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Defining the Microglia Response during the Time Course of Chronic Neurodegeneration

Journal of virology, 2016-03, Vol.90 (6), p.3003-3017

2016

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Follicular dendritic cell-specific prion protein (PrP) expression alone is sufficient to sustain prion infection in the spleen

PLoS pathogens, 2011-12, Vol.7 (12), p.e1002402-e1002402

2011

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Development of a sensitive real-time quaking-induced conversion (RT-QuIC) assay for application in prion-infected blood

PloS one, 2023-11, Vol.18 (11), p.e0293845-e0293845

2023

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Public health risks from subclinical variant CJD

PLoS pathogens, 2017-11, Vol.13 (11), p.e1006642-e1006642

2017

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Preclinical transmission of prions by blood transfusion is influenced by donor genotype and route of infection

PLoS pathogens, 2021-02, Vol.17 (2), p.e1009276

2021

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Spermine increases acetylation of tubulins and facilitates autophagic degradation of prion aggregates

Scientific reports, 2018-07, Vol.8 (1), p.10004-17, Article 10004

2018

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No Adaptation of the Prion Strain in a Heterozygous Case of Variant Creutzfeldt-Jakob Disease

Emerging infectious diseases, 2020-06, Vol.26 (6), p.1300-1303

2020

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Cellular prion protein regulates β-secretase cleavage of the Alzheimer's amyloid precursor protein

Proceedings of the National Academy of Sciences - PNAS, 2007-06, Vol.104 (26), p.11062-11067

2007

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Subclinical infection occurs frequently following low dose exposure to prions by blood transfusion

Scientific reports, 2022-06, Vol.12 (1), p.10923-10923, Article 10923

2022

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Post-translational changes to PrP alter transmissible spongiform encephalopathy strain properties

The EMBO journal, 2013-03, Vol.32 (5), p.756-769

2013

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Pro-death NMDA receptor signaling is promoted by the GluN2B C-terminus independently of Dapk1

eLife, 2017-07, Vol.6

2017

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Insights into Mechanisms of Chronic Neurodegeneration

International Journal of Molecular Sciences, 2016-01, Vol.17 (1), p.82-82

2016

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Dissociation of Prion Protein Amyloid Seeding from Transmission of a Spongiform Encephalopathy

Journal of Virology, 2013-11, Vol.87 (22), p.12349-12356

2013

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Host PrP glycosylation: a major factor determining the outcome of prion infection

PLoS biology, 2008-04, Vol.6 (4), p.e100-e100

2008

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Similarities of Variant Creutzfeldt-Jakob Disease Strain in Mother and Son in Spain to UK Reference Case

Emerging infectious diseases, 2017-09, Vol.23 (9), p.1593-1596

2017

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MM2-Thalamic Creutzfeldt-Jakob Disease: Neuropathological, Biochemical and Transmission Studies Identify a Distinctive Prion Strain

Brain pathology (Zurich, Switzerland), 2012-09, Vol.22 (5), p.662-669

2012

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Increased Susceptibility of Human-PrP Transgenic Mice to Bovine Spongiform Encephalopathy Infection following Passage in Sheep

Journal of Virology, 2011-02, Vol.85 (3), p.1174-1181

2011

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Mice devoid of prion protein have cognitive deficits that are rescued by reconstitution of PrP in neurons

Neurobiology of disease, 2005-06, Vol.19 (1), p.255-265

2005

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Glycosylation of PrPC Determines Timing of Neuroinvasion and Targeting in the Brain following Transmissible Spongiform Encephalopathy Infection by a Peripheral Route

Journal of Virology, 2010-04, Vol.84 (7), p.3464-3475

2010

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Unravelling the glial response in the pathogenesis of Alzheimer's disease

The FASEB journal, 2018-11, Vol.32 (11), p.5766-5777

2018

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Cellular prion protein expression is not regulated by the Alzheimer's amyloid precursor protein intracellular domain

PloS one, 2012-02, Vol.7 (2), p.e31754

2012

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Conditional Deletion of Prnp Rescues Behavioral and Synaptic Deficits after Disease Onset in Transgenic Alzheimer's Disease

The Journal of neuroscience, 2017-09, Vol.37 (38), p.9207-9221

2017

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Changing a single amino acid in the N-terminus of murine PrP alters TSE incubation time across three species barriers

The EMBO journal, 2001-09, Vol.20 (18), p.5070-5078

2001

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Oral Prion Neuroinvasion Occurs Independently of PrPC Expression in the Gut Epithelium

Journal of virology, 2018-10, Vol.92 (19)

2018

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The glycosylation status of PrPC is a key factor in determining transmissible spongiform encephalopathy transmission between species

Journal of virology, 2015-05, Vol.89 (9), p.4738-4747

2015

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Defining the Microglia Response during the Time Course of Chronic Neurodegeneration

Follicular dendritic cell-specific prion protein (PrP) expression alone is sufficient to sustain prion infection in the spleen

Development of a sensitive real-time quaking-induced conversion (RT-QuIC) assay for application in prion-infected blood

Public health risks from subclinical variant CJD

Preclinical transmission of prions by blood transfusion is influenced by donor genotype and route of infection

Spermine increases acetylation of tubulins and facilitates autophagic degradation of prion aggregates

No Adaptation of the Prion Strain in a Heterozygous Case of Variant Creutzfeldt-Jakob Disease

Cellular prion protein regulates β-secretase cleavage of the Alzheimer's amyloid precursor protein

Subclinical infection occurs frequently following low dose exposure to prions by blood transfusion

Post-translational changes to PrP alter transmissible spongiform encephalopathy strain properties

Pro-death NMDA receptor signaling is promoted by the GluN2B C-terminus independently of Dapk1

Insights into Mechanisms of Chronic Neurodegeneration

Dissociation of Prion Protein Amyloid Seeding from Transmission of a Spongiform Encephalopathy

Host PrP glycosylation: a major factor determining the outcome of prion infection

Similarities of Variant Creutzfeldt-Jakob Disease Strain in Mother and Son in Spain to UK Reference Case

MM2-Thalamic Creutzfeldt-Jakob Disease: Neuropathological, Biochemical and Transmission Studies Identify a Distinctive Prion Strain

Increased Susceptibility of Human-PrP Transgenic Mice to Bovine Spongiform Encephalopathy Infection following Passage in Sheep

Mice devoid of prion protein have cognitive deficits that are rescued by reconstitution of PrP in neurons

Glycosylation of PrPC Determines Timing of Neuroinvasion and Targeting in the Brain following Transmissible Spongiform Encephalopathy Infection by a Peripheral Route

Unravelling the glial response in the pathogenesis of Alzheimer's disease

Cellular prion protein expression is not regulated by the Alzheimer's amyloid precursor protein intracellular domain

Conditional Deletion of Prnp Rescues Behavioral and Synaptic Deficits after Disease Onset in Transgenic Alzheimer's Disease

Changing a single amino acid in the N-terminus of murine PrP alters TSE incubation time across three species barriers

Oral Prion Neuroinvasion Occurs Independently of PrPC Expression in the Gut Epithelium

The glycosylation status of PrPC is a key factor in determining transmissible spongiform encephalopathy transmission between species

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