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Metabolic drivers of non-alcoholic fatty liver disease

Molecular metabolism (Germany), 2021-08, Vol.50, p.101143-101143, Article 101143

2021

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Leptin and Insulin Act on POMC Neurons to Promote the Browning of White Fat

Cell, 2015-01, Vol.160 (1-2), p.88-104

2015

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Metabolic Targets in Nonalcoholic Fatty Liver Disease

Cellular and molecular gastroenterology and hepatology, 2019-01, Vol.8 (2), p.247-267

2019

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Intracellular Signals Mediating the Food Intake-Suppressive Effects of Hindbrain Glucagon-like Peptide-1 Receptor Activation

Cell metabolism, 2011-03, Vol.13 (3), p.320-330

2011

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Adipocyte Gi signaling is essential for maintaining whole-body glucose homeostasis and insulin sensitivity

Nature communications, 2020-06, Vol.11 (1), p.2995-2995, Article 2995

2020

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Liver-specific deletion of protein tyrosine phosphatase (PTP) 1B improves obesity- and pharmacologically induced endoplasmic reticulum stress

Biochemical journal, 2011-09, Vol.438 (2), p.369-378

2011

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Elevated Hypothalamic TCPTP in Obesity Contributes to Cellular Leptin Resistance

Cell metabolism, 2011-11, Vol.14 (5), p.684-699

2011

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Activation of the hypothalamic–pituitary–adrenal axis by exogenous and endogenous GDF15

Proceedings of the National Academy of Sciences - PNAS, 2021-07, Vol.118 (27), p.1-10

2021

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Protein-tyrosine Phosphatase 1B (PTP1B) Is a Novel Regulator of Central Brain-derived Neurotrophic Factor and Tropomyosin Receptor Kinase B (TrkB) Signaling

The Journal of biological chemistry, 2014-11, Vol.289 (46), p.31682-31692

2014

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Endogenous Leptin Signaling in the Caudal Nucleus Tractus Solitarius and Area Postrema Is Required for Energy Balance Regulation

Cell metabolism, 2010-01, Vol.11 (1), p.77-83

2010

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Liver-Specific Deletion of Protein-Tyrosine Phosphatase 1B (PTP1B) Improves Metabolic Syndrome and Attenuates Diet-Induced Endoplasmic Reticulum Stress

Diabetes (New York, N.Y.), 2009-03, Vol.58 (3), p.590-599

2009

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Protein tyrosine phosphatase PTP1B is involved in hippocampal synapse formation and learning

PloS one, 2012-07, Vol.7 (7), p.e41536

2012

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Response to Kunos et al. and Lotersztajn and Mallat

The Journal of clinical investigation, 2022-01, Vol.132 (1), p.1-1

2022

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Central regulation of metabolism by protein tyrosine phosphatases

Frontiers in neuroscience, 2013-01, Vol.6, p.192-192

2013

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Pharmacologic inhibition of ketohexokinase prevents fructose-induced metabolic dysfunction

Molecular metabolism (Germany), 2021-06, Vol.48, p.101196, Article 101196

2021

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Endogenous Leptin Signaling in the Caudal Nucleus Tractus Solitarius and Area Postrema Is Required for Energy Balance Regulation

Cell metabolism, 2016-04, Vol.23 (4), p.744-744

2016

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Improved Glucose Homeostasis in Mice with Muscle-Specific Deletion of Protein-Tyrosine Phosphatase 1B

Molecular and Cellular Biology, 2007-11, Vol.27 (21), p.7727-7734

2007

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Selective activation of Gs signaling in adipocytes causes striking metabolic improvements in mice

Molecular metabolism (Germany), 2019-09, Vol.27, p.83-91

2019

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PTP1B and SHP2 in POMC neurons reciprocally regulate energy balance in mice

The Journal of clinical investigation, 2010-03, Vol.120 (3), p.720-734

2010

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Hepatic Activin E mediates liver-adipose inter-organ communication, suppressing adipose lipolysis in response to elevated serum fatty acids

