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The KCNQ1 potassium channel is down-regulated by ubiquitylating enzymes of the Nedd4/Nedd4-like family

Cardiovascular research, 2007-04, Vol.74 (1), p.64-74

2007

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Complex Brugada syndrome inheritance in a family harbouring compound SCN5A and CACNA1C mutations

Basic research in cardiology, 2014-11, Vol.109 (6), p.446-446, Article 446

2014

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Guest Editors' Introduction

Journal of molecular and cellular cardiology, 2010, Vol.48 (1), p.1-1

2010

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Guest Editors' Introduction

Journal of molecular and cellular cardiology, 2010-01, Vol.48 (1), p.1

2010

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Cardiac arrhythmias are a major clinical problem. Guest editors' introduction

Journal of molecular and cellular cardiology, 2010-01, Vol.48 (1), p.1-1

2010

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HIV-Tat induces a decrease in IKr and IKs via reduction in phosphatidylinositol-(4,5)-bisphosphate availability

Journal of molecular and cellular cardiology, 2016-10

2016

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β3-Adrenoceptor Control the Cystic Fibrosis Transmembrane Conductance Regulator through a cAMP/Protein Kinase A-independent Pathway

The Journal of biological chemistry, 1999-03, Vol.274 (10), p.6107-6113

1999

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Guest Editors' Introduction

Journal of molecular and cellular cardiology, 2009, Vol.48 (1), p.1-1

2009

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IKs response to protein kinase A-dependent KCNQ1 phosphorylation requires direct interaction with microtubules

Cardiovascular research, 2008-08, Vol.79 (3), p.427-435

2008

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A Molecular Substrate for Long QT in HIV Patients: Tat Protein Reduces IKR in Human Induced Pluripotent Stem Cells-Derived Cardiomyocytes

Biophysical journal, 2016-02, Vol.110 (3), p.103a-103a

2016

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HIV-Tat Induces a Decrease in IKr and IKs via Reduction in Phosphatidylinositol-(4,5)-Bisphosphate Availability

Biophysical journal, 2017-02, Vol.112 (3), p.405a-405a

2017

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HIV-Tat Induces a Decrease in I Kr and I Ks via Reduction in Phosphatidylinositol-(4,5)-Bisphosphate Availability

Biophysical journal, 2017-02, Vol.112 (3), p.405

2017

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A Common Antitussive Drug, Clobutinol, Precipitates the Long QT Syndrome 2

Molecular pharmacology, 2004-11, Vol.66 (5), p.1093-1102

2004

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Hyperexpression of recombinant CFTR in heterologous cells alters its physiological properties

American Journal of Physiology: Cell Physiology, 1998-02, Vol.274 (2), p.C310-C318

1998

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HIV-Tat induces a decrease in IKr and IKsvia reduction in phosphatidylinositol-(4,5)-bisphosphate availability

Journal of molecular and cellular cardiology, 2016-10, Vol.99, p.1-13

2016

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Gastrointestinal prokinetic drugs have different affinity for the human cardiac human ether-à-gogo K(+) channel

The Journal of pharmacology and experimental therapeutics, 2001-12, Vol.299 (3), p.1007

2001

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New KCNQ1 mutations leading to haploinsufficiency in a general population: Defective trafficking of a KvLQT1 mutant

Cardiovascular research, 2004-07, Vol.63 (1), p.60-68

2004

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KCNQ1 K+ channel-mediated cardiac channelopathies

Methods in molecular biology (Clifton, N.J.), 2006, Vol.337, p.167

2006

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0417 : SCN5A+/ΔQKP mice present late sodium current associated with long QT syndrome, dilated cardiomyopathy and ranolazine-sensitive spontaneous ventricular arrhythmias

Archives of Cardiovascular Diseases Supplements, 2015-04, Vol.7 (2), p.169-169

2015

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Dual Effect of PIP2 on Shaker K+ Channels

Biophysical journal, 2013-01, Vol.104 (2), p.464a-464a

2013

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In KCNQ1 Channels, a Long QT Mutation Induces a Regulation by Cholesterol Instead of Phosphatidylinositol-4,5-Bisphosphate

