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Inter-individual predictors of pain inhibition during performance of a competing cognitive task

Scientific reports, 2020-12, Vol.10 (1), p.21785-21785, Article 21785

2020

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Predictors and impact of postoperative atrial fibrillation following thoracic surgery: a state‐of‐the‐art review

Anaesthesia, 2023-04, Vol.78 (4), p.491-500

2023

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Hypotrichosis-lymphedema-telangiectasia-renal defect associated with a truncating mutation in the SOX18 gene

Clinical genetics, 2015-04, Vol.87 (4), p.378-382

2015

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Mutations in the VEGFR3 Signaling Pathway Explain 36% of Familial Lymphedema

Molecular syndromology, 2013-09, Vol.4 (6), p.257-266

2013

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Human chromosome map of lymphedema-lymphangiogenesis genes: Template for current and future discovery

Lymphology, 2021, Vol.54 (4), p.167

2021

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Splice‐site mutations in VEGFC cause loss of function and Nonne‐Milroy‐like primary lymphedema

Clinical genetics, 2018-07, Vol.94 (1), p.179-181

2018

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Vascular malformations: localized defects in vascular morphogenesis

Clinical genetics, 2003-05, Vol.63 (5), p.340-351

2003

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Four common glomulin mutations cause two thirds of glomuvenous malformations (“familial glomangiomas”): evidence for a founder effect

Journal of medical genetics, 2005-02, Vol.42 (2), p.e13-e13

2005

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Genetic causes of vascular malformations

Human molecular genetics, 2007-10, Vol.16 (R2), p.R140-R149

2007

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Loss of ADAMTS3 activity causes Hennekam lymphangiectasia-lymphedema syndrome 3

Human molecular genetics, 2017-11, Vol.26 (21), p.4095-4104

2017

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Genotypes and Phenotypes of 162 Families with a Glomulin Mutation

Molecular syndromology, 2013-04, Vol.4 (4), p.157-164

2013

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Antenatal presentation of hereditary lymphedema type I

European journal of medical genetics, 2015-06, Vol.58 (6), p.329-331

2015

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EVALUATION OF REAL-WORLD MANAGEMENT STRATEGIES ADOPTED IN PATIENTS WITH ATRIAL FIBRILLATION ON ANTICOAGULANTS FOLLOWING AN EPISODE OF CLINICALLY SIGNIFICANT BLEEDING

Canadian journal of cardiology, 2023-10, Vol.39 (10), p.S198-S198

2023

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EVALUATION OF REAL-WORLD MANAGEMENT STRATEGIES ADOPTED IN PATIENTS WITH ATRIAL FIBRILLATION ON ANTICOAGULANTS FOLLOWING AN EPISODE OF CLINICALLY SIGNIFICANT BLEEDING

Canadian journal of diabetes, 2023-11, Vol.47 (7), p.S163-S163

2023

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Mutations in a Novel Factor, Glomulin, Are Responsible for Glomuvenous Malformations (“Glomangiomas”)

American journal of human genetics, 2002-04, Vol.70 (4), p.866-874

2002

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Hydrodynamics in Meandering Compound Channels with Varied Emergent Floodplain Vegetation Densities: A 3D Numerical Modeling Study

Brouillard, Nicolas P

2021

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ProQuest

A Gene for Inherited Cutaneous Venous Anomalies (“Glomangiomas”) Localizes to Chromosome 1p21-22

American journal of human genetics, 1999-07, Vol.65 (1), p.125-133

1999

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Nonvisualized Kidney on Bone Scintigraphy Due to a Depressed Diaphragm

Clinical nuclear medicine, 1994-09, Vol.19 (9), p.832-833

1994

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HIV/AIDS and Croatian migrant workers

Collegium antropologicum, 2006-12, Vol.30 Suppl 2 (Supp. 2), p.105-114

2006

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BARRIERS TO CARE: ETHNIC DIFFERENCES IN ELDERLY ATTITUDES TOWARD MENTAL HEALTH CARE PROVIDERS

