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Efficient, Complete Deletion of Synaptic Proteins using CRISPR

Neuron (Cambridge, Mass.), 2014-09, Vol.83 (5), p.1051-1057

2014

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The CaMKII/NMDA receptor complex controls hippocampal synaptic transmission by kinase-dependent and independent mechanisms

Nature communications, 2018-05, Vol.9 (1), p.2069-21, Article 2069

2018

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Dysfunction of homeostatic control of dopamine by astrocytes in the developing prefrontal cortex leads to cognitive impairments

Molecular psychiatry, 2020-04, Vol.25 (4), p.732-749

2020

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Synaptic Vesicle Recycling Pathway Determines Neurotransmitter Content and Release Properties

Neuron (Cambridge, Mass.), 2019-05, Vol.102 (4), p.786-800.e5

2019

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Identification of the functional domain of the dense core vesicle biogenesis factor HID-1

PloS one, 2023-09, Vol.18 (9), p.e0291977-e0291977

2023

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Widespread dysregulation of peptide hormone release in mice lacking adaptor protein AP-3

PLoS genetics, 2013-09, Vol.9 (9), p.e1003812-e1003812

2013

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RNAi screen identifies a role for adaptor protein AP-3 in sorting to the regulated secretory pathway

The Journal of cell biology, 2010-12, Vol.191 (6), p.1173-1187

2010

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Author Correction: The CaMKII/NMDA receptor complex controls hippocampal synaptic transmission by kinase-dependent and independent mechanisms

Nature communications, 2018-12, Vol.9 (1), p.5205-2, Article 5205

2018

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253-LB: A Novel Membrane Binding Domain in Conjunction with Golga5 Recruits the Secretory Granule Biogenesis Factor HID-1 to the Golgi

Diabetes (New York, N.Y.), 2020-06, Vol.69 (Supplement_1)

ASENSIO, CEDRIC S.

2020

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Efficient, Complete Deletion of Synaptic Proteins using CRISPR

Neuron (Cambridge, Mass.), 2015-11, Vol.88 (3), p.617-617

2015

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EIPR1 controls dense-core vesicle cargo retention and EARP complex localization in insulin-secreting cells

Molecular biology of the cell, 2020-01, Vol.31 (1), p.59-79

2020

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Differential sorting behavior for soluble and transmembrane cargoes at the trans -Golgi network in endocrine cells

Molecular biology of the cell, 2020-02, Vol.31 (3), p.157-166

2020

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Self-Assembly of VPS41 Promotes Sorting Required for Biogenesis of the Regulated Secretory Pathway

Developmental cell, 2013-11, Vol.27 (4), p.425-437

2013

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HID-1 controls formation of large dense core vesicles by influencing cargo sorting and trans -Golgi network acidification

Molecular biology of the cell, 2017-12, Vol.28 (26), p.3870-3880

2017

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Pancreatic β-Cell-Specific Deletion of VPS41 Causes Diabetes Due to Defects in Insulin Secretion

Diabetes (New York, N.Y.), 2021-02, Vol.70 (2), p.436-448

2021

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Application of Cas9/CRISPR to the study of synaptic function

M.S. Médecine sciences, 2015-02, Vol.31 (2), p.137

Asensio, Cédric S

2015

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39-OR: VPS41 Contributes to Insulin Granule Biogenesis

Diabetes (New York, N.Y.), 2019-06, Vol.68 (Supplement_1)

2019

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Identification of the functional domain of the dense core vesicle biogenesis factor HID-1

PloS one, 2023-01, Vol.18 (9), p.e0291977

2023

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Application de la méthode Cas9/CRISPR à l’étude de la fonction synaptique

M.S. Médecine sciences, 2015-02, Vol.31 (2), p.137-138

Asensio, Cédric S.

2015

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Dysfunction of homeostatic control of dopamine by astrocytes in the developing prefrontal cortex leads to cognitive impairments

Molecular psychiatry, 2020-04, Vol.25 (4), p.732-749

2020

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Dissecting the Role of Synaptic Proteins with CRISPR

Genome Editing in Neurosciences, 2017

2017

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The Lack of β-Adrenoceptors Results in Enhanced Insulin Sensitivity in Mice Exhibiting Increased Adiposity and Glucose Intolerance

Diabetes (New York, N.Y.), 2005-12, Vol.54 (12), p.3490-3495

2005

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Changes in Glycemia by Leptin Administration or High- Fat Feeding in Rodent Models of Obesity/Type 2 Diabetes Suggest a Link between Resistin Expression and Control of Glucose Homeostasis

Endocrinology (Philadelphia), 2004-05, Vol.145 (5), p.2206-2213

2004

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The control of UCP1 is dissociated from that of PGC-1α or of mitochondriogenesis as revealed by a study using β-less mouse brown adipocytes in culture

FEBS letters, 2006-08, Vol.580 (19), p.4661-4666

2006

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Efficient, Complete Deletion of Synaptic Proteins using CRISPR

The CaMKII/NMDA receptor complex controls hippocampal synaptic transmission by kinase-dependent and independent mechanisms

Dysfunction of homeostatic control of dopamine by astrocytes in the developing prefrontal cortex leads to cognitive impairments

Synaptic Vesicle Recycling Pathway Determines Neurotransmitter Content and Release Properties

Identification of the functional domain of the dense core vesicle biogenesis factor HID-1

Widespread dysregulation of peptide hormone release in mice lacking adaptor protein AP-3

RNAi screen identifies a role for adaptor protein AP-3 in sorting to the regulated secretory pathway

Author Correction: The CaMKII/NMDA receptor complex controls hippocampal synaptic transmission by kinase-dependent and independent mechanisms

253-LB: A Novel Membrane Binding Domain in Conjunction with Golga5 Recruits the Secretory Granule Biogenesis Factor HID-1 to the Golgi

Efficient, Complete Deletion of Synaptic Proteins using CRISPR

EIPR1 controls dense-core vesicle cargo retention and EARP complex localization in insulin-secreting cells

Differential sorting behavior for soluble and transmembrane cargoes at the trans -Golgi network in endocrine cells

Self-Assembly of VPS41 Promotes Sorting Required for Biogenesis of the Regulated Secretory Pathway

HID-1 controls formation of large dense core vesicles by influencing cargo sorting and trans -Golgi network acidification

Pancreatic β-Cell-Specific Deletion of VPS41 Causes Diabetes Due to Defects in Insulin Secretion

Application of Cas9/CRISPR to the study of synaptic function

39-OR: VPS41 Contributes to Insulin Granule Biogenesis

Identification of the functional domain of the dense core vesicle biogenesis factor HID-1

Application de la méthode Cas9/CRISPR à l’étude de la fonction synaptique

Dysfunction of homeostatic control of dopamine by astrocytes in the developing prefrontal cortex leads to cognitive impairments

Dissecting the Role of Synaptic Proteins with CRISPR

Mutations in HID1 Cause Syndromic Infantile Encephalopathy and Hypopituitarism

The Lack of β-Adrenoceptors Results in Enhanced Insulin Sensitivity in Mice Exhibiting Increased Adiposity and Glucose Intolerance

Changes in Glycemia by Leptin Administration or High- Fat Feeding in Rodent Models of Obesity/Type 2 Diabetes Suggest a Link between Resistin Expression and Control of Glucose Homeostasis

The control of UCP1 is dissociated from that of PGC-1α or of mitochondriogenesis as revealed by a study using β-less mouse brown adipocytes in culture

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