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Distribution of Na/K-ATPase alpha 3 isoform, a sodium-potassium P-type pump associated with rapid-onset of dystonia parkinsonism (RDP) in the adult mouse brain

Journal of comparative neurology (1911), 2011-02, Vol.519 (2), p.376-404

2011

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Glutamate-system defects behind psychiatric manifestations in a familial hemiplegic migraine type 2 disease-mutation mouse model

Scientific reports, 2016-02, Vol.6 (1), p.22047-22047, Article 22047

2016

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A prospective evaluation of the diagnostic potential of EBV-DNA in plasma and whole blood

Journal of clinical virology, 2023-10, Vol.167, p.105579-105579, Article 105579

2023

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Ectodermal dysplasia with immunodeficiency caused by a branch-point mutation in IKBKG/NEMO

Journal of allergy and clinical immunology, 2016-12, Vol.138 (6), p.1706-1709.e4

2016

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Pazopanib-Induced Liver Toxicity in Patients With Metastatic Renal Cell Carcinoma: Effect of UGT1A1 Polymorphism on Pazopanib Dose Reduction, Safety, and Patient Outcomes

Clinical genitourinary cancer, 2020-02, Vol.18 (1), p.62-68.e2

2020

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Migraine- and dystonia-related disease-mutations of Na+/K+-ATPases: Relevance of behavioral studies in mice to disease symptoms and neurological manifestations in humans

Neuroscience and biobehavioral reviews, 2012-02, Vol.36 (2), p.855-871

2012

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Mapping of the minimal inorganic phosphate transporting unit of human PiT2 suggests a structure universal to PiT-related proteins from all kingdoms of life

BMC biochemistry, 2011-05, Vol.12 (1), p.21-21

2011

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Two Highly Conserved Glutamate Residues Critical for Type III Sodium-dependent Phosphate Transport Revealed by Uncoupling Transport Function from Retroviral Receptor Function

The Journal of biological chemistry, 2002-11, Vol.277 (45), p.42741-42747

2002

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Cognitive deficits caused by a disease-mutation in the α3 Na(+)/K(+)-ATPase isoform

Scientific reports, 2016-08, Vol.6 (1), p.31972-31972, Article 31972

2016

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The Central Half of Pit2 Is Not Required for Its Function as a Retroviral Receptor

Journal of Virology, 2004-09, Vol.78 (17), p.9564-9567

2004

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Characterization of transport mechanisms and determinants critical for Na+-dependent Pi symport of the PiT family paralogs human PiT1 and PiT2

American Journal of Physiology: Cell Physiology, 2006-12, Vol.291 (6), p.C1377-C1387

2006

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Characterization of transport mechanisms and determinants critical for Na + -dependent P i symport of the PiT family paralogs human PiT1 and PiT2

American Journal of Physiology: Cell Physiology, 2006-12, Vol.291 (6), p.C1377-C1387

2006

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Evolutionary and experimental analyses of inorganic phosphate transporter PiT family reveals two related signature sequences harboring highly conserved aspartic acids critical for sodium‐dependent phosphate transport function of human PiT2

The FEBS journal, 2005-06, Vol.272 (12), p.3060-3074

2005

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Cognitive deficits caused by a disease-mutation in the [alpha]3 Na+/K+-ATPase isoform

Scientific reports, 2016-08, Vol.6, p.31972

2016

Volltextzugriff (PDF)

Characterization of transport mechanisms and determinants critical for Na+-dependent Pi symport of the PiT family paralogs human PiT1 and PiT2

American Journal of Physiology: Cell Physiology, 2006, Vol.60 (6)

2006

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Evolutionary and experimental analyses of inorganic phosphate transporter PiT family reveals two related signature sequences harboring highly conserved aspartic acids

The FEBS journal, 2005-06, Vol.272 (12), p.3060

2005

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Ectodermal dysplasia with immunodeficiency caused by a branch-point mutation in IKBKG/NEMO

The Journal of allergy and clinical immunology, 2016, Vol.138 (6), p.1706-1709.e4

2016

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Distribution of Na/K-ATPase alpha 3 isoform, a sodium-potassium P-type pump associated with rapid-onset of dystonia parkinsonism (RDP) in the adult mouse brain

Glutamate-system defects behind psychiatric manifestations in a familial hemiplegic migraine type 2 disease-mutation mouse model

A prospective evaluation of the diagnostic potential of EBV-DNA in plasma and whole blood

Ectodermal dysplasia with immunodeficiency caused by a branch-point mutation in IKBKG/NEMO

Dihydropyrimidine dehydrogenase (DPD) genotype and phenotype among Danish cancer patients: prevalence and correlation between DPYD-genotype variants and P-uracil concentrations

Pazopanib-Induced Liver Toxicity in Patients With Metastatic Renal Cell Carcinoma: Effect of UGT1A1 Polymorphism on Pazopanib Dose Reduction, Safety, and Patient Outcomes

Migraine- and dystonia-related disease-mutations of Na+/K+-ATPases: Relevance of behavioral studies in mice to disease symptoms and neurological manifestations in humans

Mapping of the minimal inorganic phosphate transporting unit of human PiT2 suggests a structure universal to PiT-related proteins from all kingdoms of life

Two Highly Conserved Glutamate Residues Critical for Type III Sodium-dependent Phosphate Transport Revealed by Uncoupling Transport Function from Retroviral Receptor Function

Cognitive deficits caused by a disease-mutation in the α3 Na(+)/K(+)-ATPase isoform

The Central Half of Pit2 Is Not Required for Its Function as a Retroviral Receptor

Characterization of transport mechanisms and determinants critical for Na+-dependent Pi symport of the PiT family paralogs human PiT1 and PiT2

Characterization of transport mechanisms and determinants critical for Na + -dependent P i symport of the PiT family paralogs human PiT1 and PiT2

Evolutionary and experimental analyses of inorganic phosphate transporter PiT family reveals two related signature sequences harboring highly conserved aspartic acids critical for sodium‐dependent phosphate transport function of human PiT2

Cognitive deficits caused by a disease-mutation in the [alpha]3 Na+/K+-ATPase isoform

Characterization of transport mechanisms and determinants critical for Na+-dependent Pi symport of the PiT family paralogs human PiT1 and PiT2

Evolutionary and experimental analyses of inorganic phosphate transporter PiT family reveals two related signature sequences harboring highly conserved aspartic acids

Ectodermal dysplasia with immunodeficiency caused by a branch-point mutation in IKBKG/NEMO

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