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The immunology of multiple sclerosis

Nature reviews. Immunology, 2022-12, Vol.22 (12), p.734-750

2022

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A repair pathway lost in multiple sclerosis provides a new drug opportunity

Nature immunology, 2024-03, Vol.25 (3), p.385-386

2024

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CD8+ MAIT cells infiltrate into the CNS and alterations in their blood frequencies correlate with IL‐18 serum levels in multiple sclerosis

European journal of immunology, 2014-10, Vol.44 (10), p.3119-3128

2014

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TNF receptor 1 genetic risk mirrors outcome of anti-TNF therapy in multiple sclerosis

Nature (London), 2012-08, Vol.488 (7412), p.508-511

2012

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Identification of early neurodegenerative pathways in progressive multiple sclerosis

Nature neuroscience, 2022-07, Vol.25 (7), p.944-955

2022

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Structural and regulatory diversity shape HLA-C protein expression levels

Nature communications, 2017-06, Vol.8 (1), p.15924-12, Article 15924

2017

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Allosteric TYK2 inhibition: redefining autoimmune disease therapy beyond JAK1-3 inhibitors

EBioMedicine, 2023-11, Vol.97, p.104840-104840, Article 104840

2023

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Resolving TYK2 locus genotype-to-phenotype differences in autoimmunity

Science translational medicine, 2016-11, Vol.8 (363), p.363ra149

2016

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Mouse fetal growth restriction through parental and fetal immune gene variation and intercellular communications cascade

Nature communications, 2022-07, Vol.13 (1), p.4398-4398, Article 4398

2022

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Identifying CNS-colonizing T cells as potential therapeutic targets to prevent progression of multiple sclerosis

Med (New York, N.Y. : Online), 2021-03, Vol.2 (3), p.296-312.e8

2021

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Acid-sensing ion channel 1 is involved in both axonal injury and demyelination in multiple sclerosis and its animal model

Brain (London, England : 1878), 2011-02, Vol.134 (Pt 2), p.571-584

2011

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Type II (tositumomab) anti-CD20 monoclonal antibody out performs type I (rituximab-like) reagents in B-cell depletion regardless of complement activation

Blood, 2008-11, Vol.112 (10), p.4170-4177

2008

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A novel neurodegenerative spectrum disorder in patients with MLKL deficiency

Cell death & disease, 2020-05, Vol.11 (5), p.303-303, Article 303

2020

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TCR transgenic mice that shed light on immune and environmental regulators in multiple sclerosis

The Journal of immunology (1950), 2013-04, Vol.190 (7), p.3015-3017

2013

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Acute Inflammatory Diseases of the Central Nervous System After SARS-CoV-2 Vaccination

Neurology : neuroimmunology & neuroinflammation, 2023-01, Vol.10 (1)

2023

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Bridging the gap from genetic association to functional understanding: the next generation of mouse models of multiple sclerosis

Immunological reviews, 2012-07, Vol.248 (1), p.10-22

2012

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Invariant NKT Cells Promote CD8+ Cytotoxic T Cell Responses by Inducing CD70 Expression on Dendritic Cells

The Journal of immunology (1950), 2008-04, Vol.180 (7), p.4615-4620

2008

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Multiple sclerosis: Molecular mimicry of an antimyelin HLA class I restricted T-cell receptor

Neurology : neuroimmunology & neuroinflammation, 2016-08, Vol.3 (4), p.e241-e241

2016

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Bridging the gap from genetic association to functional understanding: the next generation of mouse models of multiple sclerosis: Neuroimmunology

Immunological reviews, 2012, Vol.248, p.10-22

2012

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CD 8 + MAIT cells infiltrate into the CNS and alterations in their blood frequencies correlate with IL ‐18 serum levels in multiple sclerosis

European journal of immunology, 2014-10, Vol.44 (10), p.3119-3128

2014

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CD8 super(+) MAIT cells infiltrate into the CNS and alterations in their blood frequencies correlate with IL-18 serum levels in multiple sclerosis

European journal of immunology, 2014-10, Vol.44 (10), p.3119-3128

2014

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Invariant NKT Cells Promote CD8 super(+) Cytotoxic T Cell Responses by Inducing CD70 Expression on Dendritic Cells

The Journal of immunology (1950), 2008-04, Vol.180 (7), p.4615-4620

2008

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The effect of manipulating apoptotic cell uptake on their immunogenicity

Attfield, Kathrine Elizabeth

2009

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The immunology of multiple sclerosis

A repair pathway lost in multiple sclerosis provides a new drug opportunity

CD8+ MAIT cells infiltrate into the CNS and alterations in their blood frequencies correlate with IL‐18 serum levels in multiple sclerosis

TNF receptor 1 genetic risk mirrors outcome of anti-TNF therapy in multiple sclerosis

Identification of early neurodegenerative pathways in progressive multiple sclerosis

Structural and regulatory diversity shape HLA-C protein expression levels

Allosteric TYK2 inhibition: redefining autoimmune disease therapy beyond JAK1-3 inhibitors

Resolving TYK2 locus genotype-to-phenotype differences in autoimmunity

Elevated genetic risk for multiple sclerosis emerged in steppe pastoralist populations

Mouse fetal growth restriction through parental and fetal immune gene variation and intercellular communications cascade

Identifying CNS-colonizing T cells as potential therapeutic targets to prevent progression of multiple sclerosis

Antibody agonists trigger immune receptor signaling through local exclusion of receptor-type protein tyrosine phosphatases

Acid-sensing ion channel 1 is involved in both axonal injury and demyelination in multiple sclerosis and its animal model

Type II (tositumomab) anti-CD20 monoclonal antibody out performs type I (rituximab-like) reagents in B-cell depletion regardless of complement activation

A novel neurodegenerative spectrum disorder in patients with MLKL deficiency

TCR transgenic mice that shed light on immune and environmental regulators in multiple sclerosis

Acute Inflammatory Diseases of the Central Nervous System After SARS-CoV-2 Vaccination

Bridging the gap from genetic association to functional understanding: the next generation of mouse models of multiple sclerosis

Invariant NKT Cells Promote CD8+ Cytotoxic T Cell Responses by Inducing CD70 Expression on Dendritic Cells

Multiple sclerosis: Molecular mimicry of an antimyelin HLA class I restricted T-cell receptor

Bridging the gap from genetic association to functional understanding: the next generation of mouse models of multiple sclerosis: Neuroimmunology

CD 8 + MAIT cells infiltrate into the CNS and alterations in their blood frequencies correlate with IL ‐18 serum levels in multiple sclerosis

CD8 super(+) MAIT cells infiltrate into the CNS and alterations in their blood frequencies correlate with IL-18 serum levels in multiple sclerosis

Invariant NKT Cells Promote CD8 super(+) Cytotoxic T Cell Responses by Inducing CD70 Expression on Dendritic Cells

The effect of manipulating apoptotic cell uptake on their immunogenicity

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