The Gerontologist, 2004-10, Vol.44 (1), p.648-649

2004

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Glomulin is predominantly expressed in vascular smooth muscle cells in the embryonic and adult mouse

Gene Expression Patterns, 2004-05, Vol.4 (3), p.351-358

2004

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High-Resolution Physical and Transcript Map of the Locus for Venous Malformations with Glomus Cells (VMGLOM) on Chromosome 1p21–p22

Genomics (San Diego, Calif.), 2000-07, Vol.67 (1), p.96-101

2000

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Glomulin is a specific marker of vascular smooth muscle cell differentiation

Vascular pharmacology, 2006-09, Vol.45 (3), p.e122-e122

2006

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Lack of glomulin, a specific marker of vascular smooth muscle cell differentiation, causes glomuvenous malformations

Vascular pharmacology, 2006-09, Vol.45 (3), p.183-184

2006

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Linkage disequilibrium narrows locus for venous malformation with glomus cells (VMGLOM) to a single 1.48 Mbp YAC

European journal of human genetics : EJHG, 2001-01, Vol.9 (1), p.34-38

2001

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Inter-individual predictors of pain inhibition during performance of a competing cognitive task

Predictors and impact of postoperative atrial fibrillation following thoracic surgery: a state‐of‐the‐art review

Hypotrichosis-lymphedema-telangiectasia-renal defect associated with a truncating mutation in the SOX18 gene

Mutations in the VEGFR3 Signaling Pathway Explain 36% of Familial Lymphedema

Human chromosome map of lymphedema-lymphangiogenesis genes: Template for current and future discovery

Splice‐site mutations in VEGFC cause loss of function and Nonne‐Milroy‐like primary lymphedema

Vascular malformations: localized defects in vascular morphogenesis

Four common glomulin mutations cause two thirds of glomuvenous malformations (“familial glomangiomas”): evidence for a founder effect

Genetic causes of vascular malformations

Loss of ADAMTS3 activity causes Hennekam lymphangiectasia-lymphedema syndrome 3

Genotypes and Phenotypes of 162 Families with a Glomulin Mutation

Antenatal presentation of hereditary lymphedema type I

EVALUATION OF REAL-WORLD MANAGEMENT STRATEGIES ADOPTED IN PATIENTS WITH ATRIAL FIBRILLATION ON ANTICOAGULANTS FOLLOWING AN EPISODE OF CLINICALLY SIGNIFICANT BLEEDING

EVALUATION OF REAL-WORLD MANAGEMENT STRATEGIES ADOPTED IN PATIENTS WITH ATRIAL FIBRILLATION ON ANTICOAGULANTS FOLLOWING AN EPISODE OF CLINICALLY SIGNIFICANT BLEEDING

Mutations in a Novel Factor, Glomulin, Are Responsible for Glomuvenous Malformations (“Glomangiomas”)

Hydrodynamics in Meandering Compound Channels with Varied Emergent Floodplain Vegetation Densities: A 3D Numerical Modeling Study

A Gene for Inherited Cutaneous Venous Anomalies (“Glomangiomas”) Localizes to Chromosome 1p21-22

Nonvisualized Kidney on Bone Scintigraphy Due to a Depressed Diaphragm

HIV/AIDS and Croatian migrant workers

BARRIERS TO CARE: ETHNIC DIFFERENCES IN ELDERLY ATTITUDES TOWARD MENTAL HEALTH CARE PROVIDERS

Glomulin is predominantly expressed in vascular smooth muscle cells in the embryonic and adult mouse

High-Resolution Physical and Transcript Map of the Locus for Venous Malformations with Glomus Cells (VMGLOM) on Chromosome 1p21–p22

Glomulin is a specific marker of vascular smooth muscle cell differentiation

Lack of glomulin, a specific marker of vascular smooth muscle cell differentiation, causes glomuvenous malformations

Linkage disequilibrium narrows locus for venous malformation with glomus cells (VMGLOM) to a single 1.48 Mbp YAC

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