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A transcription factor is the target of propranolol treatment in infantile hemangioma

The Journal of clinical investigation, 2022-02, Vol.132 (3)

2022

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Corticosteroid Suppression of VEGF-A in Infantile Hemangioma-Derived Stem Cells

The New England journal of medicine, 2010-03, Vol.362 (11), p.1005-1013

2010

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VEGFR-1 Mediates Endothelial Differentiation and Formation of Blood Vessels in a Murine Model of Infantile Hemangioma

The American journal of pathology, 2011-11, Vol.179 (5), p.2266-2277

2011

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Constitutive Active Mutant TIE2 Induces Enlarged Vascular Lumen Formation with Loss of Apico-basal Polarity and Pericyte Recruitment

Scientific reports, 2019-08, Vol.9 (1), p.12352-12, Article 12352

2019

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Endoglin regulates mural cell adhesion in the circulatory system

Cellular and molecular life sciences : CMLS, 2016-04, Vol.73 (8), p.1715-1739

2016

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Constitutively active PIK3CA mutations are expressed by lymphatic and vascular endothelial cells in capillary lymphatic venous malformation

Angiogenesis (London), 2020-08, Vol.23 (3), p.425-442

2020

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MEK inhibition reduced vascular tumor growth and coagulopathy in a mouse model with hyperactive GNAQ

Nature communications, 2023-04, Vol.14 (1), p.1929-1929, Article 1929

2023

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EGFL6 Regulates the Asymmetric Division, Maintenance, and Metastasis of ALDH+ Ovarian Cancer Cells

Cancer research (Chicago, Ill.), 2016-11, Vol.76 (21), p.6396-6409

2016

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A transcription factor is the target of propranolol treatment in infantile hemangioma

The Journal of clinical investigation, 2022-02, Vol.132 (3), p.1-3

2022

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Combined mTOR and MEK inhibition is an effective therapy in a novel mouse model for angiosarcoma

Oncotarget, 2018-05, Vol.9 (37), p.24750-24765

2018

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SOCS3 is an endogenous inhibitor of pathologic angiogenesis

Blood, 2012-10, Vol.120 (14), p.2925-2929

2012

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A Xenograft Model for Venous Malformation

Methods in molecular biology (Clifton, N.J.), 2021, Vol.2206, p.179

2021

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Cellular and molecular mechanisms of PIK3CA-related vascular anomalies

Vascular biology, 2019-06, Vol.1 (1), p.H33-H40

2019

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Cooperation between human fibrocytes and endothelial colony-forming cells increases angiogenesis via the CXCR4 pathway

Thrombosis and haemostasis, 2014, Vol.112 (5), p.1002

2014

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Vasculogenesis in infantile hemangioma

Angiogenesis (London), 2009-06, Vol.12 (2), p.197-207

2009

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Endothelial progenitor cells from infantile hemangioma and umbilical cord blood display unique cellular responses to endostatin

Blood, 2006-08, Vol.108 (3), p.915-921

2006

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Capillary malformations

The Journal of clinical investigation, 2024-04, Vol.134 (8), p.1-9

2024

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Ablation of Venous Malformations by Photothermal Therapy with Intravenous Gold Nanoshells

Nano letters, 2023-08, Vol.23 (15), p.7092-7099

2023

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Pericytes from infantile hemangioma display proangiogenic properties and dysregulated angiopoietin-1

Arteriosclerosis, thrombosis, and vascular biology, 2013-03, Vol.33 (3), p.501

2013

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Rapamycin improves TIE2-mutated venous malformation in murine model and human subjects

The Journal of clinical investigation, 2015-09, Vol.125 (9), p.3491-3504

2015

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Suppressed NFAT-dependent VEGFR1 expression and constitutive VEGFR2 signaling in infantile hemangioma

Nature medicine, 2008-11, Vol.14 (11), p.1236-1246

2008

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Protocol for three-dimensional whole-mount imaging of the vascular network in the intestinal muscle

STAR protocols, 2024-07, Vol.5 (3), p.103170, Article 103170

2024

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AKT hyper-phosphorylation associated with PI3K mutations in lymphatic endothelial cells from a patient with lymphatic malformation

Angiogenesis (London), 2015-04, Vol.18 (2), p.151-162

2015

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Ponatinib Combined With Rapamycin Causes Regression of Murine Venous Malformation

Arteriosclerosis, thrombosis, and vascular biology, 2019-03, Vol.39 (3), p.496

2019

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NRASQ61R mutation in human endothelial cells causes vascular malformations

Angiogenesis (London), 2022-08, Vol.25 (3), p.331-342

2022

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A transcription factor is the target of propranolol treatment in infantile hemangioma

Corticosteroid Suppression of VEGF-A in Infantile Hemangioma-Derived Stem Cells

VEGFR-1 Mediates Endothelial Differentiation and Formation of Blood Vessels in a Murine Model of Infantile Hemangioma

Constitutive Active Mutant TIE2 Induces Enlarged Vascular Lumen Formation with Loss of Apico-basal Polarity and Pericyte Recruitment

Endoglin regulates mural cell adhesion in the circulatory system

Constitutively active PIK3CA mutations are expressed by lymphatic and vascular endothelial cells in capillary lymphatic venous malformation

MEK inhibition reduced vascular tumor growth and coagulopathy in a mouse model with hyperactive GNAQ

EGFL6 Regulates the Asymmetric Division, Maintenance, and Metastasis of ALDH+ Ovarian Cancer Cells

A transcription factor is the target of propranolol treatment in infantile hemangioma

Combined mTOR and MEK inhibition is an effective therapy in a novel mouse model for angiosarcoma

SOCS3 is an endogenous inhibitor of pathologic angiogenesis

A Xenograft Model for Venous Malformation

Cellular and molecular mechanisms of PIK3CA-related vascular anomalies

Cooperation between human fibrocytes and endothelial colony-forming cells increases angiogenesis via the CXCR4 pathway

Vasculogenesis in infantile hemangioma

Endothelial progenitor cells from infantile hemangioma and umbilical cord blood display unique cellular responses to endostatin

Capillary malformations

Ablation of Venous Malformations by Photothermal Therapy with Intravenous Gold Nanoshells

Pericytes from infantile hemangioma display proangiogenic properties and dysregulated angiopoietin-1

Rapamycin improves TIE2-mutated venous malformation in murine model and human subjects

Suppressed NFAT-dependent VEGFR1 expression and constitutive VEGFR2 signaling in infantile hemangioma

Protocol for three-dimensional whole-mount imaging of the vascular network in the intestinal muscle

AKT hyper-phosphorylation associated with PI3K mutations in lymphatic endothelial cells from a patient with lymphatic malformation

Ponatinib Combined With Rapamycin Causes Regression of Murine Venous Malformation

NRASQ61R mutation in human endothelial cells causes vascular malformations

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