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A Chemoproteomic Approach to Query the Degradable Kinome Using a Multi-kinase Degrader

Cell chemical biology, 2018-01, Vol.25 (1), p.88-99.e6

2018

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Functional screening identifies CRLF2 in precursor B-cell acute lymphoblastic leukemia

Proceedings of the National Academy of Sciences - PNAS, 2010-01, Vol.107 (1), p.252-257

2010

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Inhibition of USP10 induces degradation of oncogenic FLT3

Nature chemical biology, 2017-12, Vol.13 (12), p.1207-1215

2017

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Anti‐SARS‐CoV‐2 activity of targeted kinase inhibitors: Repurposing clinically available drugs for COVID‐19 therapy

Journal of medical virology, 2023-01, Vol.95 (1), p.e28157-n/a

2023

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Characterization of AMN107, a selective inhibitor of native and mutant Bcr-Abl

Cancer cell, 2005-02, Vol.7 (2), p.129-141

2005

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Chimeric Antigen Receptor T Cell Therapy in Acute Myeloid Leukemia: Trials and Tribulations

Hematology reports, 2023-12, Vol.15 (4), p.608-626

2023

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Discovery of a Potent and Selective DDR1 Receptor Tyrosine Kinase Inhibitor

ACS chemical biology, 2013-10, Vol.8 (10), p.2145-2150

2013

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Reversible resistance induced by FLT3 inhibition: a novel resistance mechanism in mutant FLT3-expressing cells

PloS one, 2011-09, Vol.6 (9), p.e25351

2011

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Synergy between BRD9- and IKZF3-Targeting as a Therapeutic Strategy for Multiple Myeloma

Cancers, 2024-04, Vol.16 (7), p.1319

2024

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Inhibition of Wild-Type p53-Expressing AML by the Novel Small Molecule HDM2 Inhibitor CGM097

Molecular cancer therapeutics, 2015-10, Vol.14 (10), p.2249-2259

2015

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Repurposing clinically available drugs and therapies for pathogenic targets to combat SARS‐CoV‐2

MedComm (2020), 2023-06, Vol.4 (3), p.e254-n/a

2023

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Gene expression profiling analysis of CRTC1-MAML2 fusion oncogene-induced transcriptional program in human mucoepidermoid carcinoma cells

BMC cancer, 2015-10, Vol.15 (1), p.803-803, Article 803

2015

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BRD9 degraders as chemosensitizers in acute leukemia and multiple myeloma

Blood cancer journal (New York), 2022-07, Vol.12 (7), p.110-110, Article 110

2022

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The CRTC1-MAML2 fusion is the major oncogenic driver in mucoepidermoid carcinoma

JCI insight, 2021-04, Vol.6 (7)

2021

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Inhibition of mutant FLT3 receptors in leukemia cells by the small molecule tyrosine kinase inhibitor PKC412

Cancer cell, 2002-06, Vol.1 (5), p.433-443

2002

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FLT3 inhibition and mechanisms of drug resistance in mutant FLT3-positive AML

Drug resistance updates, 2009-06, Vol.12 (3), p.81-89

2009

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Selective Akt inhibitors synergize with tyrosine kinase inhibitors and effectively override stroma-associated cytoprotection of mutant FLT3-positive AML cells

PloS one, 2013-02, Vol.8 (2), p.e56473

2013

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Identification of a Family of Mastermind-Like Transcriptional Coactivators for Mammalian Notch Receptors

Molecular and Cellular Biology, 2002-11, Vol.22 (21), p.7688-7700

2002

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Notch1 activation enhances proliferation via activation of cdc2 and delays differentiation of myeloid progenitors

Leukemia research, 2018-09, Vol.72, p.34-44

2018

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STI571 inactivation of the gastrointestinal stromal tumor c-KIT oncoprotein : biological and clinical implications

Oncogene, 2001-08, Vol.20 (36), p.5054-5058

2001

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Inhibitors of the Transcription Factor STAT3 Decrease Growth and Induce Immune Response Genes in Models of Malignant Pleural Mesothelioma (MPM)

Cancers, 2020-12, Vol.13 (1), p.7

2020

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Combination of Rapamycin and Protein Tyrosine Kinase (PTK) Inhibitors for the Treatment of Leukemias Caused by Oncogenic PTKs

Proceedings of the National Academy of Sciences - PNAS, 2004-03, Vol.101 (9), p.3130-3135

2004

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Growth Suppression of Pre-T Acute Lymphoblastic Leukemia Cells by Inhibition of Notch Signaling

Molecular and Cellular Biology, 2003-01, Vol.23 (2), p.655-664

2003

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Repurposing of Kinase Inhibitors for Treatment of COVID-19

Pharmaceutical research, 2020-09, Vol.37 (9), p.167, Article 167

2020

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MAML1, a human homologue of Drosophila Mastermind, is a transcriptional co-activator for NOTCH receptors

Nature genetics, 2000-12, Vol.26 (4), p.484-489

2000

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A Chemoproteomic Approach to Query the Degradable Kinome Using a Multi-kinase Degrader

Functional screening identifies CRLF2 in precursor B-cell acute lymphoblastic leukemia

Inhibition of USP10 induces degradation of oncogenic FLT3

Anti‐SARS‐CoV‐2 activity of targeted kinase inhibitors: Repurposing clinically available drugs for COVID‐19 therapy

Characterization of AMN107, a selective inhibitor of native and mutant Bcr-Abl

Chimeric Antigen Receptor T Cell Therapy in Acute Myeloid Leukemia: Trials and Tribulations

Discovery of a Potent and Selective DDR1 Receptor Tyrosine Kinase Inhibitor

Reversible resistance induced by FLT3 inhibition: a novel resistance mechanism in mutant FLT3-expressing cells

Synergy between BRD9- and IKZF3-Targeting as a Therapeutic Strategy for Multiple Myeloma

Inhibition of Wild-Type p53-Expressing AML by the Novel Small Molecule HDM2 Inhibitor CGM097

Repurposing clinically available drugs and therapies for pathogenic targets to combat SARS‐CoV‐2

Gene expression profiling analysis of CRTC1-MAML2 fusion oncogene-induced transcriptional program in human mucoepidermoid carcinoma cells

BRD9 degraders as chemosensitizers in acute leukemia and multiple myeloma

The CRTC1-MAML2 fusion is the major oncogenic driver in mucoepidermoid carcinoma

Inhibition of mutant FLT3 receptors in leukemia cells by the small molecule tyrosine kinase inhibitor PKC412

FLT3 inhibition and mechanisms of drug resistance in mutant FLT3-positive AML

Selective Akt inhibitors synergize with tyrosine kinase inhibitors and effectively override stroma-associated cytoprotection of mutant FLT3-positive AML cells

Identification of a Family of Mastermind-Like Transcriptional Coactivators for Mammalian Notch Receptors

Notch1 activation enhances proliferation via activation of cdc2 and delays differentiation of myeloid progenitors

STI571 inactivation of the gastrointestinal stromal tumor c-KIT oncoprotein : biological and clinical implications

Inhibitors of the Transcription Factor STAT3 Decrease Growth and Induce Immune Response Genes in Models of Malignant Pleural Mesothelioma (MPM)

Combination of Rapamycin and Protein Tyrosine Kinase (PTK) Inhibitors for the Treatment of Leukemias Caused by Oncogenic PTKs

Growth Suppression of Pre-T Acute Lymphoblastic Leukemia Cells by Inhibition of Notch Signaling

Repurposing of Kinase Inhibitors for Treatment of COVID-19

MAML1, a human homologue of Drosophila Mastermind, is a transcriptional co-activator for NOTCH receptors

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