1

Antiretroviral intensification and valproic acid lack sustained effect on residual HIV-1 viremia or resting CD4+ cell infection

The effect of raltegravir intensification on low-level residual viremia in HIV-infected patients on antiretroviral therapy: a randomized controlled trial

Clonal Expansion of Infected CD4+ T Cells in People Living with HIV

Integration in oncogenes plays only a minor role in determining the in vivo distribution of HIV integration sites before or during suppressive antiretroviral therapy

Low-Level Viremia Persists for at Least 7 Years in Patients on Suppressive Antiretroviral Therapy

Immediate antiviral therapy appears to restrict resting CD4⁺ cell HIV-1 infection without accelerating the decay of latent infection

Single cell analysis of lymph node tissue from HIV-1 infected patients reveals that the majority of CD4+ T-cells contain one HIV-1 DNA molecule

Likely role of APOBEC3G-mediated G-to-A mutations in HIV-1 evolution and drug resistance

Majority of CD4+T cells from peripheral blood of HIV-1—infected individuals contain only one HIV DNA molecule

An analytical pipeline for identifying and mapping the integration sites of HIV and other retroviruses

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