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Can we switch production of toxic Aβ oligomers to neuroprotective Aβ monomers to allow synapse regeneration

Neural regeneration research, 2017-09, Vol.12 (9), p.1437-1438

Bate, Clive

2017

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GPI-anchor signal sequence influences PrPC sorting, shedding and signalling, and impacts on different pathomechanistic aspects of prion disease in mice

PLoS pathogens, 2019-01, Vol.15 (1), p.e1007520-e1007520

2019

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cAMP-Inhibits Cytoplasmic Phospholipase A₂ and Protects Neurons against Amyloid-β-Induced Synapse Damage

Biology (Basel, Switzerland), 2015-09, Vol.4 (3), p.591-606

2015

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Squalestatin Cures Prion-infected Neurons and Protects Against Prion Neurotoxicity

The Journal of biological chemistry, 2004-04, Vol.279 (15), p.14983-14990

2004

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Amyloid-β-induced Synapse Damage Is Mediated via Cross-linkage of Cellular Prion Proteins

The Journal of biological chemistry, 2011-11, Vol.286 (44), p.37955-37963

2011

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A camelid anti-PrP antibody abrogates PrP replication in prion-permissive neuroblastoma cell lines

PloS one, 2010-03, Vol.5 (3), p.e9804

2010

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Monoacylated Cellular Prion Proteins Reduce Amyloid-β-Induced Activation of Cytoplasmic Phospholipase A2 and Synapse Damage

Biology (Basel, Switzerland), 2015-06, Vol.4 (2), p.367-382

2015

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Interferon-γ increases neuronal death in response to amyloid-β1-42

Journal of neuroinflammation, 2006-03, Vol.3 (1), p.7-7

2006

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Sequestration of free cholesterol in cell membranes by prions correlates with cytoplasmic phospholipase A2 activation

BMC biology, 2008-02, Vol.6 (1), p.8-8

2008

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Neurodegeneration Induced by Clustering of Sialylated Glycosylphosphatidylinositols of Prion Proteins

The Journal of biological chemistry, 2012-03, Vol.287 (11), p.7935-7944

2012

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Phospholipase A2 Inhibitors or Platelet-activating Factor Antagonists Prevent Prion Replication

The Journal of biological chemistry, 2004-08, Vol.279 (35), p.36405-36411

2004

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Can we switch production of toxic A[beta] oligomers to neuroprotective A[beta] monomers to allow synapse regeneration?

Neural regeneration research, 2017-09, Vol.12 (9), p.1437

Bate, Clive

2017

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Ethanol protects cultured neurons against amyloid-β and α-synuclein-induced synapse damage

Neuropharmacology, 2011-12, Vol.61 (8), p.1406-1412

2011

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Cholesterol synthesis inhibitors protect against platelet-activating factor-induced neuronal damage

Journal of neuroinflammation, 2007, Vol.4 (1), p.5-5

2007

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Polyunsaturated Fatty Acids Protect Against Prion-Mediated Synapse Damage In Vitro

Neurotoxicity research, 2010-04, Vol.17 (3), p.203-214

2010

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Docosahexaenoic and eicosapentaenoic acids increase prion formation in neuronal cells

BMC biology, 2008-09, Vol.6 (1), p.39-39, Article 39

2008

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Sialylated glycosylphosphatidylinositols suppress the production of toxic amyloid-β oligomers

Biochemical journal, 2017-09, Vol.474 (17), p.3045-3058

2017

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α-synuclein induced synapse damage is enhanced by amyloid-β1-42

Molecular neurodegeneration, 2010-12, Vol.5 (1), p.55-55

2010

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Enhanced neuronal degradation of amyloid-[beta] oligomers allows synapse regeneration

Neural regeneration research, 2015-05, Vol.10 (5)

Bate, Clive

2015

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An in vitro model for synaptic loss in neurodegenerative diseases suggests a neuroprotective role for valproic acid via inhibition of cPLA2 dependent signalling

Neuropharmacology, 2016-02, Vol.101, p.566-575

2016

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Squalestatin alters the intracellular trafficking of a neurotoxic prion peptide

BMC neuroscience, 2007-11, Vol.8 (1), p.99-99, Article 99

2007

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Phospholipase A2 inhibitors protect against prion and Aβ mediated synapse degeneration

Molecular neurodegeneration, 2010-04, Vol.5 (1), p.13-13

2010

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Glimepiride reduces CD14 expression and cytokine secretion from macrophages

Journal of neuroinflammation, 2014-06, Vol.11 (1), p.115-115, Article 115

2014

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Amyloid-β1–40 Inhibits Amyloid-β1–42 Induced Activation of Cytoplasmic Phospholipase A2 and Synapse Degeneration

Journal of Alzheimer's disease, 2010-09, Vol.21 (3), p.985-993

2010

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Glimepiride protects neurons against amyloid-β-induced synapse damage

Neuropharmacology, 2016-02, Vol.101, p.225-236

2016

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Can we switch production of toxic Aβ oligomers to neuroprotective Aβ monomers to allow synapse regeneration

GPI-anchor signal sequence influences PrPC sorting, shedding and signalling, and impacts on different pathomechanistic aspects of prion disease in mice

cAMP-Inhibits Cytoplasmic Phospholipase A₂ and Protects Neurons against Amyloid-β-Induced Synapse Damage

Squalestatin Cures Prion-infected Neurons and Protects Against Prion Neurotoxicity

Amyloid-β-induced Synapse Damage Is Mediated via Cross-linkage of Cellular Prion Proteins

A camelid anti-PrP antibody abrogates PrP replication in prion-permissive neuroblastoma cell lines

Monoacylated Cellular Prion Proteins Reduce Amyloid-β-Induced Activation of Cytoplasmic Phospholipase A2 and Synapse Damage

Interferon-γ increases neuronal death in response to amyloid-β1-42

Sequestration of free cholesterol in cell membranes by prions correlates with cytoplasmic phospholipase A2 activation

Neurodegeneration Induced by Clustering of Sialylated Glycosylphosphatidylinositols of Prion Proteins

Phospholipase A2 Inhibitors or Platelet-activating Factor Antagonists Prevent Prion Replication

Can we switch production of toxic A[beta] oligomers to neuroprotective A[beta] monomers to allow synapse regeneration?

Ethanol protects cultured neurons against amyloid-β and α-synuclein-induced synapse damage

Cholesterol synthesis inhibitors protect against platelet-activating factor-induced neuronal damage

Polyunsaturated Fatty Acids Protect Against Prion-Mediated Synapse Damage In Vitro

Docosahexaenoic and eicosapentaenoic acids increase prion formation in neuronal cells

Sialylated glycosylphosphatidylinositols suppress the production of toxic amyloid-β oligomers

α-synuclein induced synapse damage is enhanced by amyloid-β1-42

Enhanced neuronal degradation of amyloid-[beta] oligomers allows synapse regeneration

An in vitro model for synaptic loss in neurodegenerative diseases suggests a neuroprotective role for valproic acid via inhibition of cPLA2 dependent signalling

Squalestatin alters the intracellular trafficking of a neurotoxic prion peptide

Phospholipase A2 inhibitors protect against prion and Aβ mediated synapse degeneration

Glimepiride reduces CD14 expression and cytokine secretion from macrophages

Amyloid-β1–40 Inhibits Amyloid-β1–42 Induced Activation of Cytoplasmic Phospholipase A2 and Synapse Degeneration

Glimepiride protects neurons against amyloid-β-induced synapse damage

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