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Genomic Imprinting and Physiological Processes in Mammals

Cell, 2019-02, Vol.176 (5), p.952-965

2019

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Neonatal oxytocin gives the tempo of social and feeding behaviors

Frontiers in molecular neuroscience, 2022-12, Vol.15, p.1071719-1071719

2022

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Oxytocin-based therapies for treatment of Prader-Willi and Schaaf-Yang syndromes: evidence, disappointments, and future research strategies

Translational psychiatry, 2022-08, Vol.12 (1), p.318-318, Article 318

2022

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Modulation of the thermosensory system by oxytocin

Frontiers in molecular neuroscience, 2023-01, Vol.15, p.1075305-1075305

2023

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The Use of Oxytocin to Improve Feeding and Social Skills in Infants With Prader-Willi Syndrome

Pediatrics (Evanston), 2017-02, Vol.139 (2)

2017

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Early life oxytocin treatment improves thermo-sensory reactivity and maternal behavior in neonates lacking the autism-associated gene Magel2

Neuropsychopharmacology, 2022-10, Vol.47 (11), p.1901-1912

2022

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Necdin shapes serotonergic development and SERT activity modulating breathing in a mouse model for Prader-Willi syndrome

eLife, 2017-10, Vol.6

2017

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Stochastic loss of silencing of the imprinted Ndn/NDN allele, in a mouse model and humans with prader-willi syndrome, has functional consequences

PLoS genetics, 2013-09, Vol.9 (9), p.e1003752-e1003752

2013

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Oxytocin administration in neonates shapes hippocampal circuitry and restores social behavior in a mouse model of autism

Molecular psychiatry, 2021-12, Vol.26 (12), p.7582-7595

2021

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Vasopressin: An effective treatment for autism spectrum disorders?

M.S. Médecine sciences, 2021-10, Vol.37 (10), p.848

2021

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Necdin Plays a Role in the Serotonergic Modulation of the Mouse Respiratory Network: Implication for Prader-Willi Syndrome

The Journal of neuroscience, 2008-02, Vol.28 (7), p.1745-1755

2008

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The impact of oxytocin on neurite outgrowth and synaptic proteins in Magel2‐deficient mice

Developmental biology, 2021-05, Vol.81 (4), p.366-388

2021

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A single postnatal injection of oxytocin rescues the lethal feeding behaviour in mouse newborns deficient for the imprinted Magel2 gene

Human molecular genetics, 2010-12, Vol.19 (24), p.4895-4905

2010

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An Early Postnatal Oxytocin Treatment Prevents Social and Learning Deficits in Adult Mice Deficient for Magel2 , a Gene Involved in Prader-Willi Syndrome and Autism

Biological psychiatry (1969), 2015-07, Vol.78 (2), p.85-94

2015

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Wired for eating: how is an active feeding circuitry established in the postnatal brain?

Current opinion in neurobiology, 2018-10, Vol.52, p.165-171

2018

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Functional consequences of necdin nucleocytoplasmic localization

PloS one, 2012-03, Vol.7 (3), p.e33786

2012

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Breathing deficits of the Prader-Willi syndrome

Respiratory physiology & neurobiology, 2009-08, Vol.168 (1), p.119-124

2009

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Oxytocin receptors in the Magel2 mouse model of autism: Specific region, age, sex and oxytocin treatment effects

Frontiers in neuroscience, 2023-03, Vol.17, p.1026939-1026939

2023

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Correction of vasopressin deficit in the lateral septum ameliorates social deficits of mouse autism model

The Journal of clinical investigation, 2021-01, Vol.131 (2), p.1-14

2021

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Colocalization of Oxtr with Prader-Willi syndrome transcripts in the trigeminal ganglion of neonatal mice

Human molecular genetics, 2020-07, Vol.29 (12), p.2065-2075

2020

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Sensory defects in Necdin deficient mice result from a loss of sensory neurons correlated within an increase of developmental programmed cell death

BMC developmental biology, 2006-11, Vol.6 (1), p.56-56, Article 56

2006

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Ontogenesis of oxytocin pathways in the mammalian brain: late maturation and psychosocial disorders

Frontiers in neuroanatomy, 2015-01, Vol.8, p.164-164

2015

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ADAR2-mediated editing of RNA substrates in the nucleolus is inhibited by C/D small nucleolar RNAs

The Journal of cell biology, 2005-06, Vol.169 (5), p.745-753

2005

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Inactivation of Socs3 in the hypothalamus enhances the hindbrain response to endogenous satiety signals via oxytocin signaling

The Journal of neuroscience, 2012-11, Vol.32 (48), p.17097-17107

2012

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Tubacin prevents neuronal migration defects and epileptic activity caused by rat Srpx2 silencing in utero

Brain (London, England : 1878), 2013-08, Vol.136 (Pt 8), p.2457-2473

2013

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Genomic Imprinting and Physiological Processes in Mammals

Neonatal oxytocin gives the tempo of social and feeding behaviors

Oxytocin-based therapies for treatment of Prader-Willi and Schaaf-Yang syndromes: evidence, disappointments, and future research strategies

Modulation of the thermosensory system by oxytocin

The Use of Oxytocin to Improve Feeding and Social Skills in Infants With Prader-Willi Syndrome

Early life oxytocin treatment improves thermo-sensory reactivity and maternal behavior in neonates lacking the autism-associated gene Magel2

Necdin shapes serotonergic development and SERT activity modulating breathing in a mouse model for Prader-Willi syndrome

Stochastic loss of silencing of the imprinted Ndn/NDN allele, in a mouse model and humans with prader-willi syndrome, has functional consequences

Oxytocin administration in neonates shapes hippocampal circuitry and restores social behavior in a mouse model of autism

Vasopressin: An effective treatment for autism spectrum disorders?

Necdin Plays a Role in the Serotonergic Modulation of the Mouse Respiratory Network: Implication for Prader-Willi Syndrome

The impact of oxytocin on neurite outgrowth and synaptic proteins in Magel2‐deficient mice

A single postnatal injection of oxytocin rescues the lethal feeding behaviour in mouse newborns deficient for the imprinted Magel2 gene

An Early Postnatal Oxytocin Treatment Prevents Social and Learning Deficits in Adult Mice Deficient for Magel2 , a Gene Involved in Prader-Willi Syndrome and Autism

Wired for eating: how is an active feeding circuitry established in the postnatal brain?

Functional consequences of necdin nucleocytoplasmic localization

Breathing deficits of the Prader-Willi syndrome

Oxytocin receptors in the Magel2 mouse model of autism: Specific region, age, sex and oxytocin treatment effects

Correction of vasopressin deficit in the lateral septum ameliorates social deficits of mouse autism model

Colocalization of Oxtr with Prader-Willi syndrome transcripts in the trigeminal ganglion of neonatal mice

Sensory defects in Necdin deficient mice result from a loss of sensory neurons correlated within an increase of developmental programmed cell death

Ontogenesis of oxytocin pathways in the mammalian brain: late maturation and psychosocial disorders

ADAR2-mediated editing of RNA substrates in the nucleolus is inhibited by C/D small nucleolar RNAs

Inactivation of Socs3 in the hypothalamus enhances the hindbrain response to endogenous satiety signals via oxytocin signaling

Tubacin prevents neuronal migration defects and epileptic activity caused by rat Srpx2 silencing in utero

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