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The MSP‐RON axis stimulates cancer cell growth in models of triple negative breast cancer

Molecular oncology, 2020-08, Vol.14 (8), p.1868-1880

2020

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Disrupting MLV integrase:BET protein interaction biases integration into quiescent chromatin and delays but does not eliminate tumor activation in a MYC/Runx2 mouse model

PLoS pathogens, 2019-12, Vol.15 (12), p.e1008154

2019

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RUNX‐mediated growth arrest and senescence are attenuated by diverse mechanisms in cells expressing RUNX1 fusion oncoproteins

Journal of cellular biochemistry, 2018-03, Vol.119 (3), p.2750-2762

2018

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Specialized Tfh cell subsets driving type-1 and type-2 humoral responses in lymphoid tissue

Cell discovery, 2024-06, Vol.10 (1), p.64-64, Article 64

2024

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Gamma-Retrovirus Integration Marks Cell Type-Specific Cancer Genes: A Novel Profiling Tool in Cancer Genomics

PloS one, 2016-04, Vol.11 (4), p.e0154070

2016

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Runx Regulation of Sphingolipid Metabolism and Survival Signaling

Cancer research (Chicago, Ill.), 2010-07, Vol.70 (14), p.5860-5869

2010

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Oncogene-induced senescence: An essential role for Runx

Cell cycle (Georgetown, Tex.), 2008-08, Vol.7 (15), p.2333-2340

2008

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Insertional mutagenesis and deep profiling reveals gene hierarchies and a Myc/p53-dependent bottleneck in lymphomagenesis

PLoS genetics, 2014-02, Vol.10 (2), p.e1004167

2014

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Runx2 disruption promotes immortalization and confers resistance to oncogene-induced senescence in primary murine fibroblasts

Cancer research (Chicago, Ill.), 2007-12, Vol.67 (23), p.11263-11271

2007

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Frequent infection of human cancer xenografts with murine endogenous retroviruses in vivo

Viruses, 2015-04, Vol.7 (4), p.2014-2029

2015

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Gut γδ T cells as guardians, disruptors, and instigators of cancer

Immunological reviews, 2020-11, Vol.298 (1), p.198-217

2020

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Evi-1 Transforming and Repressor Activities Are Mediated by CtBP Co-repressor Proteins

The Journal of biological chemistry, 2001-07, Vol.276 (28), p.25834-25840

2001

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RUNX1 transformation of primary embryonic fibroblasts is revealed in the absence of p53

Oncogene, 2004-07, Vol.23 (32), p.5476-5486

2004

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PD-1 and TIM-3 differentially regulate subsets of mouse IL-17A-producing γδ T cells

The Journal of experimental medicine, 2023-02, Vol.220 (2)

2023

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Collaboration of MYC and RUNX2 in lymphoma simulates T‐cell receptor signaling and attenuates p53 pathway activity

Journal of cellular biochemistry, 2019-10, Vol.120 (10), p.18332-18345

2019

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IL-27 maintains cytotoxic Ly6C+ γδ T cells that arise from immature precursors

The EMBO journal, 2024-05, Vol.43 (14), p.2878-2907

2024

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The RUNX Genes as Conditional Oncogenes: Insights from Retroviral Targeting and Mouse Models

Advances in experimental medicine and biology, 2017, Vol.962, p.247-264

2017

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Barriers to Infection of Human Cells by Feline Leukemia Virus: Insights into Resistance to Zoonosis

Journal of virology, 2017-03, Vol.91 (5)

2017

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Addiction to RUNX in lymphoma

Aging (Albany, NY.), 2016-09, Vol.8 (9), p.1832-1833

2016

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Gene array analysis reveals a common Runx transcriptional programme controlling cell adhesion and survival

Oncogene, 2008-10, Vol.27 (44), p.5856-5866

2008

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RUNX1 and its fusion oncoprotein derivative, RUNX1-ETO, induce senescence-like growth arrest independently of replicative stress

Oncogene, 2009-07, Vol.28 (27), p.2502-2512

2009

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Addiction to Runx1 is partially attenuated by loss of p53 in the Eμ-Myc lymphoma model

Oncotarget, 2016-04, Vol.7 (17), p.22973-22987

2016

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NKG2D signaling regulates IL-17A-producing γδT cells in mice to promote cancer progression

Discovery immunology, 2022, Vol.1 (1), p.kyac002-kyac002

2022

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The MSP‐RON axis stimulates cancer cell growth in models of triple negative breast cancer

Disrupting MLV integrase:BET protein interaction biases integration into quiescent chromatin and delays but does not eliminate tumor activation in a MYC/Runx2 mouse model

RUNX‐mediated growth arrest and senescence are attenuated by diverse mechanisms in cells expressing RUNX1 fusion oncoproteins

Specialized Tfh cell subsets driving type-1 and type-2 humoral responses in lymphoid tissue

Gamma-Retrovirus Integration Marks Cell Type-Specific Cancer Genes: A Novel Profiling Tool in Cancer Genomics

Runx Regulation of Sphingolipid Metabolism and Survival Signaling

Oncogene-induced senescence: An essential role for Runx

Insertional mutagenesis and deep profiling reveals gene hierarchies and a Myc/p53-dependent bottleneck in lymphomagenesis

Runx2 disruption promotes immortalization and confers resistance to oncogene-induced senescence in primary murine fibroblasts

Frequent infection of human cancer xenografts with murine endogenous retroviruses in vivo

Gut γδ T cells as guardians, disruptors, and instigators of cancer

Evi-1 Transforming and Repressor Activities Are Mediated by CtBP Co-repressor Proteins

RUNX1 transformation of primary embryonic fibroblasts is revealed in the absence of p53

PD-1 and TIM-3 differentially regulate subsets of mouse IL-17A-producing γδ T cells

Collaboration of MYC and RUNX2 in lymphoma simulates T‐cell receptor signaling and attenuates p53 pathway activity

IL-27 maintains cytotoxic Ly6C+ γδ T cells that arise from immature precursors

The RUNX Genes as Conditional Oncogenes: Insights from Retroviral Targeting and Mouse Models

Barriers to Infection of Human Cells by Feline Leukemia Virus: Insights into Resistance to Zoonosis

Addiction to RUNX in lymphoma

b-Catenin Drives Butyrophilin-like Molecule Loss and gd T-cell Exclusion in Colon Cancer

Gene array analysis reveals a common Runx transcriptional programme controlling cell adhesion and survival

β-Catenin Drives Butyrophilin-like Molecule Loss and γδ T-cell Exclusion in Colon Cancer

RUNX1 and its fusion oncoprotein derivative, RUNX1-ETO, induce senescence-like growth arrest independently of replicative stress

Addiction to Runx1 is partially attenuated by loss of p53 in the Eμ-Myc lymphoma model

NKG2D signaling regulates IL-17A-producing γδT cells in mice to promote cancer progression

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