Biophysical journal, 2014-01, Vol.106 (2), p.140a-140a

2014

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Opposite Effects of the S4–S5 Linker and PIP2 on Voltage-Gated Channel Function: KCNQ1/KCNE1 and Other Channels

Frontiers in pharmacology, 2012, Vol.3

2012

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Arrhythmias precede cardiomyopathy and remodeling of Ca 2+ handling proteins in a novel model of long QT syndrome

Journal of molecular and cellular cardiology, 2018-08, Vol.123, p.13

2018

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KCNE1-KCNQ1 osmoregulation by interaction of phosphatidylinositol-4,5-bisphosphate with Mg 2+ and polyamines: Mechanism of KCNE1-KCNQ1 osmoregulation

The Journal of physiology, 2010-09, Vol.588 (18), p.3471-3483

2010

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KCNQ1-R539W Mutation Substitutes Cholesterol for Phosphatidylinositol-4, 5-Bisphosphate in Channel Regulation

Biophysical journal, 2011-02, Vol.100 (3), p.428a-428a

2011

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The KCNQ1 potassium channel is down-regulated by ubiquitylating enzymes of the Nedd4/Nedd4-like family

Complex Brugada syndrome inheritance in a family harbouring compound SCN5A and CACNA1C mutations

Guest Editors' Introduction

Guest Editors' Introduction

Cardiac arrhythmias are a major clinical problem. Guest editors' introduction

HIV-Tat induces a decrease in IKr and IKs via reduction in phosphatidylinositol-(4,5)-bisphosphate availability

β3-Adrenoceptor Control the Cystic Fibrosis Transmembrane Conductance Regulator through a cAMP/Protein Kinase A-independent Pathway

Guest Editors' Introduction

IKs response to protein kinase A-dependent KCNQ1 phosphorylation requires direct interaction with microtubules

A Molecular Substrate for Long QT in HIV Patients: Tat Protein Reduces IKR in Human Induced Pluripotent Stem Cells-Derived Cardiomyocytes

HIV-Tat Induces a Decrease in IKr and IKs via Reduction in Phosphatidylinositol-(4,5)-Bisphosphate Availability

HIV-Tat Induces a Decrease in I Kr and I Ks via Reduction in Phosphatidylinositol-(4,5)-Bisphosphate Availability

A Common Antitussive Drug, Clobutinol, Precipitates the Long QT Syndrome 2

Hyperexpression of recombinant CFTR in heterologous cells alters its physiological properties

HIV-Tat induces a decrease in IKr and IKsvia reduction in phosphatidylinositol-(4,5)-bisphosphate availability

Gastrointestinal prokinetic drugs have different affinity for the human cardiac human ether-à-gogo K(+) channel

New KCNQ1 mutations leading to haploinsufficiency in a general population: Defective trafficking of a KvLQT1 mutant

KCNQ1 K+ channel-mediated cardiac channelopathies

0417 : SCN5A+/ΔQKP mice present late sodium current associated with long QT syndrome, dilated cardiomyopathy and ranolazine-sensitive spontaneous ventricular arrhythmias

Dual Effect of PIP2 on Shaker K+ Channels

In KCNQ1 Channels, a Long QT Mutation Induces a Regulation by Cholesterol Instead of Phosphatidylinositol-4,5-Bisphosphate

Opposite Effects of the S4–S5 Linker and PIP2 on Voltage-Gated Channel Function: KCNQ1/KCNE1 and Other Channels

Arrhythmias precede cardiomyopathy and remodeling of Ca 2+ handling proteins in a novel model of long QT syndrome

KCNE1-KCNQ1 osmoregulation by interaction of phosphatidylinositol-4,5-bisphosphate with Mg 2+ and polyamines: Mechanism of KCNE1-KCNQ1 osmoregulation

KCNQ1-R539W Mutation Substitutes Cholesterol for Phosphatidylinositol-4, 5-Bisphosphate in Channel Regulation

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