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VDAC1 in the diseased myocardium and the effect of VDAC1-interacting compound on atrial fibrosis induced by hyperaldosteronism

Scientific reports, 2020-12, Vol.10 (1), p.22101-22101, Article 22101

2020

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VDAC1 functions in Ca2+ homeostasis and cell life and death in health and disease

Cell calcium (Edinburgh), 2018-01, Vol.69, p.81-100

2018

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Voltage-dependent Anion Channel 1-based Peptides Interact with Bcl-2 to Prevent Antiapoptotic Activity

The Journal of biological chemistry, 2010-02, Vol.285 (9), p.6053-6062

2010

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Oligomerization of the Mitochondrial Protein Voltage-Dependent Anion Channel Is Coupled to the Induction of Apoptosis

Molecular and Cellular Biology, 2010-12, Vol.30 (24), p.5698-5709

2010

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Targeting Liver Cancer and Associated Pathologies in Mice with a Mitochondrial VDAC1-Based Peptide

Neoplasia (New York, N.Y.), 2018-06, Vol.20 (6), p.594-609

2018

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VDAC1-interacting anion transport inhibitors inhibit VDAC1 oligomerization and apoptosis

Biochimica et biophysica acta, 2016-07, Vol.1863 (7), p.1612-1623

2016

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VDAC oligomers form mitochondrial pores to release mtDNA fragments and promote lupus-like disease

Science (American Association for the Advancement of Science), 2019-12, Vol.366 (6472), p.1531-1536

2019

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The role of calcium in VDAC1 oligomerization and mitochondria-mediated apoptosis

Biochimica et biophysica acta, 2013-07, Vol.1833 (7), p.1745-1754

2013

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NCLX is an essential component of mitochondrial Na⁺/Ca²⁺ exchange

Proceedings of the National Academy of Sciences - PNAS, 2010-01, Vol.107 (1), p.436-441

2010

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Misfolded Mutant SOD1 Directly Inhibits VDAC1 Conductance in a Mouse Model of Inherited ALS

Neuron (Cambridge, Mass.), 2010-08, Vol.67 (4), p.575-587

2010

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Novel Compounds Targeting the Mitochondrial Protein VDAC1 Inhibit Apoptosis and Protect against Mitochondrial Dysfunction

The Journal of biological chemistry, 2016-11, Vol.291 (48), p.24986-25003

2016

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Voltage-dependent Anion Channel 1-based Peptides Interact with Hexokinase to Prevent Its Anti-apoptotic Activity

The Journal of biological chemistry, 2009-02, Vol.284 (6), p.3946-3955

2009

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Uncovering the role of VDAC in the regulation of cell life and death

Journal of bioenergetics and biomembranes, 2008-06, Vol.40 (3), p.183-191

2008

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Mitochondrial VDAC, the Na+/Ca2+ Exchanger, and the Ca2+ Uniporter in Ca2+ Dynamics and Signaling

Membrane Dynamics and Calcium Signaling, p.323-347

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Apoptosis is regulated by the VDAC1 N-terminal region and by VDAC oligomerization: release of cytochrome c, AIF and Smac/Diablo

Biochimica et biophysica acta, 2010-06, Vol.1797 (6-7), p.1281-1291

2010

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The Expression Level of the Voltage-Dependent Anion Channel Controls Life and Death of the Cell

Proceedings of the National Academy of Sciences - PNAS, 2006-04, Vol.103 (15), p.5787-5792

2006

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Key regions of VDAC1 functioning in apoptosis induction and regulation by hexokinase

Biochimica et biophysica acta, 2009-05, Vol.1787 (5), p.421-430

2009

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Mitochondrial VDAC1 Silencing Leads to Metabolic Rewiring and the Reprogramming of Tumour Cells into Advanced Differentiated States

Cancers, 2018-12, Vol.10 (12), p.499

2018

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Silencing VDAC1 Expression by siRNA Inhibits Cancer Cell Proliferation and Tumor Growth In Vivo

