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CuII(atsm) inhibits ferroptosis: Implications for treatment of neurodegenerative disease

British journal of pharmacology, 2020-02, Vol.177 (3), p.656-667

2020

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Risk prediction of late-onset Alzheimer’s disease implies an oligogenic architecture

Nature communications, 2020-09, Vol.11 (1), p.4799-4799, Article 4799

2020

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Motor and cognitive deficits in aged tau knockout mice in two background strains

Molecular neurodegeneration, 2014-08, Vol.9 (1), p.29-29, Article 29

2014

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Iron accumulates in Huntington's disease neurons: protection by deferoxamine

PloS one, 2013-10, Vol.8 (10), p.e77023-e77023

2013

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Clioquinol rescues Parkinsonism and dementia phenotypes of the tau knockout mouse

Neurobiology of disease, 2015-09, Vol.81, p.168-175

2015

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An iron–dopamine index predicts risk of parkinsonian neurodegeneration in the substantia nigra pars compacta

Chemical science (Cambridge), 2014, Vol.5 (6), p.2160-2169

2014

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Copper Ionophores as Novel Antiobesity Therapeutics

Molecules (Basel, Switzerland), 2020-10, Vol.25 (21), p.4957

2020

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Copper as a target for prostate cancer therapeutics: copper-ionophore pharmacology and altering systemic copper distribution

Oncotarget, 2016-06, Vol.7 (24), p.37064-37080

2016

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Increasing Cu bioavailability inhibits Aβ oligomers and tau phosphorylation

Proceedings of the National Academy of Sciences - PNAS, 2009-01, Vol.106 (2), p.381-386

2009

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Mitochondrial oxidative stress causes hyperphosphorylation of tau

PloS one, 2007-06, Vol.2 (6), p.e536-e536

2007

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Altered selenium status in Huntington's disease: Neuroprotection by selenite in the N171-82Q mouse model

Neurobiology of disease, 2014-11, Vol.71, p.34-42

2014

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Platinum-based inhibitors of amyloid-β as therapeutic agents for Alzheimer's disease

Proceedings of the National Academy of Sciences - PNAS, 2008-05, Vol.105 (19), p.6813-6818

2008

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Neuroprotective copper bis(thiosemicarbazonato) complexes promote neurite elongation

PloS one, 2014-02, Vol.9 (2), p.e90070-e90070

2014

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Presenilins Promote the Cellular Uptake of Copper and Zinc and Maintain Copper Chaperone of SOD1-dependent Copper/Zinc Superoxide Dismutase Activity

The Journal of biological chemistry, 2011-03, Vol.286 (11), p.9776-9786

2011

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Iron accumulation confers neurotoxicity to a vulnerable population of nigral neurons: implications for Parkinson's disease

Molecular neurodegeneration, 2014-07, Vol.9 (1), p.27-27, Article 27

2014

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Mechanisms of copper ion mediated Huntington's disease progression

PloS one, 2007-03, Vol.2 (3), p.e334-e334

2007

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Decreased Plasma Iron in Alzheimer’s Disease Is Due to Transferrin Desaturation

ACS chemical neuroscience, 2015-03, Vol.6 (3), p.398-402

2015

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Zn-DTSM, A Zinc Ionophore with Therapeutic Potential for Acrodermatitis Enteropathica?

Nutrients, 2019-01, Vol.11 (1), p.206

2019

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Effects of Neonatal Iron Feeding and Chronic Clioquinol Administration on the Parkinsonian Human A53T Transgenic Mouse

ACS chemical neuroscience, 2016-03, Vol.7 (3), p.360-366

2016

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Cysteine Oxidation within N-terminal Mutant Huntingtin Promotes Oligomerization and Delays Clearance of Soluble Protein

The Journal of biological chemistry, 2011-05, Vol.286 (20), p.18320-18330

2011

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Increased metal content in the TDP-43A315T transgenic mouse model of frontotemporal lobar degeneration and amyotrophic lateral sclerosis

Frontiers in aging neuroscience, 2014-02, Vol.6

2014

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Copper and Zinc Binding Modulates the Aggregation and Neurotoxic Properties of the Prion Peptide PrP106−126

Biochemistry (Easton), 2001-07, Vol.40 (27), p.8073-8084

2001

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Neonatal iron supplementation potentiates oxidative stress, energetic dysfunction and neurodegeneration in the R6/2 mouse model of Huntington's disease

Redox biology, 2015-04, Vol.4 (C), p.363-374

2015

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Increased zinc and manganese in parallel with neurodegeneration, synaptic protein changes and activation of Akt/GSK3 signaling in ovine CLN6 neuronal ceroid lipofuscinosis

PloS one, 2013-03, Vol.8 (3), p.e58644-e58644

2013

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Manganese chelation therapy extends survival in a mouse model of M1000 prion disease

Journal of neurochemistry, 2010-07, Vol.114 (2), p.440-451

2010

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CuII(atsm) inhibits ferroptosis: Implications for treatment of neurodegenerative disease

Risk prediction of late-onset Alzheimer’s disease implies an oligogenic architecture

Motor and cognitive deficits in aged tau knockout mice in two background strains

Iron accumulates in Huntington's disease neurons: protection by deferoxamine

Clioquinol rescues Parkinsonism and dementia phenotypes of the tau knockout mouse

An iron–dopamine index predicts risk of parkinsonian neurodegeneration in the substantia nigra pars compacta

Copper Ionophores as Novel Antiobesity Therapeutics

Copper as a target for prostate cancer therapeutics: copper-ionophore pharmacology and altering systemic copper distribution

Increasing Cu bioavailability inhibits Aβ oligomers and tau phosphorylation

Mitochondrial oxidative stress causes hyperphosphorylation of tau

Altered selenium status in Huntington's disease: Neuroprotection by selenite in the N171-82Q mouse model

Platinum-based inhibitors of amyloid-β as therapeutic agents for Alzheimer's disease

Neuroprotective copper bis(thiosemicarbazonato) complexes promote neurite elongation

Presenilins Promote the Cellular Uptake of Copper and Zinc and Maintain Copper Chaperone of SOD1-dependent Copper/Zinc Superoxide Dismutase Activity

Iron accumulation confers neurotoxicity to a vulnerable population of nigral neurons: implications for Parkinson's disease

Mechanisms of copper ion mediated Huntington's disease progression

Decreased Plasma Iron in Alzheimer’s Disease Is Due to Transferrin Desaturation

Zn-DTSM, A Zinc Ionophore with Therapeutic Potential for Acrodermatitis Enteropathica?

Effects of Neonatal Iron Feeding and Chronic Clioquinol Administration on the Parkinsonian Human A53T Transgenic Mouse

Cysteine Oxidation within N-terminal Mutant Huntingtin Promotes Oligomerization and Delays Clearance of Soluble Protein

Increased metal content in the TDP-43A315T transgenic mouse model of frontotemporal lobar degeneration and amyotrophic lateral sclerosis

Copper and Zinc Binding Modulates the Aggregation and Neurotoxic Properties of the Prion Peptide PrP106−126

Neonatal iron supplementation potentiates oxidative stress, energetic dysfunction and neurodegeneration in the R6/2 mouse model of Huntington's disease

Increased zinc and manganese in parallel with neurodegeneration, synaptic protein changes and activation of Akt/GSK3 signaling in ovine CLN6 neuronal ceroid lipofuscinosis

Manganese chelation therapy extends survival in a mouse model of M1000 prion disease

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