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Promoting a more diverse and inclusive research workforce through the research scholars program

BMC medical education, 2024-01, Vol.24 (1), p.98-98, Article 98

2024

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Therapeutic Assessment of Combination Therapy with a Neprilysin Inhibitor and Angiotensin Type 1 Receptor Antagonist on Angiotensin II-Induced Atherosclerosis, Abdominal Aortic Aneurysms, and Hypertension

The Journal of pharmacology and experimental therapeutics, 2021-06, Vol.377 (3), p.326

2021

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Adipocyte-Derived Serum Amyloid A Promotes Angiotensin II-Induced Abdominal Aortic Aneurysms in Obese C57BL/6J Mice

Arteriosclerosis, thrombosis, and vascular biology, 2022-05, Vol.42 (5), p.632

2022

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Mechanisms of abdominal aortic aneurysm formation

Current atherosclerosis reports, 2002-05, Vol.4 (3), p.222-227

2002

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Hypercholesterolemia stimulates angiotensin peptide synthesis and contributes to atherosclerosis through the AT1A receptor

Circulation (New York, N.Y.), 2004-12, Vol.110 (25), p.3849-3857

2004

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Subcutaneous Angiotensin II Infusion using Osmotic Pumps Induces Aortic Aneurysms in Mice

Journal of visualized experiments, 2015-09 (103)

2015

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Corrigendum to “Deficiency of receptor-associated protein attenuates angiotensin II-induced atherosclerosis in hypercholesterolemic mice without influencing abdominal aortic aneurysms” [Atherosclerosis 220 (2) 375–380]

Atherosclerosis, 2013-10, Vol.230 (2), p.300-300

2013

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AT1-receptor antagonism reverses the blood pressure elevation associated with diet-induced obesity

American journal of physiology. Regulatory, integrative and comparative physiology, 2005-07, Vol.289 (1), p.R181-R186

2005

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Abstract MP62: Sars-cov-2 Spike Protein Regulation Of Angiotensin Converting Enzyme 2 And Tissue Renin-angiotensin Systems: Influence Of Biologic Sex

Hypertension (Dallas, Tex. 1979), 2021-09, Vol.78 (Suppl_1)

2021

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Abstract P197: Increased Serotonin In Visceral Adipose Tissue May Contribute To Stimulate Sensory Neurons Mediating Obesity Hypertension In Mice Exposed To Early Life Stress

Hypertension (Dallas, Tex. 1979), 2021-09, Vol.78 (Suppl_1)

2021

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Abstract 249: Male Mice with XX Sex Chromosome Complement Exhibit Reduced Angiotensin II-Induced Hypertension and Atherosclerosis

Hypertension (Dallas, Tex. 1979), 2014-09, Vol.64 (suppl_1)

2014

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Abstract 100: Mas Receptor Expression in Bone Marrow Stem/Progenitor Cells is Both Required and Sufficient for the Mobilization of Cells in Response to Ischemic Injury

Hypertension (Dallas, Tex. 1979), 2019-09, Vol.74 (Suppl_1)

2019

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Abstract 16557: Hemodynamic Stress Activates a Comprehensive Adaptive Program in Murine Aortic Cells That Protects the Aortic Wall and Maintains Aortic Homeostasis

Circulation (New York, N.Y.), 2020-11, Vol.142 (Suppl_3 Suppl 3), p.A16557-A16557

2020

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Abstract 63: ACE2 Deficiency Reduces Insulin Content of Isolated Pancreatic Islets and Plasma Insulin Concentrations Post-Glucose Challenge in Obese C57BL/6 Mice

Hypertension (Dallas, Tex. 1979), 2013-09, Vol.62 (suppl_1)

2013

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Abstract 15325: Dynamic Change From Mitochondrial Compensation to Mitochondrial Failure During Aortic Disease Progression and Dissection Formation

Circulation (New York, N.Y.), 2022-11, Vol.146 (Suppl_1)

2022

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Abstract 15539: Single-cell Analysis of Aortic Tissues From Patients With Marfan Syndrome Reveals Changes in Smooth Muscle Cell Differentiation

Circulation (New York, N.Y.), 2020-11, Vol.142 (Suppl_3 Suppl 3), p.A15539-A15539

2020

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Abstract 15530: Single-cell Analysis in Aortic Aneurysmal Tissue From Patients With Marfan Syndrome Reveals Increased Tgf-beta Production but Downregulation of Downstream Canonical Tgf-beta Signaling Pathways

Circulation (New York, N.Y.), 2020-11, Vol.142 (Suppl_3 Suppl 3), p.A15530-A15530

2020

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Deletion of p47phox attenuates angiotensin II-induced abdominal aortic aneurysm formation in apolipoprotein E-deficient mice

Circulation (New York, N.Y.), 2006-08, Vol.114 (5), p.404-413

2006

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Fat cell metabolism: insulin, fatty acids, and renin