Molecular therapy. Nucleic acids, 2014, Vol.3 (4), p.e159-e159, Article e159

2014

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Non-apoptotic activity of the mitochondrial protein SMAC/Diablo in lung cancer: Novel target to disrupt survival, inflammation, and immunosuppression

Frontiers in oncology, 2022-09, Vol.12, p.992260-992260

2022

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Overexpression of the mitochondrial anti-viral signaling protein, MAVS, in cancers is associated with cell survival and inflammation

Molecular therapy. Nucleic acids, 2023-09, Vol.33, p.713-732

2023

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VDAC1 N-terminus is essential both for apoptosis and the protective effect of anti-apoptotic proteins

Journal of cell science, 2009-06, Vol.122 (11), p.1906-1916

2009

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Editorial: Altered Expression of Proteins in Cancer: Function and Potential Therapeutic Targets

Frontiers in oncology, 2022-06, Vol.12, p.949139-949139

2022

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Oligomerization of the Mitochondrial Protein VDAC1: From Structure to Function and Cancer Therapy

Progress in Molecular Biology and Translational Science, 2013, Vol.117, p.303-334

2013

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Calcium binding and translocation by the voltage-dependent anion channel: a possible regulatory mechanism in mitochondrial function

Biochemical journal, 2001-08, Vol.358 (Pt 1), p.147-155

2001

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VDAC1 in the diseased myocardium and the effect of VDAC1-interacting compound on atrial fibrosis induced by hyperaldosteronism

VDAC1 functions in Ca2+ homeostasis and cell life and death in health and disease

Voltage-dependent Anion Channel 1-based Peptides Interact with Bcl-2 to Prevent Antiapoptotic Activity

Oligomerization of the Mitochondrial Protein Voltage-Dependent Anion Channel Is Coupled to the Induction of Apoptosis

Targeting Liver Cancer and Associated Pathologies in Mice with a Mitochondrial VDAC1-Based Peptide

VDAC1-interacting anion transport inhibitors inhibit VDAC1 oligomerization and apoptosis

VDAC oligomers form mitochondrial pores to release mtDNA fragments and promote lupus-like disease

The role of calcium in VDAC1 oligomerization and mitochondria-mediated apoptosis

NCLX is an essential component of mitochondrial Na⁺/Ca²⁺ exchange

Misfolded Mutant SOD1 Directly Inhibits VDAC1 Conductance in a Mouse Model of Inherited ALS

Novel Compounds Targeting the Mitochondrial Protein VDAC1 Inhibit Apoptosis and Protect against Mitochondrial Dysfunction

Voltage-dependent Anion Channel 1-based Peptides Interact with Hexokinase to Prevent Its Anti-apoptotic Activity

Uncovering the role of VDAC in the regulation of cell life and death

Mitochondrial VDAC, the Na+/Ca2+ Exchanger, and the Ca2+ Uniporter in Ca2+ Dynamics and Signaling

Apoptosis is regulated by the VDAC1 N-terminal region and by VDAC oligomerization: release of cytochrome c, AIF and Smac/Diablo

The Expression Level of the Voltage-Dependent Anion Channel Controls Life and Death of the Cell

Key regions of VDAC1 functioning in apoptosis induction and regulation by hexokinase

Mitochondrial VDAC1 Silencing Leads to Metabolic Rewiring and the Reprogramming of Tumour Cells into Advanced Differentiated States

Silencing VDAC1 Expression by siRNA Inhibits Cancer Cell Proliferation and Tumor Growth In Vivo

Non-apoptotic activity of the mitochondrial protein SMAC/Diablo in lung cancer: Novel target to disrupt survival, inflammation, and immunosuppression

Overexpression of the mitochondrial anti-viral signaling protein, MAVS, in cancers is associated with cell survival and inflammation

VDAC1 N-terminus is essential both for apoptosis and the protective effect of anti-apoptotic proteins

Editorial: Altered Expression of Proteins in Cancer: Function and Potential Therapeutic Targets

Oligomerization of the Mitochondrial Protein VDAC1: From Structure to Function and Cancer Therapy

Calcium binding and translocation by the voltage-dependent anion channel: a possible regulatory mechanism in mitochondrial function

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