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Extracellular Vesicles: Investigating the Pathophysiology of Diabetes-Associated Hypertension and Diabetic Nephropathy

Biology (Basel, Switzerland), 2023-08, Vol.12 (8), p.1138

Alli, Abdel A

2023

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Lipidomic and proteomic analysis of exosomes from mouse cortical collecting duct cells

The FASEB journal, 2017-12, Vol.31 (12), p.5399-5408

2017

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Exosomal GAPDH from Proximal Tubule Cells Regulate ENaC Activity

PloS one, 2016-11, Vol.11 (11), p.e0165763-e0165763

2016

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Augmentation of Cathepsin Isoforms in Diabetic db/db Mouse Kidneys Is Associated with an Increase in Renal MARCKS Expression and Proteolysis

International journal of molecular sciences, 2023-08, Vol.24 (15), p.12484

2023

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Lipid Profiles of Urinary Extracellular Vesicles Released during the Inactive and Active Phases of Aged Male Mice with Spontaneous Hypertension

International journal of molecular sciences, 2022-12, Vol.23 (23), p.15397

2022

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Dapagliflozin Treatment Augments Bioactive Phosphatidylethanolamine Concentrations in Kidney Cortex Membrane Fractions of Hypertensive Diabetic db/db Mice and Alters the Density of Lipid Rafts in Mouse Proximal Tubule Cells

International journal of molecular sciences, 2023-01, Vol.24 (2), p.1408

2023

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COVID-19 Plasma Extracellular Vesicles Increase the Density of Lipid Rafts in Human Small Airway Epithelial Cells

International journal of molecular sciences, 2023-01, Vol.24 (2), p.1654

2023

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C Type Natriuretic Peptide Receptor Activation Inhibits Sodium Channel Activity in Human Aortic Endothelial Cells by Activating the Diacylglycerol-Protein Kinase C Pathway

International journal of molecular sciences, 2022-11, Vol.23 (22), p.13959

2022

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ENaC is regulated by natriuretic peptide receptor-dependent cGMP signaling

American journal of physiology. Renal physiology, 2013-04, Vol.304 (7), p.F930-F937

2013

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Human Alpha-1 Antitrypsin Attenuates ENaC and MARCKS and Lowers Blood Pressure in Hypertensive Diabetic db/db Mice

Biomolecules (Basel, Switzerland), 2022-12, Vol.13 (1), p.66

2022

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Cathepsin B Is Secreted Apically from Xenopus 2F3 Cells and Cleaves the Epithelial Sodium Channel (ENaC) to Increase Its Activity

The Journal of biological chemistry, 2012-08, Vol.287 (36), p.30073-30083

2012

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Tempol Alters Urinary Extracellular Vesicle Lipid Content and Release While Reducing Blood Pressure during the Development of Salt-Sensitive Hypertension

Biomolecules (Basel, Switzerland), 2021-12, Vol.11 (12), p.1804

2021

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Transgenic Mice Overexpressing Human Alpha-1 Antitrypsin Exhibit Low Blood Pressure and Altered Epithelial Transport Mechanisms in the Inactive and Active Cycles

Frontiers in physiology, 2021-09, Vol.12, p.710313-710313

2021

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Epithelial Sodium Channel-α Mediates the Protective Effect of the TNF-Derived TIP Peptide in Pneumolysin-Induced Endothelial Barrier Dysfunction

Frontiers in immunology, 2017-07, Vol.8, p.842

2017

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The Pharmacological Inhibition of CaMKII Regulates Sodium Chloride Cotransporter Activity in mDCT15 Cells

Biology (Basel, Switzerland), 2021-12, Vol.10 (12), p.1335

2021

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Enrichment of Bioactive Lipids in Urinary Extracellular Vesicles and Evidence of Apoptosis in Kidneys of Hypertensive Diabetic Cathepsin B Knockout Mice after Streptozotocin Treatment

Biomedicines, 2024-05, Vol.12 (5), p.1038

2024

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WNK4 inhibition of ENaC is independent of Nedd4-2-mediated ENaC ubiquitination

American journal of physiology. Renal physiology, 2013-07, Vol.305 (1), p.F31-F41

2013

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Iron Inhibits the Translation and Activity of the Renal Epithelial Sodium Channel

Biology (Basel, Switzerland), 2022-01, Vol.11 (1), p.123

2022

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Decreased MARCKS Protein Expression in Kidney Cortex Membrane Fractions of Cathepsin B Knockout Mice Is Associated with Reduced Lysophosphatidylcholine and Protein Kinase C Activity

Biomedicines, 2023-05, Vol.11 (5), p.1489

2023

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Activation of multiple receptors stimulates extracellular vesicle release from trophoblast cells

