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Sodium–glucose cotransporter-2 inhibition and the potential for renal protection in diabetic nephropathy

Current opinion in nephrology and hypertension, 2015-01, Vol.24 (1), p.96-103

2015

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Characterisation of glomerular haemodynamic responses to SGLT2 inhibition in patients with type 1 diabetes and renal hyperfiltration

Diabetologia, 2014-12, Vol.57 (12), p.2599-2602

2014

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Glycosuria-mediated urinary uric acid excretion in patients with uncomplicated type 1 diabetes mellitus

American journal of physiology. Renal physiology, 2015-01, Vol.308 (2), p.F77-F83

2015

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Hyperfiltration, urinary albumin excretion, and ambulatory blood pressure in adolescents with Type 1 diabetes mellitus

American journal of physiology. Renal physiology, 2018-04, Vol.314 (4), p.F667-F674

2018

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The Gomez' equations and renal hemodynamic function in kidney disease research

American journal of physiology. Renal physiology, 2016-11, Vol.311 (5), p.F967-F975

2016

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SGLT2 Inhibition in Patients With Type 2 Diabetes Mellitus Post-Nephrectomy: A Single-Center Case Series

Canadian journal of kidney health and disease, 2021-12, Vol.8, p.20543581211065528-20543581211065528

2021

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The neurorepellent, Slit2, prevents macrophage lipid loading by inhibiting CD36-dependent binding and internalization of oxidized low-density lipoprotein

Scientific reports, 2021-02, Vol.11 (1), p.3614-3614, Article 3614

2021

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Influence of sex on hyperfiltration in patients with uncomplicated type 1 diabetes

American journal of physiology. Renal physiology, 2017-04, Vol.312 (4), p.F599-F606

2017

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The ubiquitin-activating enzyme E1 as a therapeutic target for the treatment of leukemia and multiple myeloma

Blood, 2010-03, Vol.115 (11), p.2251-2259

2010

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The antiparasitic agent ivermectin induces chloride-dependent membrane hyperpolarization and cell death in leukemia cells

Blood, 2010-11, Vol.116 (18), p.3593-3603

2010

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Erratum to: Characterisation of glomerular haemodynamic responses to SGLT2 inhibition in patients with type 1 diabetes and renal hyperfiltration

Diabetologia, 2017-06, Vol.60 (6), p.1159-1160

2017

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Therapeutic potential of mitochondrial translation inhibition for treatment of acute myeloid leukemia

Expert review of hematology, 2012-04, Vol.5 (2), p.117-119

2012

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Bacteria Challenge in Smoke-exposed Mice Exacerbates Inflammation and Skews the Inflammatory Profile

American journal of respiratory and critical care medicine, 2009-04, Vol.179 (8), p.666-675

2009

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Improving Sexual Function in People With Chronic Kidney Disease: A Narrative Review of an Unmet Need in Nephrology Research

Canadian Journal of Kidney Health and Disease, 2020-01, Vol.7, p.2054358120952202-2054358120952202

2020

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Metabolic adaptation to chronic inhibition of mitochondrial protein synthesis in acute myeloid leukemia cells

PloS one, 2013-03, Vol.8 (3), p.e58367

2013

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AML cells have low spare reserve capacity in their respiratory chain that renders them susceptible to oxidative metabolic stress

Blood, 2015-03, Vol.125 (13), p.2120-2130

2015

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MITOCHONDRIAL METABOLISM IS PRESERVED FOLLOWING NORMOTHERMIC EX-VIVO KIDNEY PERFUSION OF GRAFTS PROCURED FOLLOWING CARDIAC DEATH

Transplantation, 2020-09, Vol.104 (S3), p.S249-S249

2020

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Lysosomal disruption preferentially targets acute myeloid leukemia cells and progenitors

The Journal of clinical investigation, 2013-01, Vol.123 (1), p.315-328

2013

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Cigarette Smoke Exposure Attenuates Cytokine Production by Mouse Alveolar Macrophages

American journal of respiratory cell and molecular biology, 2008-02, Vol.38 (2), p.218-226

