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Defining sporadic Creutzfeldt-Jakob disease strains and their transmission properties

Proceedings of the National Academy of Sciences - PNAS, 2010-06, Vol.107 (26), p.12005-12010

2010

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Distribution of Misfolded Prion Protein Seeding Activity Alone Does Not Predict Regions of Neurodegeneration

PLoS biology, 2016-11, Vol.14 (11), p.e1002579-e1002579

2016

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Defining the Microglia Response during the Time Course of Chronic Neurodegeneration

Journal of virology, 2016-03, Vol.90 (6), p.3003-3017

2016

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Follicular dendritic cell-specific prion protein (PrP) expression alone is sufficient to sustain prion infection in the spleen

PLoS pathogens, 2011-12, Vol.7 (12), p.e1002402-e1002402

2011

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Development of a sensitive real-time quaking-induced conversion (RT-QuIC) assay for application in prion-infected blood

PloS one, 2023-11, Vol.18 (11), p.e0293845-e0293845

2023

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Public health risks from subclinical variant CJD

PLoS pathogens, 2017-11, Vol.13 (11), p.e1006642-e1006642

2017

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Preclinical transmission of prions by blood transfusion is influenced by donor genotype and route of infection

PLoS pathogens, 2021-02, Vol.17 (2), p.e1009276

2021

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All clinically-relevant blood components transmit prion disease following a single blood transfusion: a sheep model of vCJD

PloS one, 2011-08, Vol.6 (8), p.e23169-e23169

2011

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Spermine increases acetylation of tubulins and facilitates autophagic degradation of prion aggregates

Scientific reports, 2018-07, Vol.8 (1), p.10004-17, Article 10004

2018

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No Adaptation of the Prion Strain in a Heterozygous Case of Variant Creutzfeldt-Jakob Disease

Emerging infectious diseases, 2020-06, Vol.26 (6), p.1300-1303

2020

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Identification of a homology-independent linchpin domain controlling mouse and bank vole prion protein conversion

PLoS pathogens, 2020-09, Vol.16 (9), p.e1008875-e1008875

2020

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Accumulation of prion protein in the brain that is not associated with transmissible disease

Proceedings of the National Academy of Sciences - PNAS, 2007-03, Vol.104 (11), p.4712-4717

2007

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Cellular prion protein regulates β-secretase cleavage of the Alzheimer's amyloid precursor protein

Proceedings of the National Academy of Sciences - PNAS, 2007-06, Vol.104 (26), p.11062-11067

2007

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Subclinical infection occurs frequently following low dose exposure to prions by blood transfusion

Scientific reports, 2022-06, Vol.12 (1), p.10923-10923, Article 10923

2022

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Post-translational changes to PrP alter transmissible spongiform encephalopathy strain properties

The EMBO journal, 2013-03, Vol.32 (5), p.756-769

2013

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High Titers of Transmissible Spongiform Encephalopathy Infectivity Associated with Extremely Low Levels of PrPSc in Vivo

The Journal of biological chemistry, 2007-12, Vol.282 (49), p.35878-35886

2007

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Pro-death NMDA receptor signaling is promoted by the GluN2B C-terminus independently of Dapk1

eLife, 2017-07, Vol.6

2017

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Insights into Mechanisms of Chronic Neurodegeneration

International Journal of Molecular Sciences, 2016-01, Vol.17 (1), p.82-82

2016

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Variably protease-sensitive prionopathy, a unique prion variant with inefficient transmission properties

Emerging infectious diseases, 2014-12, Vol.20 (12), p.1969-1979

2014

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Dissociation of Prion Protein Amyloid Seeding from Transmission of a Spongiform Encephalopathy

Journal of Virology, 2013-11, Vol.87 (22), p.12349-12356

2013

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Host PrP glycosylation: a major factor determining the outcome of prion infection

PLoS biology, 2008-04, Vol.6 (4), p.e100-e100

2008

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Similarities of Variant Creutzfeldt-Jakob Disease Strain in Mother and Son in Spain to UK Reference Case

Emerging infectious diseases, 2017-09, Vol.23 (9), p.1593-1596

2017

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Ablation of Prion Protein in Wild Type Human Amyloid Precursor Protein (APP) Transgenic Mice Does Not Alter The Proteolysis of APP, Levels of Amyloid-β or Pathologic Phenotype

PloS one, 2016-07, Vol.11 (7), p.e0159119

2016

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MM2-Thalamic Creutzfeldt-Jakob Disease: Neuropathological, Biochemical and Transmission Studies Identify a Distinctive Prion Strain

Brain pathology (Zurich, Switzerland), 2012-09, Vol.22 (5), p.662-669

2012

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Increased Susceptibility of Human-PrP Transgenic Mice to Bovine Spongiform Encephalopathy Infection following Passage in Sheep

Journal of Virology, 2011-02, Vol.85 (3), p.1174-1181

2011

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Defining sporadic Creutzfeldt-Jakob disease strains and their transmission properties

Distribution of Misfolded Prion Protein Seeding Activity Alone Does Not Predict Regions of Neurodegeneration

Defining the Microglia Response during the Time Course of Chronic Neurodegeneration

Follicular dendritic cell-specific prion protein (PrP) expression alone is sufficient to sustain prion infection in the spleen

Development of a sensitive real-time quaking-induced conversion (RT-QuIC) assay for application in prion-infected blood

Public health risks from subclinical variant CJD

Preclinical transmission of prions by blood transfusion is influenced by donor genotype and route of infection

All clinically-relevant blood components transmit prion disease following a single blood transfusion: a sheep model of vCJD

Spermine increases acetylation of tubulins and facilitates autophagic degradation of prion aggregates

No Adaptation of the Prion Strain in a Heterozygous Case of Variant Creutzfeldt-Jakob Disease

Identification of a homology-independent linchpin domain controlling mouse and bank vole prion protein conversion

Accumulation of prion protein in the brain that is not associated with transmissible disease

Cellular prion protein regulates β-secretase cleavage of the Alzheimer's amyloid precursor protein

Subclinical infection occurs frequently following low dose exposure to prions by blood transfusion

Post-translational changes to PrP alter transmissible spongiform encephalopathy strain properties

High Titers of Transmissible Spongiform Encephalopathy Infectivity Associated with Extremely Low Levels of PrPSc in Vivo

Pro-death NMDA receptor signaling is promoted by the GluN2B C-terminus independently of Dapk1

Insights into Mechanisms of Chronic Neurodegeneration

Variably protease-sensitive prionopathy, a unique prion variant with inefficient transmission properties

Dissociation of Prion Protein Amyloid Seeding from Transmission of a Spongiform Encephalopathy

Host PrP glycosylation: a major factor determining the outcome of prion infection

Similarities of Variant Creutzfeldt-Jakob Disease Strain in Mother and Son in Spain to UK Reference Case

Ablation of Prion Protein in Wild Type Human Amyloid Precursor Protein (APP) Transgenic Mice Does Not Alter The Proteolysis of APP, Levels of Amyloid-β or Pathologic Phenotype

MM2-Thalamic Creutzfeldt-Jakob Disease: Neuropathological, Biochemical and Transmission Studies Identify a Distinctive Prion Strain

Increased Susceptibility of Human-PrP Transgenic Mice to Bovine Spongiform Encephalopathy Infection following Passage in Sheep

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