Physiological reports, 2020-10, Vol.8 (20), p.e14592-n/a

2020

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402-P: ENaC Regulation in Mouse Cortical Collecting Duct Cells by EVs Released during the Inactive and Active Cycles of Salt-Loaded Hypertensive Diabetic db/db Mice

Diabetes (New York, N.Y.), 2023-06, Vol.72 (Supplement_1), p.1

2023

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Metformin Alleviates Diabetes-Associated Hypertension by Attenuating the Renal Epithelial Sodium Channel

Biomedicines, 2023-01, Vol.11 (2), p.305

2023

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Kidney tubular epithelial cell ferroptosis links glomerular injury to tubulointerstitial pathology in lupus nephritis

Clinical immunology (Orlando, Fla.), 2023-03, Vol.248, p.109213-109213, Article 109213

2023

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Lipidomic analysis of urinary exosomes from hereditary α‐tryptasemia patients and healthy volunteers

FASEB bioAdvances, 2019-10, Vol.1 (10), p.624-638

2019

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The C type natriuretic peptide receptor tethers AHNAK1 at the plasma membrane to potentiate arachidonic acid-induced calcium mobilization

American Journal of Physiology: Cell Physiology, 2009-11, Vol.297 (5), p.C1157-C1167

2009

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Extracellular Vesicles: Investigating the Pathophysiology of Diabetes-Associated Hypertension and Diabetic Nephropathy

Lipidomic and proteomic analysis of exosomes from mouse cortical collecting duct cells

Exosomal GAPDH from Proximal Tubule Cells Regulate ENaC Activity

Augmentation of Cathepsin Isoforms in Diabetic db/db Mouse Kidneys Is Associated with an Increase in Renal MARCKS Expression and Proteolysis

Lipid Profiles of Urinary Extracellular Vesicles Released during the Inactive and Active Phases of Aged Male Mice with Spontaneous Hypertension

Dapagliflozin Treatment Augments Bioactive Phosphatidylethanolamine Concentrations in Kidney Cortex Membrane Fractions of Hypertensive Diabetic db/db Mice and Alters the Density of Lipid Rafts in Mouse Proximal Tubule Cells

COVID-19 Plasma Extracellular Vesicles Increase the Density of Lipid Rafts in Human Small Airway Epithelial Cells

C Type Natriuretic Peptide Receptor Activation Inhibits Sodium Channel Activity in Human Aortic Endothelial Cells by Activating the Diacylglycerol-Protein Kinase C Pathway

ENaC is regulated by natriuretic peptide receptor-dependent cGMP signaling

Human Alpha-1 Antitrypsin Attenuates ENaC and MARCKS and Lowers Blood Pressure in Hypertensive Diabetic db/db Mice

Cathepsin B Is Secreted Apically from Xenopus 2F3 Cells and Cleaves the Epithelial Sodium Channel (ENaC) to Increase Its Activity

Tempol Alters Urinary Extracellular Vesicle Lipid Content and Release While Reducing Blood Pressure during the Development of Salt-Sensitive Hypertension

Transgenic Mice Overexpressing Human Alpha-1 Antitrypsin Exhibit Low Blood Pressure and Altered Epithelial Transport Mechanisms in the Inactive and Active Cycles

Epithelial Sodium Channel-α Mediates the Protective Effect of the TNF-Derived TIP Peptide in Pneumolysin-Induced Endothelial Barrier Dysfunction

The Pharmacological Inhibition of CaMKII Regulates Sodium Chloride Cotransporter Activity in mDCT15 Cells

Enrichment of Bioactive Lipids in Urinary Extracellular Vesicles and Evidence of Apoptosis in Kidneys of Hypertensive Diabetic Cathepsin B Knockout Mice after Streptozotocin Treatment

WNK4 inhibition of ENaC is independent of Nedd4-2-mediated ENaC ubiquitination

Iron Inhibits the Translation and Activity of the Renal Epithelial Sodium Channel

Decreased MARCKS Protein Expression in Kidney Cortex Membrane Fractions of Cathepsin B Knockout Mice Is Associated with Reduced Lysophosphatidylcholine and Protein Kinase C Activity

Activation of multiple receptors stimulates extracellular vesicle release from trophoblast cells

402-P: ENaC Regulation in Mouse Cortical Collecting Duct Cells by EVs Released during the Inactive and Active Cycles of Salt-Loaded Hypertensive Diabetic db/db Mice

Metformin Alleviates Diabetes-Associated Hypertension by Attenuating the Renal Epithelial Sodium Channel

Kidney tubular epithelial cell ferroptosis links glomerular injury to tubulointerstitial pathology in lupus nephritis

Lipidomic analysis of urinary exosomes from hereditary α‐tryptasemia patients and healthy volunteers

The C type natriuretic peptide receptor tethers AHNAK1 at the plasma membrane to potentiate arachidonic acid-induced calcium mobilization

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