Current hypertension reports, 2000-04, Vol.2 (2), p.132-138

Cassis, L A

2000

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Abstract 013: Genetic Ablation of Mas Receptor Impairs Mobilization of Bone Marrow Progenitor Cells

Hypertension (Dallas, Tex. 1979), 2016-09, Vol.68 (suppl_1)

2016

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Administration of 17[Beta]-estradiol to ovariectomized obese female mice

American journal of physiology: endocrinology and metabolism, 2015-06, Vol.308 (12), p.E1066

2015

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TGF-[beta] Neutralization Enhances AngII-Induced Aortic Rupture and Aneurysm in Both Thoracic and Abdominal Regions

PloS one, 2016-04, Vol.11 (4)

2016

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Correction: Corrigendum: Structure and functions of angiotensinogen

Hypertension research, 2016-11, Vol.39 (11), p.827-827

2016

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Corrigendum: Structure and functions of angiotensinogen

Hypertension research, 2016-11, Vol.39 (11), p.827-827

2016

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The renin-angiotensin system: a target of and contributor to dyslipidemias, altered glucose homeostasis, and hypertension of the metabolic syndrome

American journal of physiology. Heart and circulatory physiology, 2012, Vol.71 (3)

2012

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Promoting a more diverse and inclusive research workforce through the research scholars program

Therapeutic Assessment of Combination Therapy with a Neprilysin Inhibitor and Angiotensin Type 1 Receptor Antagonist on Angiotensin II-Induced Atherosclerosis, Abdominal Aortic Aneurysms, and Hypertension

Adipocyte-Derived Serum Amyloid A Promotes Angiotensin II-Induced Abdominal Aortic Aneurysms in Obese C57BL/6J Mice

Mechanisms of abdominal aortic aneurysm formation

Hypercholesterolemia stimulates angiotensin peptide synthesis and contributes to atherosclerosis through the AT1A receptor

Subcutaneous Angiotensin II Infusion using Osmotic Pumps Induces Aortic Aneurysms in Mice

Corrigendum to “Deficiency of receptor-associated protein attenuates angiotensin II-induced atherosclerosis in hypercholesterolemic mice without influencing abdominal aortic aneurysms” [Atherosclerosis 220 (2) 375–380]

AT1-receptor antagonism reverses the blood pressure elevation associated with diet-induced obesity

Abstract MP62: Sars-cov-2 Spike Protein Regulation Of Angiotensin Converting Enzyme 2 And Tissue Renin-angiotensin Systems: Influence Of Biologic Sex

Abstract P197: Increased Serotonin In Visceral Adipose Tissue May Contribute To Stimulate Sensory Neurons Mediating Obesity Hypertension In Mice Exposed To Early Life Stress

Abstract 249: Male Mice with XX Sex Chromosome Complement Exhibit Reduced Angiotensin II-Induced Hypertension and Atherosclerosis

Abstract 100: Mas Receptor Expression in Bone Marrow Stem/Progenitor Cells is Both Required and Sufficient for the Mobilization of Cells in Response to Ischemic Injury

Abstract 16557: Hemodynamic Stress Activates a Comprehensive Adaptive Program in Murine Aortic Cells That Protects the Aortic Wall and Maintains Aortic Homeostasis

Abstract 63: ACE2 Deficiency Reduces Insulin Content of Isolated Pancreatic Islets and Plasma Insulin Concentrations Post-Glucose Challenge in Obese C57BL/6 Mice

Abstract 15325: Dynamic Change From Mitochondrial Compensation to Mitochondrial Failure During Aortic Disease Progression and Dissection Formation

Abstract 15539: Single-cell Analysis of Aortic Tissues From Patients With Marfan Syndrome Reveals Changes in Smooth Muscle Cell Differentiation

Abstract 15530: Single-cell Analysis in Aortic Aneurysmal Tissue From Patients With Marfan Syndrome Reveals Increased Tgf-beta Production but Downregulation of Downstream Canonical Tgf-beta Signaling Pathways

Deletion of p47phox attenuates angiotensin II-induced abdominal aortic aneurysm formation in apolipoprotein E-deficient mice

Fat cell metabolism: insulin, fatty acids, and renin

Abstract 013: Genetic Ablation of Mas Receptor Impairs Mobilization of Bone Marrow Progenitor Cells

Administration of 17[Beta]-estradiol to ovariectomized obese female mice

TGF-[beta] Neutralization Enhances AngII-Induced Aortic Rupture and Aneurysm in Both Thoracic and Abdominal Regions

Correction: Corrigendum: Structure and functions of angiotensinogen

Corrigendum: Structure and functions of angiotensinogen

The renin-angiotensin system: a target of and contributor to dyslipidemias, altered glucose homeostasis, and hypertension of the metabolic syndrome

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