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The keystone of Alzheimer pathogenesis might be sought in Aβ physiology

Neuroscience, 2015-10, Vol.307, p.26-36

2015

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Extracellular Tau Oligomers Produce An Immediate Impairment of LTP and Memory

Scientific reports, 2016-01, Vol.6 (1), p.19393, Article 19393

2016

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FIRST‐IN‐CLASS HAT ACTIVATOR HIGHLY SYNERGISTIC WITH PAN‐HDAC INHIBITOR ROMIDEPSIN LEADING TO PROFOUND HISTONE ACETYLATION CYTOTOXICITY

Hematological oncology, 2019-06, Vol.37 (S2), p.125-126

2019

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Correction: Abolishing Tau cleavage by caspases at Aspartate421 causes memory/synaptic plasticity deficits and pre-pathological Tau alterations

Translational psychiatry, 2018-08, Vol.8 (1), p.1-1, Article 172

2018

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Rapid Increase in Clusters of Presynaptic Proteins at Onset of Long-Lasting Potentiation

Science (American Association for the Advancement of Science), 2001-11, Vol.294 (5546), p.1547-1550

2001

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Nitric oxide as a retrograde messenger during long-term potentiation in hippocampus

Progress in brain research, 1998, Vol.118, p.155

1998

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Tests of the Roles of Two Diffusible Substances in Long-Term Potentiation: Evidence for Nitric Oxide as a Possible Early Retrograde Messenger

Proceedings of the National Academy of Sciences - PNAS, 1991-12, Vol.88 (24), p.11285-11289

1991

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Activity-dependent long-term enhancement of transmitter release by presynaptic 3′,5′-cyclic GMP in cultured hippocampal neurons

Nature (London), 1995-07, Vol.376 (6535), p.74-80

1995

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Requirement of Hippocampal Neurogenesis for the Behavioral Effects of Antidepressants

Science (American Association for the Advancement of Science), 2003-08, Vol.301 (5634), p.805-809

2003

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Persistent improvement in synaptic and cognitive functions in an Alzheimer mouse model after rolipram treatment

The Journal of clinical investigation, 2004-12, Vol.114 (11), p.1624-1634

2004

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Learning and Memory and Synaptic Plasticity Are Impaired in a Mouse Model of Rett Syndrome

The Journal of neuroscience, 2006-01, Vol.26 (1), p.319-327

2006

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Amyloid β-Peptide Inhibition of the PKA/CREB Pathway and Long-Term Potentiation: Reversibility by Drugs That Enhance cAMP Signaling

Proceedings of the National Academy of Sciences - PNAS, 2002-10, Vol.99 (20), p.13217-13221

2002

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Phosphodiesterase 5 Inhibition Improves Synaptic Function, Memory, and Amyloid-{beta} Load in an Alzheimer's Disease Mouse Model

The Journal of neuroscience, 2009-06, Vol.29 (25), p.8075-8086

2009

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Progressive age-related development of Alzheimer-like pathology in APP/PS1 mice

Annals of neurology, 2004-06, Vol.55 (6), p.801-814

2004

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Presynaptic CaMKII Is Necessary for Synaptic Plasticity in Cultured Hippocampal Neurons

Neuron (Cambridge, Mass.), 2004-04, Vol.42 (1), p.129-141

2004

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Amyloid-beta peptide inhibits activation of the nitric oxide/cGMP/cAMP-responsive element-binding protein pathway during hippocampal synaptic plasticity

The Journal of neuroscience, 2005-07, Vol.25 (29), p.6887-6897

2005

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ABAD Directly Links Aβ to Mitochondrial Toxicity in Alzheimer's Disease

Science (American Association for the Advancement of Science), 2004-04, Vol.304 (5669), p.448-452

2004

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ABAD enhances Aβ‐induced cell stress via mitochondrial dysfunction

The FASEB journal, 2005-04, Vol.19 (6), p.1-25

2005

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RAGE potentiates Aβ-induced perturbation of neuronal function in transgenic mice

The EMBO journal, 2004-10, Vol.23 (20), p.4096-4105

2004

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Dendritic Location of Neural BC1 RNA

