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Cytotoxicity of paclitaxel in breast cancer is due to chromosome missegregation on multipolar spindles

Science translational medicine, 2014-03, Vol.6 (229), p.229ra43

2014

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Analysis of the "centrosome-ome" identifies MCPH1 deletion as a cause of centrosome amplification in human cancer

Scientific reports, 2020-07, Vol.10 (1), p.11921, Article 11921

2020

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CHARTS: a web application for characterizing and comparing tumor subpopulations in publicly available single-cell RNA-seq data sets

BMC bioinformatics, 2021-02, Vol.22 (1), p.83-83, Article 83

2021

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Centrosome amplification induces high grade features and is prognostic of worse outcomes in breast cancer

BMC cancer, 2016-01, Vol.16 (47), p.47-47, Article 47

2016

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High Mitotic Activity of Polo-like Kinase 1 Is Required for Chromosome Segregation and Genomic Integrity in Human Epithelial Cells

The Journal of biological chemistry, 2012-12, Vol.287 (51), p.42812-42825

2012

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Anillin Phosphorylation Controls Timely Membrane Association and Successful Cytokinesis

PLoS genetics, 2017-01, Vol.13 (1), p.e1006511-e1006511

2017

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Plk1 self-organization and priming phosphorylation of HsCYK-4 at the spindle midzone regulate the onset of division in human cells

PLoS biology, 2009-05, Vol.7 (5), p.e1000111-e1000111

2009

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Centriole Overduplication is the Predominant Mechanism Leading to Centrosome Amplification in Melanoma

Molecular cancer research, 2018-03, Vol.16 (3), p.517-527

2018

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Metabolic Heterogeneity in Patient Tumor-Derived Organoids by Primary Site and Drug Treatment

Frontiers in oncology, 2020-05, Vol.10, p.553-553

2020

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High nuclear TPX2 expression correlates with TP53 mutation and poor clinical behavior in a large breast cancer cohort, but is not an independent predictor of chromosomal instability

BMC cancer, 2021-02, Vol.21 (1), p.186-186, Article 186

2021

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Diverse microtubule-targeted anticancer agents kill cells by inducing chromosome missegregation on multipolar spindles

PLoS biology, 2023-10, Vol.21 (10), p.e3002339-e3002339

2023

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Polo-like kinase 4 maintains centriolar satellite integrity by phosphorylation of centrosomal protein 131 (CEP131)

The Journal of biological chemistry, 2019-04, Vol.294 (16), p.6531-6549

2019

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Quantifying chromosomal instability from intratumoral karyotype diversity using agent-based modeling and Bayesian inference

eLife, 2022-04, Vol.11

2022

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Accuracy of models to prognosticate survival after surgery for pancreatic cancer in the era of neoadjuvant therapy

Journal of surgical oncology, 2023-08, Vol.128 (2), p.280-288

2023

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Chemical genetics reveals the requirement for Polo-like kinase 1 activity in positioning RhoA and triggering cytokinesis in human cells

Proceedings of the National Academy of Sciences - PNAS, 2007-03, Vol.104 (11), p.4383-4388

2007

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Photosensitive lichenoid skin reaction to capecitabine

BMC cancer, 2017-12, Vol.17 (1), p.866-866, Article 866

2017

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Synchronous bilateral breast cancer in a patient with Nager syndrome

Clinical breast cancer, 2017-06, Vol.17 (3), p.e151-e153

2017

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Centrosome amplification is a frequent event in circulating tumor cells from subjects with metastatic breast cancer

Molecular oncology, 2020-08, Vol.14 (8), p.1898-1909

2020

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The reckoning of chromosomal instability: past, present, future

Chromosome research, 2024-02, Vol.32 (1), p.2-2, Article 2

2024

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Clinical utility of a regional precision medicine molecular tumor board and challenges to implementation

Journal of oncology pharmacy practice, 2023-07, Vol.29 (5), p.1094-1102

2023

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Acquisition of Cabozantinib-Sensitive MET D1228N Mutation During Progression on Crizotinib in MET-Amplified Triple-Negative Breast Cancer

Clinical breast cancer, 2020-08, Vol.20 (4), p.e433-e438

2020

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The Functional Significance of Posttranslational Modifications on Polo-Like Kinase 1 Revealed by Chemical Genetic Complementation

PloS one, 2016-02, Vol.11 (2), p.e0150225

2016

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Classes of therapeutics to amplify the immune response

Breast cancer research and treatment, 2022, Vol.191 (2), p.277-289

2022

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A physician-scientist preceptorship in clinical and translational research enhances training and mentorship

BMC medical education, 2019-03, Vol.19 (1), p.89-89, Article 89

2019

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Cytotoxicity of paclitaxel in breast cancer is due to chromosome missegregation on multipolar spindles

Patient-Derived Cancer Organoid Cultures to Predict Sensitivity to Chemotherapy and Radiation

Analysis of the "centrosome-ome" identifies MCPH1 deletion as a cause of centrosome amplification in human cancer

CHARTS: a web application for characterizing and comparing tumor subpopulations in publicly available single-cell RNA-seq data sets

Centrosome amplification induces high grade features and is prognostic of worse outcomes in breast cancer

High Mitotic Activity of Polo-like Kinase 1 Is Required for Chromosome Segregation and Genomic Integrity in Human Epithelial Cells

Anillin Phosphorylation Controls Timely Membrane Association and Successful Cytokinesis

Plk1 self-organization and priming phosphorylation of HsCYK-4 at the spindle midzone regulate the onset of division in human cells

Centriole Overduplication is the Predominant Mechanism Leading to Centrosome Amplification in Melanoma

Metabolic Heterogeneity in Patient Tumor-Derived Organoids by Primary Site and Drug Treatment

High nuclear TPX2 expression correlates with TP53 mutation and poor clinical behavior in a large breast cancer cohort, but is not an independent predictor of chromosomal instability

Diverse microtubule-targeted anticancer agents kill cells by inducing chromosome missegregation on multipolar spindles

Polo-like kinase 4 maintains centriolar satellite integrity by phosphorylation of centrosomal protein 131 (CEP131)

Quantifying chromosomal instability from intratumoral karyotype diversity using agent-based modeling and Bayesian inference

Accuracy of models to prognosticate survival after surgery for pancreatic cancer in the era of neoadjuvant therapy

Chemical genetics reveals the requirement for Polo-like kinase 1 activity in positioning RhoA and triggering cytokinesis in human cells

Photosensitive lichenoid skin reaction to capecitabine

Synchronous bilateral breast cancer in a patient with Nager syndrome

Centrosome amplification is a frequent event in circulating tumor cells from subjects with metastatic breast cancer

The reckoning of chromosomal instability: past, present, future

Clinical utility of a regional precision medicine molecular tumor board and challenges to implementation

Acquisition of Cabozantinib-Sensitive MET D1228N Mutation During Progression on Crizotinib in MET-Amplified Triple-Negative Breast Cancer

The Functional Significance of Posttranslational Modifications on Polo-Like Kinase 1 Revealed by Chemical Genetic Complementation

Classes of therapeutics to amplify the immune response

A physician-scientist preceptorship in clinical and translational research enhances training and mentorship

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