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Huntington's disease induced cardiac amyloidosis is reversed by modulating protein folding and oxidative stress pathways in the Drosophila heart

PLoS genetics, 2013-12, Vol.9 (12), p.e1004024

2013

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The R369 Myosin Residue within Loop 4 Is Critical for Actin Binding and Muscle Function in Drosophila

International journal of molecular sciences, 2022-02, Vol.23 (5), p.2533

2022

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TRiC/CCT chaperonins are essential for maintaining myofibril organization, cardiac physiological rhythm, and lifespan

FEBS letters, 2017-11, Vol.591 (21), p.3447-3458

2017

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Vinculin network-mediated cytoskeletal remodeling regulates contractile function in the aging heart

Science translational medicine, 2015-06, Vol.7 (292), p.292ra99

2015

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Disrupting the Myosin Converter-Relay Interface Impairs Drosophila Indirect Flight Muscle Performance

Biophysical journal, 2011-09, Vol.101 (5), p.1114-1122

2011

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Myosin Transducer Inter-Strand Communication Is Critical for Normal ATPase Activity and Myofibril Structure

Biology (Basel, Switzerland), 2022-07, Vol.11 (8), p.1137

2022

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Profilin modulates sarcomeric organization and mediates cardiomyocyte hypertrophy

Cardiovascular research, 2016-05, Vol.110 (2), p.238-248

2016

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Prolonged cross-bridge binding triggers muscle dysfunction in a Drosophila model of myosin-based hypertrophic cardiomyopathy

eLife, 2018-08, Vol.7

2018

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The UNC-45 chaperone is critical for establishing myosin-based myofibrillar organization and cardiac contractility in the Drosophila heart model

PloS one, 2011-07, Vol.6 (7), p.e22579-e22579

2011

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A mighty small heart: the cardiac proteome of adult Drosophila melanogaster

PloS one, 2011-04, Vol.6 (4), p.e18497

2011

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Getting Folded: Chaperone Proteins in Muscle Development, Maintenance and Disease

Anatomical record (Hoboken, N.J. : 2007), 2014-09, Vol.297 (9), p.1637-1649

2014

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Drosophila myosin mutants model the disparate severity of type 1 and type 2B distal arthrogryposis and indicate an enhanced actin affinity mechanism

Skeletal muscle, 2020-08, Vol.10 (1), p.1-24, Article 24

2020

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The NADPH metabolic network regulates human αB-crystallin cardiomyopathy and reductive stress in Drosophila melanogaster

PLoS genetics, 2013-06, Vol.9 (6), p.e1003544-e1003544

2013

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A Drosophila model of dominant inclusion body myopathy type 3 shows diminished myosin kinetics that reduce muscle power and yield myofibrillar defects

Disease models & mechanisms, 2017-06, Vol.10 (6), p.761-771

2017

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X-ray Crystal Structure of the UCS Domain-Containing UNC-45 Myosin Chaperone from Drosophila melanogaster

Structure (London), 2011-03, Vol.19 (3), p.397-408

2011

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Expression of the inclusion body myopathy 3 mutation in Drosophila depresses myosin function and stability and recapitulates muscle inclusions and weakness

Molecular biology of the cell, 2012-06, Vol.23 (11), p.2057-2065

2012

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Interacting-heads motif has been conserved as a mechanism of myosin II inhibition since before the origin of animals

Proceedings of the National Academy of Sciences - PNAS, 2018-02, Vol.115 (9), p.E1991-E2000

2018

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Prolonged myosin binding increases muscle stiffness in Drosophila models of Freeman-Sheldon syndrome

Biophysical journal, 2021-03, Vol.120 (5), p.844-854

2021

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X-ray diffraction from flight muscle with a headless myosin mutation: implications for interpreting reflection patterns

Frontiers in physiology, 2014-10, Vol.5, p.416-416

2014

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Aging‐affiliated post‐translational modifications of skeletal muscle myosin affect biochemical properties, myofibril structure, muscle function, and proteostasis

