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Succinylation Links Metabolism to Protein Functions

Neurochemical research, 2019-10, Vol.44 (10), p.2346-2359

2019

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Cause and consequence: Mitochondrial dysfunction initiates and propagates neuronal dysfunction, neuronal death and behavioral abnormalities in age-associated neurodegenerative diseases

Biochimica et biophysica acta, 2010-01, Vol.1802 (1), p.122-134

2010

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Benfotiamine treatment activates the Nrf2/ARE pathway and is neuroprotective in a transgenic mouse model of tauopathy

Human molecular genetics, 2018-08, Vol.27 (16), p.2874-2892

2018

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Oxidative Stress and Transcriptional Regulation in Alzheimer Disease

Alzheimer disease and associated disorders, 2007-10, Vol.21 (4), p.276-291

2007

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Mitochondrial abnormalities in Alzheimer brain: Mechanistic implications

Annals of neurology, 2005-05, Vol.57 (5), p.695-703

2005

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Novel Metabolic Abnormalities in the Tricarboxylic Acid Cycle in Peripheral Cells From Huntington's Disease Patients

PloS one, 2016-09, Vol.11 (9), p.e0160384

2016

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Obituary: In Memory of Myron Flint Beal, MD, November 6, 1950–June 12, 2021

Movement disorders, 2021-09, Vol.36 (9), p.2003-2004

2021

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Changes in inflammatory processes associated with selective vulnerability following mild impairment of oxidative metabolism

Neurobiology of disease, 2007-05, Vol.26 (2), p.353-362

2007

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Thiamine and Oxidants Interact to Modify Cellular Calcium Stores

Neurochemical research, 2010-12, Vol.35 (12), p.2107-2116

2010

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Dopaminergic cell death induced by MPP+, oxidant and specific neurotoxicants shares the common molecular mechanism

Journal of neurochemistry, 2001-02, Vol.76 (4), p.1010-1021

2001

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Selective NADH communication from α-ketoglutarate dehydrogenase to mitochondrial transhydrogenase prevents reactive oxygen species formation under reducing conditions in the heart

Basic research in cardiology, 2020-09, Vol.115 (5), p.53, Article 53

2020

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The RNA-binding protein HuR is essential for the B cell antibody response

Nature immunology, 2015-04, Vol.16 (4), p.415-425

2015

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Abnormal Glucose Metabolism in Alzheimer’s Disease: Relation to Autophagy/Mitophagy and Therapeutic Approaches

Neurochemical research, 2015-12, Vol.40 (12), p.2557-2569

2015

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Mild metabolic perturbations alter succinylation of mitochondrial proteins

Journal of neuroscience research, 2017-11, Vol.95 (11), p.2244-2252

2017

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Presenilins Are Enriched in Endoplasmic Reticulum Membranes Associated with Mitochondria

The American journal of pathology, 2009-11, Vol.175 (5), p.1810-1816

2009

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Reductions in the mitochondrial enzyme α‐ketoglutarate dehydrogenase complex in neurodegenerative disease – beneficial or detrimental?

Journal of neurochemistry, 2016-12, Vol.139 (5), p.823-838

2016

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Reductions in the mitochondrial enzyme [alpha]-ketoglutarate dehydrogenase complex in neurodegenerative disease - beneficial or detrimental?

Journal of neurochemistry, 2016-12, Vol.139 (5), p.823

2016

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Reversal of Thiamine Deficiency-Induced Neurodegeneration

Journal of neuropathology and experimental neurology, 2003-02, Vol.62 (2), p.195-207

2003

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Nutritional metabolism and cerebral bioenergetics in Alzheimer's disease and related dementias

Alzheimer's & dementia, 2023-03, Vol.19 (3), p.1041-1066

2023

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Inactivation and Reactivation of the Mitochondrial α-Ketoglutarate Dehydrogenase Complex

The Journal of biological chemistry, 2011-05, Vol.286 (20), p.17640-17648

2011

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Mice deficient in dihydrolipoyl succinyl transferase show increased vulnerability to mitochondrial toxins

Neurobiology of disease, 2009-11, Vol.36 (2), p.320-330

2009

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Abnormalities in the Tricarboxylic Acid Cycle in Huntington Disease and in a Huntington Disease Mouse Model

Journal of neuropathology and experimental neurology, 2015-06, Vol.74 (6), p.527-537

2015

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Thiamine-dependent processes and treatment strategies in neurodegeneration

Antioxidants & redox signaling, 2007-10, Vol.9 (10), p.1605-1620

2007

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The negative impact of α‐ketoglutarate dehydrogenase complex deficiency on matrix substrate‐level phosphorylation

The FASEB journal, 2013-06, Vol.27 (6), p.2392-2406

2013

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Oxidative stress in Alzheimer's disease

Neurobiology of aging, 2005-05, Vol.26 (5), p.575-578

2005

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Succinylation Links Metabolism to Protein Functions

Cause and consequence: Mitochondrial dysfunction initiates and propagates neuronal dysfunction, neuronal death and behavioral abnormalities in age-associated neurodegenerative diseases

Benfotiamine treatment activates the Nrf2/ARE pathway and is neuroprotective in a transgenic mouse model of tauopathy

Oxidative Stress and Transcriptional Regulation in Alzheimer Disease

Mitochondrial abnormalities in Alzheimer brain: Mechanistic implications

Novel Metabolic Abnormalities in the Tricarboxylic Acid Cycle in Peripheral Cells From Huntington's Disease Patients

Obituary: In Memory of Myron Flint Beal, MD, November 6, 1950–June 12, 2021

Changes in inflammatory processes associated with selective vulnerability following mild impairment of oxidative metabolism

Thiamine and Oxidants Interact to Modify Cellular Calcium Stores

Dopaminergic cell death induced by MPP+, oxidant and specific neurotoxicants shares the common molecular mechanism

Selective NADH communication from α-ketoglutarate dehydrogenase to mitochondrial transhydrogenase prevents reactive oxygen species formation under reducing conditions in the heart

The RNA-binding protein HuR is essential for the B cell antibody response

Abnormal Glucose Metabolism in Alzheimer’s Disease: Relation to Autophagy/Mitophagy and Therapeutic Approaches

Mild metabolic perturbations alter succinylation of mitochondrial proteins

Presenilins Are Enriched in Endoplasmic Reticulum Membranes Associated with Mitochondria

Reductions in the mitochondrial enzyme α‐ketoglutarate dehydrogenase complex in neurodegenerative disease – beneficial or detrimental?

Reductions in the mitochondrial enzyme [alpha]-ketoglutarate dehydrogenase complex in neurodegenerative disease - beneficial or detrimental?

Reversal of Thiamine Deficiency-Induced Neurodegeneration

Nutritional metabolism and cerebral bioenergetics in Alzheimer's disease and related dementias

Inactivation and Reactivation of the Mitochondrial α-Ketoglutarate Dehydrogenase Complex

Mice deficient in dihydrolipoyl succinyl transferase show increased vulnerability to mitochondrial toxins

Abnormalities in the Tricarboxylic Acid Cycle in Huntington Disease and in a Huntington Disease Mouse Model

Thiamine-dependent processes and treatment strategies in neurodegeneration

The negative impact of α‐ketoglutarate dehydrogenase complex deficiency on matrix substrate‐level phosphorylation

Oxidative stress in Alzheimer's disease

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