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Depressed pacemaker activity of sinoatrial node myocytes contributes to the age-dependent decline in maximum heart rate

Proceedings of the National Academy of Sciences - PNAS, 2013-10, Vol.110 (44), p.18011-18016

2013

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Triclosan impairs excitation–contraction coupling and Ca²⁺ dynamics in striated muscle

Proceedings of the National Academy of Sciences - PNAS, 2012-08, Vol.109 (35), p.14158-14163

2012

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Selective posttranslational inhibition of CaVβ1-associated voltage-dependent calcium channels with a functionalized nanobody

Nature communications, 2022-12, Vol.13 (1), p.7556-7556, Article 7556

2022

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Advances in CaV1.1 gating: New insights into permeation and voltage-sensing mechanisms

Channels (Austin, Tex.), 2023-12, Vol.17 (1), p.2167569-2167569

2023

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Complex effects on CaV2.1 channel gating caused by a CACNA1A variant associated with a severe neurodevelopmental disorder

Scientific reports, 2022-06, Vol.12 (1), p.9186-9186, Article 9186

2022

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Zebrafish as a Model System for the Study of Severe CaV2.1 (α1A) Channelopathies

Frontiers in molecular neuroscience, 2020-02, Vol.12, p.329-329

2020

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The role of action potential changes in depolarization-induced failure of excitation contraction coupling in mouse skeletal muscle

eLife, 2022-01, Vol.11

2022

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Malignant hyperthermia susceptibility arising from altered resting coupling between the skeletal muscle L-type Ca²⁺ channel and the type 1 ryanodine receptor

Proceedings of the National Academy of Sciences - PNAS, 2012-05, Vol.109 (20), p.7923-7928

2012

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Impaired Gating of an L-Type Ca2+ Channel Carrying a Mutation Linked to Malignant Hyperthermia

Biophysical journal, 2013-05, Vol.104 (9), p.1917-1922

2013

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Bridging the myoplasmic gap II: more recent advances in skeletal muscle excitation-contraction coupling

Journal of experimental biology, 2016-01, Vol.219 (Pt 2), p.175-182

2016

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A neurodevelopmental disorder caused by a dysfunctional CACNA1A allele

eNeurologicalSci, 2023-06, Vol.31, p.100456-100456, Article 100456

2023

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Cryopreservation and post-thaw characterization of dissociated human islet cells

PloS one, 2022-01, Vol.17 (1), p.e0263005-e0263005

2022

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Looking for answers to EC coupling's persistent questions

The Journal of general physiology, 2010-07, Vol.136 (1), p.7-12

2010

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Differential voltage-dependent modulation of the ACh-gated K+ current by adenosine and acetylcholine

PloS one, 2022-01, Vol.17 (1), p.e0261960-e0261960

2022

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Countering reproducibility issues in mathematical models with software engineering techniques: A case study using a one-dimensional mathematical model of the atrioventricular node

PloS one, 2021-07, Vol.16 (7), p.e0254749-e0254749

2021

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Bridging the myoplasmic gap: recent developments in skeletal muscle excitation-contraction coupling

Journal of muscle research and cell motility, 2007-04, Vol.28 (4-5), p.275-283

Bannister, Roger A

2007

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Ca(V)1.1: The atypical prototypical voltage-gated Ca²⁺ channel

Biochimica et biophysica acta, 2013-07, Vol.1828 (7), p.1587

2013

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A skeletal muscle L-type Ca2+ channel with a mutation in the selectivity filter (CaV1.1 E1014K) conducts K

The Journal of biological chemistry, 2018-03, Vol.293 (9), p.3126-3133

2018

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Into the spotlight: RGK proteins in skeletal muscle

Cell calcium (Edinburgh), 2021-09, Vol.98, p.102439-102439, Article 102439

2021

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A mutation in CaV2.1 linked to a severe neurodevelopmental disorder impairs channel gating

