UB Paderborn / Katalog / Suche / in CDI

1 – 25 von 166

Sortiert nach

Defining sporadic Creutzfeldt-Jakob disease strains and their transmission properties

Proceedings of the National Academy of Sciences - PNAS, 2010-06, Vol.107 (26), p.12005-12010

2010

Link zum Volltext

Verfügbarkeit prüfen

Distribution of Misfolded Prion Protein Seeding Activity Alone Does Not Predict Regions of Neurodegeneration

PLoS biology, 2016-11, Vol.14 (11), p.e1002579-e1002579

2016

Link zum Volltext

Verfügbarkeit prüfen

Defining the Microglia Response during the Time Course of Chronic Neurodegeneration

Journal of virology, 2016-03, Vol.90 (6), p.3003-3017

2016

Link zum Volltext

Verfügbarkeit prüfen

Follicular dendritic cell-specific prion protein (PrP) expression alone is sufficient to sustain prion infection in the spleen

PLoS pathogens, 2011-12, Vol.7 (12), p.e1002402-e1002402

2011

Link zum Volltext

Verfügbarkeit prüfen

Development of a sensitive real-time quaking-induced conversion (RT-QuIC) assay for application in prion-infected blood

PloS one, 2023-11, Vol.18 (11), p.e0293845-e0293845

2023

Link zum Volltext

Verfügbarkeit prüfen

Public health risks from subclinical variant CJD

PLoS pathogens, 2017-11, Vol.13 (11), p.e1006642-e1006642

2017

Link zum Volltext

Verfügbarkeit prüfen

Preclinical transmission of prions by blood transfusion is influenced by donor genotype and route of infection

PLoS pathogens, 2021-02, Vol.17 (2), p.e1009276

2021

Link zum Volltext

Verfügbarkeit prüfen

All clinically-relevant blood components transmit prion disease following a single blood transfusion: a sheep model of vCJD

PloS one, 2011-08, Vol.6 (8), p.e23169-e23169

2011

Link zum Volltext

Verfügbarkeit prüfen

Spermine increases acetylation of tubulins and facilitates autophagic degradation of prion aggregates

Scientific reports, 2018-07, Vol.8 (1), p.10004-17, Article 10004

2018

Link zum Volltext

Verfügbarkeit prüfen

Identification of a homology-independent linchpin domain controlling mouse and bank vole prion protein conversion

PLoS pathogens, 2020-09, Vol.16 (9), p.e1008875-e1008875

2020

Link zum Volltext

Verfügbarkeit prüfen

Accumulation of prion protein in the brain that is not associated with transmissible disease

Proceedings of the National Academy of Sciences - PNAS, 2007-03, Vol.104 (11), p.4712-4717

2007

Link zum Volltext

Verfügbarkeit prüfen

Subclinical infection occurs frequently following low dose exposure to prions by blood transfusion

Scientific reports, 2022-06, Vol.12 (1), p.10923-10923, Article 10923

2022

Link zum Volltext

Verfügbarkeit prüfen

Post-translational changes to PrP alter transmissible spongiform encephalopathy strain properties

The EMBO journal, 2013-03, Vol.32 (5), p.756-769

2013

Link zum Volltext

Verfügbarkeit prüfen

High Titers of Transmissible Spongiform Encephalopathy Infectivity Associated with Extremely Low Levels of PrPSc in Vivo

The Journal of biological chemistry, 2007-12, Vol.282 (49), p.35878-35886

2007

Link zum Volltext

Verfügbarkeit prüfen

Pro-death NMDA receptor signaling is promoted by the GluN2B C-terminus independently of Dapk1

eLife, 2017-07, Vol.6

2017

Link zum Volltext

Verfügbarkeit prüfen

Host PrP glycosylation: a major factor determining the outcome of prion infection

PLoS biology, 2008-04, Vol.6 (4), p.e100-e100

2008

Link zum Volltext

Verfügbarkeit prüfen

Similarities of Variant Creutzfeldt-Jakob Disease Strain in Mother and Son in Spain to UK Reference Case

Emerging infectious diseases, 2017-09, Vol.23 (9), p.1593-1596

2017

Link zum Volltext

Verfügbarkeit prüfen

Ablation of Prion Protein in Wild Type Human Amyloid Precursor Protein (APP) Transgenic Mice Does Not Alter The Proteolysis of APP, Levels of Amyloid-β or Pathologic Phenotype

PloS one, 2016-07, Vol.11 (7), p.e0159119

2016

Link zum Volltext

Verfügbarkeit prüfen

Mice devoid of prion protein have cognitive deficits that are rescued by reconstitution of PrP in neurons