2008

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Altered expression of disintegrin metalloproteinases and their inhibitor in human dilated cardiomyopathy

Circulation (New York, N.Y.), 2006-01, Vol.113 (2), p.238-245

2006

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Abstract 1123: AML cells have low reserve capacity in their respiratory chain complexes leading to increased sensitivity to palmitate-induced cell death

Cancer research (Chicago, Ill.), 2012-04, Vol.72 (8_Supplement), p.1123-1123

2012

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AML Cells Have Increased Mitochondrial Mass but Less Reserve in Their Respiratory Chain Complexes Leading to Heightened Sensitivity to Inhibition of Mitochondrial Protein Translation

Blood, 2011-11, Vol.118 (21), p.3585-3585

2011

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Chronic Inhibition of Mitochondrial Translation Alters Metabolism and Stemness in AML

Blood, 2012-11, Vol.120 (21), p.1364-1364

2012

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Inhibition of mitochondrial translation as a therpeutic strategy for acute myeloid leukemia

Skrtic, Marko

2012

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Sodium–glucose cotransporter-2 inhibition and the potential for renal protection in diabetic nephropathy

Characterisation of glomerular haemodynamic responses to SGLT2 inhibition in patients with type 1 diabetes and renal hyperfiltration

Glycosuria-mediated urinary uric acid excretion in patients with uncomplicated type 1 diabetes mellitus

Hyperfiltration, urinary albumin excretion, and ambulatory blood pressure in adolescents with Type 1 diabetes mellitus

The Gomez' equations and renal hemodynamic function in kidney disease research

Inhibition of Mitochondrial Translation as a Therapeutic Strategy for Human Acute Myeloid Leukemia

SGLT2 Inhibition in Patients With Type 2 Diabetes Mellitus Post-Nephrectomy: A Single-Center Case Series

The neurorepellent, Slit2, prevents macrophage lipid loading by inhibiting CD36-dependent binding and internalization of oxidized low-density lipoprotein

Influence of sex on hyperfiltration in patients with uncomplicated type 1 diabetes

The ubiquitin-activating enzyme E1 as a therapeutic target for the treatment of leukemia and multiple myeloma

The antiparasitic agent ivermectin induces chloride-dependent membrane hyperpolarization and cell death in leukemia cells

Erratum to: Characterisation of glomerular haemodynamic responses to SGLT2 inhibition in patients with type 1 diabetes and renal hyperfiltration

Therapeutic potential of mitochondrial translation inhibition for treatment of acute myeloid leukemia

Bacteria Challenge in Smoke-exposed Mice Exacerbates Inflammation and Skews the Inflammatory Profile

Improving Sexual Function in People With Chronic Kidney Disease: A Narrative Review of an Unmet Need in Nephrology Research

Metabolic adaptation to chronic inhibition of mitochondrial protein synthesis in acute myeloid leukemia cells

AML cells have low spare reserve capacity in their respiratory chain that renders them susceptible to oxidative metabolic stress

MITOCHONDRIAL METABOLISM IS PRESERVED FOLLOWING NORMOTHERMIC EX-VIVO KIDNEY PERFUSION OF GRAFTS PROCURED FOLLOWING CARDIAC DEATH

Lysosomal disruption preferentially targets acute myeloid leukemia cells and progenitors

Cigarette Smoke Exposure Attenuates Cytokine Production by Mouse Alveolar Macrophages

Altered expression of disintegrin metalloproteinases and their inhibitor in human dilated cardiomyopathy

Abstract 1123: AML cells have low reserve capacity in their respiratory chain complexes leading to increased sensitivity to palmitate-induced cell death

AML Cells Have Increased Mitochondrial Mass but Less Reserve in Their Respiratory Chain Complexes Leading to Heightened Sensitivity to Inhibition of Mitochondrial Protein Translation

Chronic Inhibition of Mitochondrial Translation Alters Metabolism and Stemness in AML

Inhibition of mitochondrial translation as a therpeutic strategy for acute myeloid leukemia

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