Proceedings of the National Academy of Sciences - PNAS, 1991-03, Vol.88 (6), p.2093-2097

1991

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α-Synuclein produces a long-lasting increase in neurotransmitter release

The EMBO journal, 2004-11, Vol.23 (22), p.4506-4516

2004

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Abolishing Tau cleavage by caspases at Aspartate421 causes memory/synaptic plasticity deficits and pre-pathological Tau alterations

Translational psychiatry, 2017-08, Vol.7 (8), p.e1198-e1198

2017

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Excitatory synaptic connections onto rat hippocampal inhibitory cells may involve a single transmitter release site

The Journal of physiology, 1994-12, Vol.481 (Pt 2), p.395-405

1994

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Calpain Mediates Calcium-Induced Activation of the Erk1,2 MAPK Pathway and Cytoskeletal Phosphorylation in Neurons: Relevance to Alzheimer's Disease

The American journal of pathology, 2004-09, Vol.165 (3), p.795-805

2004

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Correction: Abolishing Tau cleavage by caspases at Aspartate 421 causes memory/synaptic plasticity deficits and pre-pathological Tau alterations

Translational psychiatry, 2018-08, Vol.8 (1), p.172

2018

Volltextzugriff (PDF)

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The keystone of Alzheimer pathogenesis might be sought in Aβ physiology

Extracellular Tau Oligomers Produce An Immediate Impairment of LTP and Memory

FIRST‐IN‐CLASS HAT ACTIVATOR HIGHLY SYNERGISTIC WITH PAN‐HDAC INHIBITOR ROMIDEPSIN LEADING TO PROFOUND HISTONE ACETYLATION CYTOTOXICITY

Correction: Abolishing Tau cleavage by caspases at Aspartate421 causes memory/synaptic plasticity deficits and pre-pathological Tau alterations

Rapid Increase in Clusters of Presynaptic Proteins at Onset of Long-Lasting Potentiation

Nitric oxide as a retrograde messenger during long-term potentiation in hippocampus

Tests of the Roles of Two Diffusible Substances in Long-Term Potentiation: Evidence for Nitric Oxide as a Possible Early Retrograde Messenger

Activity-dependent long-term enhancement of transmitter release by presynaptic 3′,5′-cyclic GMP in cultured hippocampal neurons

Requirement of Hippocampal Neurogenesis for the Behavioral Effects of Antidepressants

Persistent improvement in synaptic and cognitive functions in an Alzheimer mouse model after rolipram treatment

Learning and Memory and Synaptic Plasticity Are Impaired in a Mouse Model of Rett Syndrome

Amyloid β-Peptide Inhibition of the PKA/CREB Pathway and Long-Term Potentiation: Reversibility by Drugs That Enhance cAMP Signaling

Phosphodiesterase 5 Inhibition Improves Synaptic Function, Memory, and Amyloid-{beta} Load in an Alzheimer's Disease Mouse Model

Progressive age-related development of Alzheimer-like pathology in APP/PS1 mice

Presynaptic CaMKII Is Necessary for Synaptic Plasticity in Cultured Hippocampal Neurons

Amyloid-beta peptide inhibits activation of the nitric oxide/cGMP/cAMP-responsive element-binding protein pathway during hippocampal synaptic plasticity

ABAD Directly Links Aβ to Mitochondrial Toxicity in Alzheimer's Disease

ABAD enhances Aβ‐induced cell stress via mitochondrial dysfunction

RAGE potentiates Aβ-induced perturbation of neuronal function in transgenic mice

Dendritic Location of Neural BC1 RNA

α-Synuclein produces a long-lasting increase in neurotransmitter release

Abolishing Tau cleavage by caspases at Aspartate421 causes memory/synaptic plasticity deficits and pre-pathological Tau alterations

Excitatory synaptic connections onto rat hippocampal inhibitory cells may involve a single transmitter release site

Calpain Mediates Calcium-Induced Activation of the Erk1,2 MAPK Pathway and Cytoskeletal Phosphorylation in Neurons: Relevance to Alzheimer's Disease

Correction: Abolishing Tau cleavage by caspases at Aspartate 421 causes memory/synaptic plasticity deficits and pre-pathological Tau alterations

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