Molecular metabolism (Germany), 2023-12, Vol.78, p.101830, Article 101830

2023

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Dorsal Hindbrain 5′-Adenosine Monophosphate-Activated Protein Kinase as an Intracellular Mediator of Energy Balance

Endocrinology (Philadelphia), 2009-05, Vol.150 (5), p.2175-2182

2009

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Adipocyte-specific protein tyrosine phosphatase 1B deletion increases lipogenesis, adipocyte cell size and is a minor regulator of glucose homeostasis

PloS one, 2012-02, Vol.7 (2), p.e32700-e32700

2012

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Protein tyrosine phosphatase 1B is a key regulator of IFNAR1 endocytosis and a target for antiviral therapies

Proceedings of the National Academy of Sciences - PNAS, 2012-11, Vol.109 (47), p.19226-19231

2012

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Intracellular Signals Mediating the Food Intake-Suppressive Effects of Hindbrain Glucagon-like Peptide-1 Receptor Activation

Cell metabolism, 2016-04, Vol.23 (4), p.745-745

2016

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Ptp1b deletion in pro-opiomelanocortin neurons increases energy expenditure and impairs endothelial function via TNF-α dependent mechanisms

Clinical science (1979), 2016-06, Vol.130 (11), p.881-893

2016

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Metabolic drivers of non-alcoholic fatty liver disease

Leptin and Insulin Act on POMC Neurons to Promote the Browning of White Fat

Metabolic Targets in Nonalcoholic Fatty Liver Disease

Intracellular Signals Mediating the Food Intake-Suppressive Effects of Hindbrain Glucagon-like Peptide-1 Receptor Activation

Adipocyte Gi signaling is essential for maintaining whole-body glucose homeostasis and insulin sensitivity

Liver-specific deletion of protein tyrosine phosphatase (PTP) 1B improves obesity- and pharmacologically induced endoplasmic reticulum stress

Elevated Hypothalamic TCPTP in Obesity Contributes to Cellular Leptin Resistance

Activation of the hypothalamic–pituitary–adrenal axis by exogenous and endogenous GDF15

Protein-tyrosine Phosphatase 1B (PTP1B) Is a Novel Regulator of Central Brain-derived Neurotrophic Factor and Tropomyosin Receptor Kinase B (TrkB) Signaling

Endogenous Leptin Signaling in the Caudal Nucleus Tractus Solitarius and Area Postrema Is Required for Energy Balance Regulation

Liver-Specific Deletion of Protein-Tyrosine Phosphatase 1B (PTP1B) Improves Metabolic Syndrome and Attenuates Diet-Induced Endoplasmic Reticulum Stress

Protein tyrosine phosphatase PTP1B is involved in hippocampal synapse formation and learning

Response to Kunos et al. and Lotersztajn and Mallat

Central regulation of metabolism by protein tyrosine phosphatases

Pharmacologic inhibition of ketohexokinase prevents fructose-induced metabolic dysfunction

Endogenous Leptin Signaling in the Caudal Nucleus Tractus Solitarius and Area Postrema Is Required for Energy Balance Regulation

Improved Glucose Homeostasis in Mice with Muscle-Specific Deletion of Protein-Tyrosine Phosphatase 1B

Selective activation of Gs signaling in adipocytes causes striking metabolic improvements in mice

PTP1B and SHP2 in POMC neurons reciprocally regulate energy balance in mice

Hepatic Activin E mediates liver-adipose inter-organ communication, suppressing adipose lipolysis in response to elevated serum fatty acids

Dorsal Hindbrain 5′-Adenosine Monophosphate-Activated Protein Kinase as an Intracellular Mediator of Energy Balance

Adipocyte-specific protein tyrosine phosphatase 1B deletion increases lipogenesis, adipocyte cell size and is a minor regulator of glucose homeostasis

Protein tyrosine phosphatase 1B is a key regulator of IFNAR1 endocytosis and a target for antiviral therapies

Intracellular Signals Mediating the Food Intake-Suppressive Effects of Hindbrain Glucagon-like Peptide-1 Receptor Activation

Ptp1b deletion in pro-opiomelanocortin neurons increases energy expenditure and impairs endothelial function via TNF-α dependent mechanisms

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