Neurobiology of disease, 2005-06, Vol.19 (1), p.255-265

2005

Link zum Volltext

Verfügbarkeit prüfen

Glycosylation of PrPC Determines Timing of Neuroinvasion and Targeting in the Brain following Transmissible Spongiform Encephalopathy Infection by a Peripheral Route

Journal of Virology, 2010-04, Vol.84 (7), p.3464-3475

2010

Link zum Volltext

Verfügbarkeit prüfen

Unravelling the glial response in the pathogenesis of Alzheimer's disease

The FASEB journal, 2018-11, Vol.32 (11), p.5766-5777

2018

Link zum Volltext

Verfügbarkeit prüfen

Constant transmission properties of variant Creutzfeldt-Jakob disease in 5 countries

Emerging infectious diseases, 2012-10, Vol.18 (10), p.1574-1579

2012

Link zum Volltext

Verfügbarkeit prüfen

Cellular prion protein expression is not regulated by the Alzheimer's amyloid precursor protein intracellular domain

PloS one, 2012-02, Vol.7 (2), p.e31754

2012

Link zum Volltext

Verfügbarkeit prüfen

Conditional Deletion of Prnp Rescues Behavioral and Synaptic Deficits after Disease Onset in Transgenic Alzheimer's Disease

The Journal of neuroscience, 2017-09, Vol.37 (38), p.9207-9221

2017

Link zum Volltext

Verfügbarkeit prüfen

Altered Glycosylated PrP Proteins Can Have Different Neuronal Trafficking in Brain but Do Not Acquire Scrapie-like Properties

The Journal of biological chemistry, 2005-12, Vol.280 (52), p.42909-42918

2005

Link zum Volltext

Verfügbarkeit prüfen

1 – 25 von 166

Suche als RSS-Feed

Aktive Filter

PublikationsformTexte

ThemaPrions - Genetics

ThemaWomen's Health

Suchergebnisse filtern

Filter anzeigen

Nur zeigen

Geprüft (Peer-Review)
(154)
Mit Volltextzugriff
(146)

Publikationsform

Sprache

Erscheinungsjahr

n.n

Thema

15 weitere anzeigen...

Weniger anzeigen...

Neu hinzugefügt

... seit 90 Tagen
(1)

Menü

Weitere Dienste

Einstellungen

Defining sporadic Creutzfeldt-Jakob disease strains and their transmission properties

Distribution of Misfolded Prion Protein Seeding Activity Alone Does Not Predict Regions of Neurodegeneration

Defining the Microglia Response during the Time Course of Chronic Neurodegeneration

Follicular dendritic cell-specific prion protein (PrP) expression alone is sufficient to sustain prion infection in the spleen

Development of a sensitive real-time quaking-induced conversion (RT-QuIC) assay for application in prion-infected blood

Public health risks from subclinical variant CJD

Preclinical transmission of prions by blood transfusion is influenced by donor genotype and route of infection

All clinically-relevant blood components transmit prion disease following a single blood transfusion: a sheep model of vCJD

Spermine increases acetylation of tubulins and facilitates autophagic degradation of prion aggregates

Identification of a homology-independent linchpin domain controlling mouse and bank vole prion protein conversion

Accumulation of prion protein in the brain that is not associated with transmissible disease

Subclinical infection occurs frequently following low dose exposure to prions by blood transfusion

Post-translational changes to PrP alter transmissible spongiform encephalopathy strain properties

High Titers of Transmissible Spongiform Encephalopathy Infectivity Associated with Extremely Low Levels of PrPSc in Vivo

Pro-death NMDA receptor signaling is promoted by the GluN2B C-terminus independently of Dapk1

Host PrP glycosylation: a major factor determining the outcome of prion infection

Similarities of Variant Creutzfeldt-Jakob Disease Strain in Mother and Son in Spain to UK Reference Case

Ablation of Prion Protein in Wild Type Human Amyloid Precursor Protein (APP) Transgenic Mice Does Not Alter The Proteolysis of APP, Levels of Amyloid-β or Pathologic Phenotype

Mice devoid of prion protein have cognitive deficits that are rescued by reconstitution of PrP in neurons

Glycosylation of PrPC Determines Timing of Neuroinvasion and Targeting in the Brain following Transmissible Spongiform Encephalopathy Infection by a Peripheral Route

Unravelling the glial response in the pathogenesis of Alzheimer's disease

Constant transmission properties of variant Creutzfeldt-Jakob disease in 5 countries

Cellular prion protein expression is not regulated by the Alzheimer's amyloid precursor protein intracellular domain

Conditional Deletion of Prnp Rescues Behavioral and Synaptic Deficits after Disease Onset in Transgenic Alzheimer's Disease

Altered Glycosylated PrP Proteins Can Have Different Neuronal Trafficking in Brain but Do Not Acquire Scrapie-like Properties

Aktive Filter

Suchergebnisse filtern