Aging cell, 2024-06, Vol.23 (6), p.e14134-n/a

2024

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Alternative N-terminal regions of Drosophila myosin heavy chain II regulate communication of the purine binding loop with the essential light chain

The Journal of biological chemistry, 2020-10, Vol.295 (42), p.14522-14535

2020

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The R249Q hypertrophic cardiomyopathy myosin mutation decreases contractility in Drosophila by impeding force production

The Journal of physiology, 2019-05, Vol.597 (9), p.2403-2420

2019

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Reductions in ATPase activity, actin sliding velocity, and myofibril stability yield muscle dysfunction in Drosophila models of myosin-based Freeman-Sheldon syndrome

Molecular biology of the cell, 2019-01, Vol.30 (1), p.30-41

2019

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The UNC-45 myosin chaperone: from worms to flies to vertebrates

International review of cell and molecular biology, 2014, Vol.313, p.103-144

2014

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X-ray Crystallographic and Molecular Dynamic Analyses of Drosophila melanogaster Embryonic Muscle Myosin Define Domains Responsible for Isoform-Specific Properties

Journal of molecular biology, 2020-01, Vol.432 (2), p.427-447

2020

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Huntington's disease induced cardiac amyloidosis is reversed by modulating protein folding and oxidative stress pathways in the Drosophila heart

The R369 Myosin Residue within Loop 4 Is Critical for Actin Binding and Muscle Function in Drosophila

TRiC/CCT chaperonins are essential for maintaining myofibril organization, cardiac physiological rhythm, and lifespan

Vinculin network-mediated cytoskeletal remodeling regulates contractile function in the aging heart

Disrupting the Myosin Converter-Relay Interface Impairs Drosophila Indirect Flight Muscle Performance

Myosin Transducer Inter-Strand Communication Is Critical for Normal ATPase Activity and Myofibril Structure

Profilin modulates sarcomeric organization and mediates cardiomyocyte hypertrophy

Prolonged cross-bridge binding triggers muscle dysfunction in a Drosophila model of myosin-based hypertrophic cardiomyopathy

The UNC-45 chaperone is critical for establishing myosin-based myofibrillar organization and cardiac contractility in the Drosophila heart model

A mighty small heart: the cardiac proteome of adult Drosophila melanogaster

Getting Folded: Chaperone Proteins in Muscle Development, Maintenance and Disease

Drosophila myosin mutants model the disparate severity of type 1 and type 2B distal arthrogryposis and indicate an enhanced actin affinity mechanism

The NADPH metabolic network regulates human αB-crystallin cardiomyopathy and reductive stress in Drosophila melanogaster

A Drosophila model of dominant inclusion body myopathy type 3 shows diminished myosin kinetics that reduce muscle power and yield myofibrillar defects

X-ray Crystal Structure of the UCS Domain-Containing UNC-45 Myosin Chaperone from Drosophila melanogaster

Expression of the inclusion body myopathy 3 mutation in Drosophila depresses myosin function and stability and recapitulates muscle inclusions and weakness

Interacting-heads motif has been conserved as a mechanism of myosin II inhibition since before the origin of animals

Prolonged myosin binding increases muscle stiffness in Drosophila models of Freeman-Sheldon syndrome

X-ray diffraction from flight muscle with a headless myosin mutation: implications for interpreting reflection patterns

Aging‐affiliated post‐translational modifications of skeletal muscle myosin affect biochemical properties, myofibril structure, muscle function, and proteostasis

Alternative N-terminal regions of Drosophila myosin heavy chain II regulate communication of the purine binding loop with the essential light chain

The R249Q hypertrophic cardiomyopathy myosin mutation decreases contractility in Drosophila by impeding force production

Reductions in ATPase activity, actin sliding velocity, and myofibril stability yield muscle dysfunction in Drosophila models of myosin-based Freeman-Sheldon syndrome

The UNC-45 myosin chaperone: from worms to flies to vertebrates

X-ray Crystallographic and Molecular Dynamic Analyses of Drosophila melanogaster Embryonic Muscle Myosin Define Domains Responsible for Isoform-Specific Properties

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