The Journal of general physiology, 2019-06, Vol.151 (6), p.850-859

2019

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Voltage sensor movements of CaV1.1 during an action potential in skeletal muscle fibers

Proceedings of the National Academy of Sciences - PNAS, 2021-10, Vol.118 (40), p.1-7

2021

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A malignant hyperthermia-inducing mutation in RYR1 (R163C): alterations in Ca2+ entry, release, and retrograde signaling to the DHPR

The Journal of general physiology, 2010-06, Vol.135 (6), p.619-628

2010

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Advances in Ca V 1.1 gating: New insights into permeation and voltage-sensing mechanisms

Channels (Austin, Tex.), 2023-12, Vol.17 (1), p.2167569

2023

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A malignant hyperthermia-inducing mutation in RYR1 (R163C): consequent alterations in the functional properties of DHPR channels

The Journal of general physiology, 2010-06, Vol.135 (6), p.629-640

2010

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Mapping Sites of Potential Proximity between the Dihydropyridine Receptor and RyR1 in Muscle Using a Cyan Fluorescent Protein-Yellow Fluorescent Protein Tandem as a Fluorescence Resonance Energy Transfer Probe

The Journal of biological chemistry, 2004-10, Vol.279 (42), p.44046-44056

2004

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Depressed pacemaker activity of sinoatrial node myocytes contributes to the age-dependent decline in maximum heart rate

Triclosan impairs excitation–contraction coupling and Ca²⁺ dynamics in striated muscle

Selective posttranslational inhibition of CaVβ1-associated voltage-dependent calcium channels with a functionalized nanobody

Advances in CaV1.1 gating: New insights into permeation and voltage-sensing mechanisms

Complex effects on CaV2.1 channel gating caused by a CACNA1A variant associated with a severe neurodevelopmental disorder

Zebrafish as a Model System for the Study of Severe CaV2.1 (α1A) Channelopathies

The role of action potential changes in depolarization-induced failure of excitation contraction coupling in mouse skeletal muscle

Malignant hyperthermia susceptibility arising from altered resting coupling between the skeletal muscle L-type Ca²⁺ channel and the type 1 ryanodine receptor

Impaired Gating of an L-Type Ca2+ Channel Carrying a Mutation Linked to Malignant Hyperthermia

Bridging the myoplasmic gap II: more recent advances in skeletal muscle excitation-contraction coupling

A neurodevelopmental disorder caused by a dysfunctional CACNA1A allele

Cryopreservation and post-thaw characterization of dissociated human islet cells

Looking for answers to EC coupling's persistent questions

Differential voltage-dependent modulation of the ACh-gated K+ current by adenosine and acetylcholine

Countering reproducibility issues in mathematical models with software engineering techniques: A case study using a one-dimensional mathematical model of the atrioventricular node

Bridging the myoplasmic gap: recent developments in skeletal muscle excitation-contraction coupling

Ca(V)1.1: The atypical prototypical voltage-gated Ca²⁺ channel

A skeletal muscle L-type Ca2+ channel with a mutation in the selectivity filter (CaV1.1 E1014K) conducts K

Into the spotlight: RGK proteins in skeletal muscle

A mutation in CaV2.1 linked to a severe neurodevelopmental disorder impairs channel gating

Voltage sensor movements of CaV1.1 during an action potential in skeletal muscle fibers

A malignant hyperthermia-inducing mutation in RYR1 (R163C): alterations in Ca2+ entry, release, and retrograde signaling to the DHPR

Advances in Ca V 1.1 gating: New insights into permeation and voltage-sensing mechanisms

A malignant hyperthermia-inducing mutation in RYR1 (R163C): consequent alterations in the functional properties of DHPR channels

Mapping Sites of Potential Proximity between the Dihydropyridine Receptor and RyR1 in Muscle Using a Cyan Fluorescent Protein-Yellow Fluorescent Protein Tandem as a Fluorescence Resonance Energy Transfer Probe

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